Zobrazeno 1 - 10
of 16
pro vyhledávání: '"Georgi Gamkrelidze"'
Autor:
Nino Oganezovi, Vincenzo Lagani, Marine Kikvidze, Georgi Gamkrelidze, Lia Tsverava, Eka Lepsveridze, Kevin M. Kelly, Revaz Solomonia
Publikováno v:
IBRO Neuroscience Reports, Vol 16, Iss , Pp 291-299 (2024)
Background and purpose: Traumatic brain injury (TBI) and its consequences remain great challenges for neurology. Consequences of TBI are associated with various alterations in the brain but little is known about long-term changes of epigenetic DNA me
Externí odkaz:
https://doaj.org/article/0b122939eae24ca08fdff8f9155232da
Autor:
Manana Kandashvili, Georgi Gamkrelidze, Lia Tsverava, Tamar Lordkipanidze, Eka Lepsveridze, Vincenzo Lagani, Maia Burjanadze, Manana Dashniani, Merab Kokaia, Revaz Solomonia
Publikováno v:
International Journal of Molecular Sciences, Vol 23, Iss 3, p 1198 (2022)
Epilepsy is a severe neurological disease characterized by spontaneous recurrent seizures (SRS). A complex pathophysiological process referred to as epileptogenesis transforms a normal brain into an epileptic one. Prevention of epileptogenesis is a s
Externí odkaz:
https://doaj.org/article/42022636bb574edd9105a8156d5091e5
Autor:
Barbara L. Trommer, Chirag Shah, Sung Hwan Yun, Georgi Gamkrelidze, Emily S. Pasternak, W. Blaine Stine, Arlene Manelli, Patrick Sullivan, Joseph F. Pasternak, Mary Jo LaDu
Publikováno v:
Neurobiology of Disease, Vol 18, Iss 1, Pp 75-82 (2005)
Amyloid-β1–42 (Aβ1–42) is crucial to Alzheimer disease (AD) pathogenesis but the conformation of the toxic Aβ species remains uncertain. AD risk is increased by apolipoprotein E4 (apoE4) and decreased by apoE2 compared with the apoE3 isoform,
Externí odkaz:
https://doaj.org/article/8ecce7e34bf64738bc47a3380f0070e8
Autor:
Georgi Gamkrelidze, W. Blaine Stine, Sung Hwan Yun, Joseph F. Pasternak, Barbara L. Trommer, Mary Jo LaDu, Patrick Sullivan
Publikováno v:
Neuroscience Letters. 403:162-165
Amyloid-beta (Abeta) is causally implicated in Alzheimer's disease and neuroplasticity failure has acquired validity as a possible mechanism of early AD pathogenesis. We have previously demonstrated that oligomeric Abeta(1-42) inhibits LTP in the den
Autor:
Chirag Shah, Georgi Gamkrelidze, Joseph F. Pasternak, W. Blaine Stine, Sung Hwan Yun, Mary Jo LaDu, Arlene M. Manelli, Emily S. Pasternak, Patrick Sullivan, Barbara L. Trommer
Publikováno v:
Neurobiology of Disease, Vol 18, Iss 1, Pp 75-82 (2005)
Amyloid-beta1-42 (Abeta1-42) is crucial to Alzheimer disease (AD) pathogenesis but the conformation of the toxic Abeta species remains uncertain. AD risk is increased by apolipoprotein E4 (apoE4) and decreased by apoE2 compared with the apoE3 isoform
Publikováno v:
BMC Neuroscience, Vol 11, Iss Suppl 1, p P137 (2010)
BMC Neuroscience
BMC Neuroscience
The vestibuloocular reflex (VOR) provides an amenable model of learning and memory. Neurons from the Area II nucleus of goldfish are necessary for VOR plasticity, carrying an efferent copy of the eye velocity motor command to the cerebellum. Recently
Autor:
Georgi Gamkrelidze, Yuri L. Arshavsky, Lioudmila B. Popova, Yuri V. Panchin, Grigori N. Orlovsky
Publikováno v:
NeuroReport. 2:169-172
The effects of gamma-aminobutyric acid (GABA) on the behaviour of the marine pteropod mollusc Clione limacina were studied. In intact molluscs, injection of GABA evoked the consummatory stage of feeding behaviour, i.e. protracting the tentacles, open
Publikováno v:
BMC Neuroscience
BMC Neuroscience, Vol 8, Iss Suppl 2, p S20 (2007)
BMC Neuroscience, Vol 8, Iss Suppl 2, p S20 (2007)
Several studies have shown that dendritic morphology and spatial distributions of active ion channels contribute significantly to neuronal firing dynamics and signal processing, however the relative importance and interactions between these mechanism
Publikováno v:
Synaptic Plasticity and Transsynaptic Signaling ISBN: 038724008X
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_________::694d2000295f4c80b091590c48e4eef2
https://doi.org/10.1007/0-387-25443-9_30
https://doi.org/10.1007/0-387-25443-9_30
Autor:
Sidhartha Tan, Alice M. Wyrwicz, Alexander Drobyshevsky, Ning Ling Luo, Limin Li, Barbara L. Trommer, Xinhai Ji, Matthew Derrick, Georgi Gamkrelidze, Fan Meng, Stephen A. Back, Sheng-Kwei Song, Douglas J. Beardsley
Disruption of oligodendrocyte lineage progression is implicated in the white-matter injury that occurs in cerebral palsy. We have previously published a model in rabbits consistent with cerebral palsy. Little is known of normal white-matter developme
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::f867b9281c2769016517e87b8b092978
https://europepmc.org/articles/PMC6724805/
https://europepmc.org/articles/PMC6724805/