Zobrazeno 1 - 10
of 166
pro vyhledávání: '"Gerald F. DiBona"'
Autor:
Markus P. Schlaich, Roland E. Schmieder, Thomas E. Lohmeier, Murray D. Esler, Márcio Galindo Kiuchi, Peter Ricci Pellegrino, Yehonatan Sharabi, Mark M. Knuepfer, Thomas H. Everett, Christopher T. Banek, Gregory D. Fink, Guido Grassi, Richard E. Katholi, Julian F. R. Paton, Michael Böhm, Ulla C. Kopp, John W. Osborn, Gerald F. DiBona, Kate M. Denton, David J. Lefer, Clive N. May, Felix Mahfoud
Publikováno v:
J Am Coll Cardiol
Three recent renal denervation studies in both drug-naïve and drug-treated hypertensive patients demonstrated a significant reduction of ambulatory blood pressure compared with respective sham control groups. Improved trial design, selection of rele
Autor:
Ulla C. Kopp, Gerald F. DiBona
Publikováno v:
Reflex Control of the Circulation ISBN: 9780367813338
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_________::7177ef14ed22aa98cc25f9d3e53bbaa5
https://doi.org/10.1201/9780367813338-17
https://doi.org/10.1201/9780367813338-17
Autor:
Gerald F. DiBona
Publikováno v:
Hypertension. 61:556-560
With the development and implementation of device-based therapeutic interventions to decrease renal and systemic nerve activity in patients with resistant hypertension, there has been an increase in research dealing with the role of the sympathetic n
Autor:
Peter Hansell, Angelica Fasching, Liselotte Pihl, Patrik Persson, Gerald F. DiBona, Fredrik Palm
Publikováno v:
American Journal of Physiology-Regulatory, Integrative and Comparative Physiology. 303:R39-R47
Glomerular filtration rate (GFR) and renal blood flow (RBF) are normally kept constant via renal autoregulation. However, early diabetes results in increased GFR and the potential mechanisms are debated. Tubuloglomerular feedback (TGF) inactivation,
Publikováno v:
Saeed, A, Dibona, G F, Marcussen, N & Guron, G 2010, ' High-NaCl intake impairs dynamic autoregulation of renal blood flow in ANG II-infused rats ', American Journal of Physiology: Regulatory, Integrative and Comparative Physiology, vol. 299, no. 5, pp. R1142-9 . https://doi.org/10.1152/ajpregu.00326.2010
The aim of this study was to investigate dynamic autoregulation of renal blood flow (RBF) in ANG II-infused rats and the influence of high-NaCl intake. Sprague-Dawley rats received ANG II (250 ng·kg−1·min−1sc) or saline vehicle (sham) for 14 da
Publikováno v:
American Journal of Physiology-Renal Physiology. 295:F1449-F1456
This study used 16 h/day measurement of renal blood flow (RBF) and arterial pressure (AP) to determine the role of nitric oxide (NO) in mediating the renal vasodilation caused by onset of type 1 diabetes. The AP and RBF power spectra were used to det
Autor:
Gerald F. DiBona
Publikováno v:
Annals of the New York Academy of Sciences. 940:395-406
Increases in renal sympathetic nerve activity (RSNA) regulate the functions of the nephron, the vasculature, and the renin-containing juxtaglomerular granular cells. As increased activity of the renin-angiotensin system can also influence nephron and
Autor:
Gerald F. DiBona, Linda L. Sawin
Publikováno v:
American Journal of Physiology-Renal Physiology. 287:F1171-F1178
The renal vasculature functions as an efficient low-pass filter of the multiple frequencies contained within renal sympathetic nerve activity. This study examined the effect of angiotensin II on the frequency response of the renal vasculature. Physio
Autor:
Gerald F. DiBona
Publikováno v:
Hypertension. 43:147-150
A major hypothesisfor the development of hypertension is that abnormal renal excretory function is critical for the initiation, development, and maintenance of primary hypertension.1 The renal body fluid feedback mechanism couples the long-term regul
Autor:
Susan Y. Jones, Gerald F. DiBona
Publikováno v:
Journal of Hypertension. 21:1539-1546
Objective To study effects of endogenous angiotensin II on responses to standardized stimulation of afferent neural input into the central portion of the arterial and cardiac baroreflexes. Design Different dietary sodium intakes were used to physiolo