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Autor:
Thomas Kuner, Frédéric M. Vaz, Geralt Deubert, Simone Hoppe, Jana Lehmann, Mia L. Pras-Raves, Jürgen G. Okun, Heinz Horstmann, Christian Schultz, Antoine H. C. van Kampen, Christoph Körber, Kathrin V. Schwarz, Hongwei Zheng, Warda Darwisch, Öznur Singin, Markus Islinger, Hans R. Waterham, Sandra Kühl, Marino von Spangenberg, Johannes Roos
Publikováno v:
Communications Biology, 3(1):713. Springer Nature
Communications Biology, Vol 3, Iss 1, Pp 1-19 (2020)
Communications Biology
Communications Biology, Vol 3, Iss 1, Pp 1-19 (2020)
Communications Biology
ACBD5 deficiency is a novel peroxisome disorder with a largely uncharacterized pathology. ACBD5 was recently identified in a tethering complex mediating membrane contacts between peroxisomes and the endoplasmic reticulum (ER). An ACBD5-deficient mous
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::6f775867a22e5939010e74acef0c73be
https://pure.amc.nl/en/publications/cerebellar-and-hepatic-alterations-in-acbd5deficient-mice-are-associated-with-unexpected-distinct-alterations-in-cellular-lipid-homeostasis(56e8ab18-25cb-4df6-8ff9-498cb92a2eba).html
https://pure.amc.nl/en/publications/cerebellar-and-hepatic-alterations-in-acbd5deficient-mice-are-associated-with-unexpected-distinct-alterations-in-cellular-lipid-homeostasis(56e8ab18-25cb-4df6-8ff9-498cb92a2eba).html