Zobrazeno 1 - 10
of 61
pro vyhledávání: '"Gema Vizcay‐Barrena"'
Autor:
Mark Rigby, Federico W. Grillo, Benjamin Compans, Guilherme Neves, Julia Gallinaro, Sophie Nashashibi, Sally Horton, Pedro M. Pereira Machado, Maria Alejandra Carbajal, Gema Vizcay-Barrena, Florian Levet, Jean-Baptiste Sibarita, Angus Kirkland, Roland A. Fleck, Claudia Clopath, Juan Burrone
Publikováno v:
Cell Reports, Vol 42, Iss 5, Pp 112397- (2023)
Summary: Excitatory synapses are typically described as single synaptic boutons (SSBs), where one presynaptic bouton contacts a single postsynaptic spine. Using serial section block-face scanning electron microscopy, we found that this textbook defin
Externí odkaz:
https://doaj.org/article/9fe2dcf6007c438582d9f4154a119fe9
Autor:
Sabrina Yahiya, Charlie N. Saunders, Sarah Hassan, Ursula Straschil, Oliver J. Fischer, Ainoa Rueda-Zubiaurre, Silvia Haase, Gema Vizcay-Barrena, Mufuliat Toyin Famodimu, Sarah Jordan, Michael J. Delves, Edward W. Tate, Anna Barnard, Matthew J. Fuchter, Jake Baum
Publikováno v:
Disease Models & Mechanisms, Vol 16, Iss 2 (2023)
Externí odkaz:
https://doaj.org/article/537be52acb9d42a7a54013ab74d8c046
Autor:
Sevda T Boyanova, Ethlyn Lloyd-Morris, Christopher Corpe, Khondaker Miraz Rahman, Doaa B Farag, Lee K Page, Hao Wang, Alice L Fleckney, Ariana Gatt, Claire Troakes, Gema Vizcay-Barrena, Roland Fleck, Suzanne J Reeves, Sarah A Thomas
Publikováno v:
PLoS ONE, Vol 18, Iss 10, p e0286278 (2023)
Blood-brain barrier (BBB) dysfunction may be involved in the increased sensitivity of Alzheimer's disease (AD) patients to antipsychotics, including amisulpride. Studies indicate that antipsychotics interact with facilitated glucose transporters (GLU
Externí odkaz:
https://doaj.org/article/0b39ddd0f90643538ab2a53245907976
Autor:
Oliver Lyons, James Walker, Christopher Seet, Mohammed Ikram, Adam Kuchta, Andrew Arnold, Magda Hernández-Vásquez, Maike Frye, Gema Vizcay-Barrena, Roland A. Fleck, Ashish S. Patel, Soundrie Padayachee, Peter Mortimer, Steve Jeffery, Siren Berland, Sahar Mansour, Pia Ostergaard, Taija Makinen, Bijan Modarai, Prakash Saha, Alberto Smith
Publikováno v:
JCI Insight, Vol 6, Iss 18 (2021)
Venous valve (VV) failure causes chronic venous insufficiency, but the molecular regulation of valve development is poorly understood. A primary lymphatic anomaly, caused by mutations in the receptor tyrosine kinase EPHB4, was recently described, wit
Externí odkaz:
https://doaj.org/article/ec0695e085a24322bfff5ed5770002cf
Autor:
Sim Lai Tung, Giorgia Fanelli, Robert Ian Matthews, Jordan Bazoer, Marilena Letizia, Gema Vizcay-Barrena, Farid N. Faruqu, Christina Philippeos, Rosalind Hannen, Khuloud T. Al-Jamal, Giovanna Lombardi, Lesley Ann Smyth
Publikováno v:
Frontiers in Cell and Developmental Biology, Vol 8 (2020)
Regulatory T cells (Tregs) are a subpopulation of CD4+ T cells with a fundamental role in maintaining immune homeostasis and inhibiting unwanted immune responses using several different mechanisms. Recently, the intercellular transfer of molecules be
Externí odkaz:
https://doaj.org/article/773536993a454f9fa1748b7fa66718d4
