Zobrazeno 1 - 10
of 190
pro vyhledávání: '"Gerald Sandler"'
Autor:
John T. Queenan, Louis M. Katz, S. Gerald Sandler, Margaret A. Keller, Clayton D. Simon, Gregory A. Denomme, Ralph R. Vassallo, Connie M. Westhoff, Willy A. Flegel, Susan T. Johnson, Meghan Delaney
Publikováno v:
Transfusion
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::13e16f228fbc2fb956eb41ec9e001d68
https://europepmc.org/articles/PMC9121350/
https://europepmc.org/articles/PMC9121350/
Publikováno v:
Transfusion. 57:2942-2945
BACKGROUND An 8-month-old recipient of a liver segment transplant had anti-D detected for the first time in her Day 5 posttransplant plasma and anti-C detected for the first time in her Day 55 posttransplant plasma. The donor's plasma contained anti-
Publikováno v:
British Journal of Haematology. 179:10-19
Approximately 0.2% to 1% of routine RhD blood typings result in a “serological weak D phenotype.” For more than 50 years, serological weak D phenotypes have been managed by policies to protect RhD-negative women of child-bearing potential from ex
Autor:
S. Gerald Sandler, John T. Queenan
Publikováno v:
Obstetrics & Gynecology. 130:633-635
Rh immunoprophylaxis for Rh-negative women requires an understanding of terminology used for Rh blood typing laboratory reports. The pathophysiology of Rh hemolytic disease of the fetus and newborn was elucidated by studies in rhesus monkeys. Subsequ
Autor:
Willy A. Flegel, S. Gerald Sandler
Publikováno v:
Transfusion. 59(7)
In 2005, the late George Garratty asked readers of TRANSFUSION, “Do we need to be more concerned about weak D antigens?”(1) His question was prompted by his observation that “… there have been increasing numbers of reports of patients who had
Autor:
Aaron A.R. Tobian, Ralph R. Vassallo, S. Gerald Sandler, Seema Kacker, Kevin D. Frick, Connie M. Westhoff, Margaret A. Keller
Publikováno v:
Transfusion. 55:2095-2103
In the United States, rates of hemolytic disease of the fetus and newborn (HDFN) have declined dramatically since the introduction of Rh immunoprophylaxis to prevent maternal–fetal alloimmunization.1,2 HDFN is caused by the transplacental passage o
Autor:
Gregory A. Denomme, Susan T. Johnson, Connie M. Westhoff, Margaret A. Keller, Ralph R. Vassallo, Meghan Delaney, Willy A. Flegel, John T. Queenan, Clayton D. Simon, S. Gerald Sandler, Louis M. Katz
Publikováno v:
Transfusion. 55:680-689
In 2014, the College of American Pathologists (CAP) Transfusion Medicine Resource Committee (TMRC) reported the results of a survey of more than 3100 laboratories concerning their policies and procedures for testing serological weak D phenotypes and
Publikováno v:
Transfusion. 55:199-204
Publikováno v:
Archives of Pathology & Laboratory Medicine. 138:620-625
Context.—Advances in RHD genotyping offer an opportunity to update policies and practices for testing weak D phenotypes and administration of Rh immune globulin to postpartum women.Objectives.—To repeat questions from a 1999 College of American P
Autor:
Lauren Lubrano, D. Brian Dawson, Jennifer L. Oliveira, Kenneth C. Swanson, Gerald Sandler, Michael J. Donnelly, James D. Hoyer
Publikováno v:
Hemoglobin. 38:137-141
Hb Memphis [α23(B4)Glu→Gln; HBA2: c.70G C (or HBA1)] is a stable hemoglobin (Hb) variant caused by a substitution of glutamine for glutamic acid at residue 23 of the α2- or α1-globin chain. Heterozygous Hb Memphis has no known clinical or hemato