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of 2 258
pro vyhledávání: '"Gentry, K"'
Autor:
Sophiya John Ephrame, Gentry K. Cork, Victoria Marshall, Margaret A. Johnston, Jenna Shawa, Ibtihal Alghusen, Amy Qiang, Aspin R. Denson, Marisa S. Carman, Halyna Fedosyuk, Russell H. Swerdlow, Chad Slawson
Publikováno v:
Frontiers in Aging Neuroscience, Vol 15 (2023)
IntroductionAberrant activation of Extracellular Signal-Regulated Kinase (ERK) signaling is associated with Alzheimer’s disease (AD) pathogenesis. For example, enhanced ERK signal activation mediated by Apolipoprotein E4 (APOE4), which is a critica
Externí odkaz:
https://doaj.org/article/4863b0fc3f5c4b62a4efa9e498052308
Autor:
Kim H; Wallace H. Coulter Department of Biomedical Engineering, Georgia Institute of Technology and Emory University School of Medicine, Atlanta, GA, USA., Zenhausern R; Wallace H. Coulter Department of Biomedical Engineering, Georgia Institute of Technology and Emory University School of Medicine, Atlanta, GA, USA., Gentry K; Wallace H. Coulter Department of Biomedical Engineering, Georgia Institute of Technology and Emory University School of Medicine, Atlanta, GA, USA., Lian L; Wallace H. Coulter Department of Biomedical Engineering, Georgia Institute of Technology and Emory University School of Medicine, Atlanta, GA, USA., Huayamares SG; Wallace H. Coulter Department of Biomedical Engineering, Georgia Institute of Technology and Emory University School of Medicine, Atlanta, GA, USA., Radmand A; Petit Institute for Bioengineering and Biosciences, Georgia Institute of Technology, Atlanta, GA, USA.; Department of Chemical Engineering, Georgia Institute of Technology, Atlanta, GA, USA., Loughrey D; Wallace H. Coulter Department of Biomedical Engineering, Georgia Institute of Technology and Emory University School of Medicine, Atlanta, GA, USA., Podilapu AR; Wallace H. Coulter Department of Biomedical Engineering, Georgia Institute of Technology and Emory University School of Medicine, Atlanta, GA, USA., Hatit MZC; Wallace H. Coulter Department of Biomedical Engineering, Georgia Institute of Technology and Emory University School of Medicine, Atlanta, GA, USA., Ni H; Wallace H. Coulter Department of Biomedical Engineering, Georgia Institute of Technology and Emory University School of Medicine, Atlanta, GA, USA., Li A; Wallace H. Coulter Department of Biomedical Engineering, Georgia Institute of Technology and Emory University School of Medicine, Atlanta, GA, USA., Shajii A; Wallace H. Coulter Department of Biomedical Engineering, Georgia Institute of Technology and Emory University School of Medicine, Atlanta, GA, USA., Peck HE; Wallace H. Coulter Department of Biomedical Engineering, Georgia Institute of Technology and Emory University School of Medicine, Atlanta, GA, USA., Han K; Wallace H. Coulter Department of Biomedical Engineering, Georgia Institute of Technology and Emory University School of Medicine, Atlanta, GA, USA., Hua X; Wallace H. Coulter Department of Biomedical Engineering, Georgia Institute of Technology and Emory University School of Medicine, Atlanta, GA, USA., Jia S; Wallace H. Coulter Department of Biomedical Engineering, Georgia Institute of Technology and Emory University School of Medicine, Atlanta, GA, USA., Martinez M; California National Primate Research Center, University of California, Davis, Davis, CA, USA., Lee C; California National Primate Research Center, University of California, Davis, Davis, CA, USA.; Department of Pediatrics, University of California, Davis, Davis, CA, USA., Santangelo PJ; Wallace H. Coulter Department of Biomedical Engineering, Georgia Institute of Technology and Emory University School of Medicine, Atlanta, GA, USA., Tarantal A; California National Primate Research Center, University of California, Davis, Davis, CA, USA.; Department of Pediatrics, University of California, Davis, Davis, CA, USA.; Department of Cell Biology and Human Anatomy, University of California, Davis, Davis, CA, USA., Dahlman JE; Wallace H. Coulter Department of Biomedical Engineering, Georgia Institute of Technology and Emory University School of Medicine, Atlanta, GA, USA. james.dahlman@emory.edu.
Publikováno v:
Nature biotechnology [Nat Biotechnol] 2024 Nov 22. Date of Electronic Publication: 2024 Nov 22.
Publikováno v:
Aquaculture Environment Interactions, 2020 Jan 01. 12, 67-80.
Externí odkaz:
https://www.jstor.org/stable/26974216
Publikováno v:
Endangered Species Research, Vol 39, Pp 105-114 (2019)
There are more species threatened with extinction now that at any point in recent history. However, resources for species conservation can be limited, and awareness of gaps in knowledge and conservation action implementation is important when allocat
Externí odkaz:
https://doaj.org/article/d7166ceec979418695151758a2c2a081
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Publikováno v:
Frontiers in Endocrinology, Vol 9 (2018)
O-linked N-acetylglucosamine, better known as O-GlcNAc, is a sugar post-translational modification participating in a diverse range of cell functions. Disruptions in the cycling of O-GlcNAc mediated by O-GlcNAc transferase (OGT) and O-GlcNAcase (OGA)
Externí odkaz:
https://doaj.org/article/4ff9e0348ace41ab88c9b207815fd065
Autor:
Rogers, Heather, Gad, Ahmed, Cork, Gentry K., Menjivar, Nico, Schoolcraft, William B., Tesfaye, Dawit, Yuan, Ye
Publikováno v:
In Fertility and Sterility October 2024 122(4) Supplement:e168-e168
Publikováno v:
In Fertility and Sterility October 2024 122(4) Supplement:e81-e81
Autor:
Chad Slawson, Jeffrey Thompson, Reegan Miller, Heather M. Wilkins, Russell H. Swerdlow, Sarah Griffith, Amy Qiang, Gentry K. Cork, Jarrod Flax
Publikováno v:
J Alzheimers Dis
Background: Alzheimer’s disease (AD) features reductions in key bioenergetic fluxes and perturbed mitochondrial function. Cytoplasmic hybrids (cybrids) generated through the transfer of AD subject mitochondria to mtDNA-depleted SH-SY5Y neuroblastom
Autor:
Todd A. Lydic, Harmony Saunders, Patrick E. Fields, Ee Phie Tan, Gentry K. Cork, Jibiao Li, Maria T. Villar, Chad Slawson, Zhen Zhang, Miranda Machacek, Antonio Artigues, Tiangang Li
Publikováno v:
Journal of Biological Chemistry. 294:8973-8990
Chronic, low-grade inflammation increases the risk for atherosclerosis, cancer, and autoimmunity in diseases such as obesity and diabetes. Levels of CD4+ T helper 17 (Th17) cells, which secrete interleukin 17A (IL-17A), are increased in obesity and c