Zobrazeno 1 - 9
of 9
pro vyhledávání: '"Genevieve A. Morelli"'
Publikováno v:
American Journal of Hematology. 72:109-114
Thromboembolic (TE) events preceding cancer have been observed. Some studies failed to find this correlation. We retrospectively examined the cancer incidence following thromboembolic events in patients at our medical center. Medical records of 183 p
Publikováno v:
Cancer treatment and research. 148
Publikováno v:
Cancer Treatment and Research ISBN: 9780387799612
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_________::a838f11355e1a0613014ef405f36d61f
https://doi.org/10.1007/978-0-387-79962-9_9
https://doi.org/10.1007/978-0-387-79962-9_9
Publikováno v:
New England Journal of Medicine. 330:1789-1790
Hereditary hemorrhagic telangiectasia (Rendu-Osler-Weber disease) is characterized by telangiectatic lesions of the nose, lips, and visceral organs including the liver, spleen, gastrointestinal tract, lungs, brain, and spinal cord1–5. It is inherit
Publikováno v:
American Journal of Hematology. 17:295-306
The effect of heparin on platelet aggregation was systematically examined on platelets in plasma (PRP), as well as on gel-filtered, washed, and formaldehyde-fixed platelets. Results indicate that, although heparin causes a mild potentiation of platel
Publikováno v:
Thrombosis Research. 42:355-362
Human endothelial cells possess antiheparin activity that neutralizes the anticoagulant action of heparin as measured by different tests of the clotting system. The antiheparin activity appears to be associated with an acid-soluble basic protein pres
Publikováno v:
Thrombosis research. 34(1)
A variety of endotoxins, when added to human platelet-rich plasma (PRP) or to suspensions of washed platelets (WP), demonstrated an inhibitory effect on platelet aggregation induced by various aggregating agents. Endotoxin blocked the release of 14C
Autor:
Zaverio M. Ruggeri, Yoshihiro Fujimura, Genevieve A. Morelli, Hussain I. Saba, A. Bruce, Theodore S. Zimmerman, Sabiha R. Saba
Publikováno v:
American journal of hematology. 30(3)
The association of Type IIB von Willebrand disease (vWD) with chronic persistent thrombocytopenia and spontaneous platelet aggregation has recently been recognized. It has been shown that IIB von Willebrand factor (vWF) can initiate platelet aggregat
Publikováno v:
XIth International Congress on Thrombosis and Haemostasis.
Heparin has been reported to inhibit platelet aggregation. Our studies show that this activity is easily demonstrable in washed platelet systems, but fails to occur in citrated platelet-rich plasma (PRP) in the presence of a variety of agonists, exce