Zobrazeno 1 - 5
of 5
pro vyhledávání: '"Gauri C. Bedi"'
Autor:
Stephanie A. Valente, Rahul D. Tendulkar, Sheen Cherian, Colin O’Rourke, Jon M. Greif, Lisa Bailey, Valery Uhl, Kevin P. Bethke, Eric D. Donnelly, Ray Rudolph, Aaron Pederson, Thomas Summer, S. Chace Lottich, Darrel L. Ross, Christine Laronga, Loretta Loftus, Andrea M. Abbott, Pond Kelemen, Ulrich Hermanto, Neil B. Friedman, Gauri C. Bedi, Jennifer E. Joh, William A. Thompson, Richard A. Hoefer, Jason P. Wilson, Song K. Kang, Barry Rosen, James Ruffer, Luis Bravo, Alice Police, Jamie M. Escallon, Anthony W. Fyles, David R. McCready, Gregory M. Graves, Nitin Rohatgi, Joyce A. Eaker, Jannine Graves, Shawna C. Willey, Eleni A. Tousimis, Brian T. Collins, Christina M. Shaw, Lee Riley, Nimisha Deb, Tricia Kelly, David L. Andolino, Marc E. Boisvert, Joanne Lyons, William Small, Stephen R. Grobmyer
Publikováno v:
Annals of surgical oncology. 23(9)
Single-dose intraoperative radiotherapy (IORT) is an emerging treatment for women with early stage breast cancer. The objective of this study was to define the frequency of IORT use, patient selection, and outcomes of patients treated in North Americ
Autor:
Milada S. Vala, Wafik S. El-Deiry, John Hilton, David Sidransky, Atul Bedi, James Barber, Gauri C. Bedi, Adil J. Akhtar, Richard J. Jones
Publikováno v:
Blood. 86:1148-1158
A critical determinant of the efficacy of antineoplastic therapy is the response of malignant cells to DNA damage induced by anticancer agents. The p53 tumor-suppressor gene is a critical component of two distinct cellular responses to DNA damage, th
Publikováno v:
Nature Medicine. 1:65-68
AIDS is associated with a high risk of certain malignancies, notably Kaposi's sarcoma (KS) and B-cell non-Hodgkin's lymphoma (NHL). The pathogenesis of these malignancies is not fully understood. One mechanism of malignant transformation recently des
Autor:
Atul Bedi, Céline Gélinas, Laura W. Engstrom, Qinwen Zeng, Ephraim J. Fuchs, Bijoyesh Mookerjee, Gauri. C. Bedi, Rajani Ravi
Publikováno v:
Nature cell biology. 3(4)
TRAIL (tumour-necrosis factor-related apoptosis ligand or Apo2L) triggers apoptosis through engagement of the death receptors TRAIL-R1 (also known as DR4) and TRAIL-R2 (DR5). Here we show that the c-Rel subunit of the transcription factor NF-kappaB i
Autor:
Siu Long Yao, Mack Mabry, Gauri C. Bedi, Saul J. Sharkis, Karen A. McKenna, Atul Bedi, David Sidransky, Richard J. Jones, Ephraim J. Fuchs, Adil J. Akhtar, Michael I. Collector, Rajani Ravi
Publikováno v:
Nature medicine. 2(10)
The induction of tumor cell death by anticancer therapy results from a genetic program of autonomous cell death termed apoptosis. Because the p53 tumor suppressor gene is a critical component for induction of apoptosis in response to DNA damage, its