Zobrazeno 1 - 10
of 36
pro vyhledávání: '"Gaurav Patki"'
Publikováno v:
Scientific Reports, Vol 7, Iss 1, Pp 1-8 (2017)
Abstract Air pollution resulting from exhaust emissions of vehicles has risen in the recent years, reportedly causing major adverse effects on the heart, lungs and the brain. Though respiratory and cardiovascular effects of these emissions are well i
Externí odkaz:
https://doaj.org/article/c85a86c4393e42059eacbbbc047bc151
Publikováno v:
Neurobiology of Disease, Vol 58, Iss , Pp 281-288 (2013)
Mitochondrial defects can have significant consequences on many aspects of neuronal physiology. In particular, deficiencies in the first enzyme complex of the mitochondrial respiratory chain (complex I) are considered to be involved in a number of hu
Externí odkaz:
https://doaj.org/article/44aa0e4f95564c7f9de7f02571bbf783
Autor:
Gaurav Patki, Ankita Salvi, Hesong Liu, Fatin Atrooz, Isam Alkadhi, Matthew Kelly, Samina Salim
Publikováno v:
PLoS ONE, Vol 10, Iss 3, p e0117498 (2015)
We have published that pharmacological induction of oxidative stress (OS) causes anxiety-like behavior in rats. Using animal models, we also have established that psychological stress induces OS and leads to anxiety-like behaviors. All evidence point
Externí odkaz:
https://doaj.org/article/1aee9d9b1cbd453c866d12d45059282d
Autor:
Gaurav Patki, Farida H Allam, Fatin Atrooz, An T Dao, Naimesh Solanki, Gaurav Chugh, Mohammad Asghar, Faizan Jafri, Ritu Bohat, Karim A Alkadhi, Samina Salim
Publikováno v:
PLoS ONE, Vol 8, Iss 9, p e74522 (2013)
Diminished estrogen influence at menopause is reported to be associated with cognitive decline, heightened anxiety and hypertension. While estrogen therapy is often prescribed to overcome these behavioral and physiological deficits, antioxidants whic
Externí odkaz:
https://doaj.org/article/63457f1773ee4b6db979ab42052604de
Publikováno v:
Frontiers in Aging Neuroscience, Vol 1 (2009)
Mitochondrial oxidative stress and dysfunction has been implicated as a possible mechanism for the onset and progression of Parkinson-like neurodegeneration. However, long-term mitochondrial defects in chronic animal neurodegenerative models have not
Externí odkaz:
https://doaj.org/article/85e6cf2ce9484131abf5043e003ef5aa
Publikováno v:
Progress in Neuro-Psychopharmacology and Biological Psychiatry. 81:80-87
Background Earlier, we have reported that post-traumatic stress disorder (PTSD)-like behaviors developed in rats that witnessed their cage mates undergo repeated traumatic stress. More recently, we published that early life physical traumatic stress
Publikováno v:
Neuroscience Letters. 600:28-32
It is becoming increasingly recognized that post-traumatic stress disorder (PTSD) can be acquired vicariously from witnessing traumatic events. Recently, we published an animal model called the “Trauma witness model” (TWM) which mimics PTSD-like
Autor:
Norma Castro-Guerrero, Takao Yagi, M. Sato, Hideto Miyoshi, Akemi Matsuno-Yagi, Subhash C. Sinha, Jesús Torres-Bacete, Prem Kumar Sinha, Gaurav Patki
Publikováno v:
Biochemistry
The NuoD segment (homologue of mitochondrial 49 kDa subunit) of the proton-translocating NADH:quinone oxidoreductase (complex I/NDH-1) from Escherichia coli is in the hydrophilic domain and bears many highly conserved amino acid residues. The three-d
Publikováno v:
Nutrition Research. 35:65-75
Previously, using the single-prolonged stress (SPS) rat model of posttraumatic stress disorder, we reported that moderate treadmill exercise, via modulation of oxidative stress-related mechanisms, rescued anxiety- and depression-like behaviors and re
Publikováno v:
Neuroscience Letters. 584:308-313
The social defeat paradigm involves aggressive encounters between Long-Evans (L-E) (resident) and Sprague-Dawley (S-D) (intruder) rats. Successful application of chronic social defeat stress in S-D rats is dependent upon selection of highly aggressiv