Autor:
Michela Borghesan, Juan Fafián-Labora, Olga Eleftheriadou, Paula Carpintero-Fernández, Marta Paez-Ribes, Gema Vizcay-Barrena, Avital Swisa, Dror Kolodkin-Gal, Pilar Ximénez-Embún, Robert Lowe, Belen Martín-Martín, Hector Peinado, Javier Muñoz, Roland A. Fleck, Yuval Dor, Ittai Ben-Porath, Anna Vossenkamper, Daniel Muñoz-Espin, Ana O’Loghlen
Publikováno v:
Cell Reports, Vol 27, Iss 13, Pp 3956-3971.e6 (2019)
Summary: Senescence is a cellular phenotype present in health and disease, characterized by a stable cell-cycle arrest and an inflammatory response called senescence-associated secretory phenotype (SASP). The SASP is important in influencing the beha
Externí odkaz:
https://doaj.org/article/b729ed53c3154141a7268dbbac8c71b2
Autor:
Annalisa Buniello, Neil J Ingham, Morag A Lewis, Andreea C Huma, Raquel Martinez‐Vega, Isabel Varela‐Nieto, Gema Vizcay‐Barrena, Roland A Fleck, Oliver Houston, Tanaya Bardhan, Stuart L Johnson, Jacqueline K White, Huijun Yuan, Walter Marcotti, Karen P Steel
Publikováno v:
EMBO Molecular Medicine, Vol 8, Iss 3, Pp 191-207 (2016)
Abstract WBP2 encodes the WW domain‐binding protein 2 that acts as a transcriptional coactivator for estrogen receptor α (ESR1) and progesterone receptor (PGR). We reported that the loss of Wbp2 expression leads to progressive high‐frequency hea
Externí odkaz:
https://doaj.org/article/0c9669943136495193cd48843b715189
Autor:
Gayathri N Sekhar, Ana R Georgian, Lisa Sanderson, Gema Vizcay-Barrena, Rachel C Brown, Paula Muresan, Roland A Fleck, Sarah A Thomas
Publikováno v:
PLoS ONE, Vol 12, Iss 3, p e0173474 (2017)
Pentamidine is an effective trypanocidal drug used against stage 1 Human African Trypanosomiasis (HAT). At the blood-brain barrier (BBB), it accumulates inside the endothelial cells but has limited entry into the brain. This study examined transporte
Externí odkaz:
https://doaj.org/article/28e9a686342e4da29a0188fb884b4905
Autor:
Paul E Grini, Tage Thorstensen, Vibeke Alm, Gema Vizcay-Barrena, Susanne S Windju, Tommy S Jørstad, Zoe A Wilson, Reidunn B Aalen
Publikováno v:
PLoS ONE, Vol 4, Iss 11, p e7817 (2009)
BACKGROUND:SET-domain proteins are histone lysine (K) methyltransferases (HMTase) implicated in defining transcriptionally permissive or repressive chromatin. The Arabidopsis ASH1 HOMOLOG 2 (ASHH2) protein (also called SDG8, EFS and CCR1) has been su
Externí odkaz:
https://doaj.org/article/192dae470d644e96a243e396680a6662
Autor:
Sevda T. Boyanova, Ethlyn Lloyd-Morris, Christopher Corpe, K. Miraz Rahman, Doaa B. Farag, Lee K. Page, Hao Wang, Alice L. Fleckney, Ariana Gatt, Claire Troakes, Gema Vizcay- Barrena, Roland Fleck, Suzanne J. Reeves, Sarah A. Thomas
Blood-brain barrier (BBB) dysfunction may be involved in the increased sensitivity of Alzheimer’s disease (AD) patients to antipsychotics, including amisulpride. Studies indicate that antipsychotics interact with facilitated glucose transporters (G
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_________::dd0cf121178726ba9009d5c125bea7cf
https://doi.org/10.1101/2023.05.15.540749
https://doi.org/10.1101/2023.05.15.540749
