Zobrazeno 1 - 6
of 6
pro vyhledávání: '"Geoffrey P. Nase"'
Publikováno v:
Clinical and Experimental Pharmacology and Physiology. 29:138-142
SUMMARY 1. Hyperglycaemia in the vast majority of humans with diabetes mellitus is the end result of profound insulin resistance secondary to obesity. For patients in treatment, hyperglycaemia is usually not sustained but, rather, occurs intermittent
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::45adeb8c5d13dab614d5bf390449314e
https://doi.org/10.1046/j.1440-1681.2002.03617.x
https://doi.org/10.1046/j.1440-1681.2002.03617.x
Autor:
Geoffrey P. Nase, H. Glenn Bohlen
Publikováno v:
American Journal of Physiology-Heart and Circulatory Physiology. 280:H621-H627
betaII protein kinase C (betaPKC) is activated during acute and chronic hyperglycemia and may alter endothelial cell function. We determined whether blockade of betaPKC protected in vivo endothelial formation of NO, as measured with NO-sensitive micr
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::8c1f7e8a56a6002715969a020eee73e8
https://doi.org/10.1152/ajpheart.2001.280.2.h621
https://doi.org/10.1152/ajpheart.2001.280.2.h621
Autor:
Geoffrey P. Nase, H. G. Bohlen
Publikováno v:
American journal of physiology. Heart and circulatory physiology. 283(1)
Obesity is a risk for type II diabetes mellitus and increased vascular resistance. Disturbances of nitric oxide (NO) physiology occur in both obese animals and humans. In obese Zucker rats, we determined whether a protein kinase C-beta II (PKC-beta I
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::f18fa004fe6b01dca9d3773c561fca95
https://pubmed.ncbi.nlm.nih.gov/12063313
https://pubmed.ncbi.nlm.nih.gov/12063313
Autor:
Geoffrey P. Nase, H. Glenn Bohlen
Publikováno v:
American journal of physiology. Heart and circulatory physiology. 279(5)
Our hypothesis was that a large fraction of resting nitric oxide (NO) formation is driven by flow-mediated mechanisms in the intestinal microvasculature of the rat. NO-sensitive microelectrodes measured the in vivo perivascular NO concentration ([NO]
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::934e2e7502ed36730b3674b96e9d849e
https://pubmed.ncbi.nlm.nih.gov/11045960
https://pubmed.ncbi.nlm.nih.gov/11045960
Publikováno v:
The American journal of physiology. 277(4)
In the rat intestinal and cerebral microvasculatures, acute d-glucose hyperglycemia suppresses endothelium-dependent dilation to ACh without affecting endothelium-independent dilation to nitroprusside. This study determined whether acute hyperglycemi
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::8dda11ffdcf9601a195aa9edbc248538
https://pubmed.ncbi.nlm.nih.gov/10516190
https://pubmed.ncbi.nlm.nih.gov/10516190
Autor:
Matthew A. Boegehold, Geoffrey P. Nase
Publikováno v:
The American journal of physiology. 274(1)
The purpose of this study was to evaluate two potential stimuli for nitric oxide (NO) release in rat intestinal arterioles during sympathetic nerve activation. To determine whether these vessels contain endothelial α2-adrenoceptors linked to thel-ar
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::b40d850de9f8c14088e91b66bd13ea18
https://pubmed.ncbi.nlm.nih.gov/9458869
https://pubmed.ncbi.nlm.nih.gov/9458869