Transplanted human cones incorporate into the retina and function in a murine cone degeneration model.

Autor: Gasparini SJ; Center for Regenerative Therapies Dresden (CRTD), Center for Molecular and Cellular Bioengineering, Technische Universität, Dresden, Germany., Tessmer K; Center for Regenerative Therapies Dresden (CRTD), Center for Molecular and Cellular Bioengineering, Technische Universität, Dresden, Germany., Reh M; Natural and Medical Sciences Institute at the University of Tübingen, Reutlingen, Germany., Wieneke S; Center for Regenerative Therapies Dresden (CRTD), Center for Molecular and Cellular Bioengineering, Technische Universität, Dresden, Germany.; German Center for Neurodegenerative Diseases (DZNE), Dresden, Germany., Carido M; Center for Regenerative Therapies Dresden (CRTD), Center for Molecular and Cellular Bioengineering, Technische Universität, Dresden, Germany., Völkner M; Center for Regenerative Therapies Dresden (CRTD), Center for Molecular and Cellular Bioengineering, Technische Universität, Dresden, Germany.; German Center for Neurodegenerative Diseases (DZNE), Dresden, Germany., Borsch O; Center for Regenerative Therapies Dresden (CRTD), Center for Molecular and Cellular Bioengineering, Technische Universität, Dresden, Germany., Swiersy A; Center for Regenerative Therapies Dresden (CRTD), Center for Molecular and Cellular Bioengineering, Technische Universität, Dresden, Germany., Zuzic M; Center for Regenerative Therapies Dresden (CRTD), Center for Molecular and Cellular Bioengineering, Technische Universität, Dresden, Germany.; University of Bonn, Department of Ophthalmology, Bonn, Germany., Goureau O; Sorbonne Université, Institut de la Vision, INSERM, CNRS, Paris, France., Kurth T; Technology Platform, Center for Molecular and Cellular Bioengineering (CMCB), Technische Universität Dresden, Dresden, Germany., Busskamp V; Center for Regenerative Therapies Dresden (CRTD), Center for Molecular and Cellular Bioengineering, Technische Universität, Dresden, Germany.; University of Bonn, Department of Ophthalmology, Bonn, Germany., Zeck G; Natural and Medical Sciences Institute at the University of Tübingen, Reutlingen, Germany.; Institute of Biomedical Electronics, Technische Universität Wien, Vienna, Austria., Karl MO; Center for Regenerative Therapies Dresden (CRTD), Center for Molecular and Cellular Bioengineering, Technische Universität, Dresden, Germany.; German Center for Neurodegenerative Diseases (DZNE), Dresden, Germany., Ader M; Center for Regenerative Therapies Dresden (CRTD), Center for Molecular and Cellular Bioengineering, Technische Universität, Dresden, Germany.

Publikováno v: The Journal of clinical investigation [J Clin Invest] 2022 Jun 15; Vol. 132 (12).

Osteoblastic glucocorticoid signaling exacerbates high-fat-diet- induced bone loss and obesity.

Autor: Kim S; Bone Research Program, ANZAC Research Institute, The University of Sydney, Sydney, NSW, Australia.; Concord Clinical School, The University of Sydney, Sydney, NSW, Australia., Henneicke H; Bone Research Program, ANZAC Research Institute, The University of Sydney, Sydney, NSW, Australia.; Department of Medicine III, Technische University Dresden Medical Center, Dresden, Germany.; Center for Healthy Aging, Technische Universität Dresden Medical Center, Dresden, Germany.; Center for Regenerative Therapies Dresden, Technische University Dresden, Dresden, Germany., Cavanagh LL; Bone Research Program, ANZAC Research Institute, The University of Sydney, Sydney, NSW, Australia., Macfarlane E; Bone Research Program, ANZAC Research Institute, The University of Sydney, Sydney, NSW, Australia., Thai LJ; Bone Research Program, ANZAC Research Institute, The University of Sydney, Sydney, NSW, Australia., Foong D; Bone Research Program, ANZAC Research Institute, The University of Sydney, Sydney, NSW, Australia., Gasparini SJ; Bone Research Program, ANZAC Research Institute, The University of Sydney, Sydney, NSW, Australia., Fong-Yee C; Bone Research Program, ANZAC Research Institute, The University of Sydney, Sydney, NSW, Australia., Swarbrick MM; Bone Research Program, ANZAC Research Institute, The University of Sydney, Sydney, NSW, Australia.; Concord Clinical School, The University of Sydney, Sydney, NSW, Australia., Seibel MJ; Bone Research Program, ANZAC Research Institute, The University of Sydney, Sydney, NSW, Australia.; Concord Clinical School, The University of Sydney, Sydney, NSW, Australia.; Department of Endocrinology and Metabolism, Concord Repatriation General Hospital, The University of Sydney, Sydney, NSW, Australia., Zhou H; Bone Research Program, ANZAC Research Institute, The University of Sydney, Sydney, NSW, Australia. h.zhou@sydney.edu.au.; Concord Clinical School, The University of Sydney, Sydney, NSW, Australia. h.zhou@sydney.edu.au.

Publikováno v: Bone research [Bone Res] 2021 Sep 01; Vol. 9 (1), pp. 40. Date of Electronic Publication: 2021 Sep 01.

Skeletal glucocorticoid signalling determines leptin resistance and obesity in aging mice.

Autor: Henneicke H; Bone Research Program, ANZAC Research Institute, The University of Sydney, Australia; Center for Regenerative Therapies TU Dresden, Technische Universität Dresden, Germany; Department of Medicine III, Technische Universität Dresden, Germany; Center for Healthy Aging, Technische Universität Dresden, Germany. Electronic address: holger.henneicke@tu-dresden.de., Kim S; Bone Research Program, ANZAC Research Institute, The University of Sydney, Australia; Concord Clinical School, The University of Sydney, Australia. Electronic address: sarah.kim1@sydney.edu.au., Swarbrick MM; Bone Research Program, ANZAC Research Institute, The University of Sydney, Australia; Concord Clinical School, The University of Sydney, Australia. Electronic address: michael.swarbrick@sydney.edu.au., Li J; Bone Research Program, ANZAC Research Institute, The University of Sydney, Australia; Key Laboratory for Space Bioscience & Biotechnology, Institute of Special Environmental Biophysics, School of Life Sciences, Northwestern Polytechnical University, China. Electronic address: lijingbao@nwpu.edu.cn., Gasparini SJ; Bone Research Program, ANZAC Research Institute, The University of Sydney, Australia. Electronic address: sylvia.gasparini@tu-dresden.de., Thai J; Bone Research Program, ANZAC Research Institute, The University of Sydney, Australia. Electronic address: joanne.thai@sydney.edu.au., Foong D; Bone Research Program, ANZAC Research Institute, The University of Sydney, Australia. Electronic address: daphne.foong@sydney.edu.au., Cavanagh LL; Bone Research Program, ANZAC Research Institute, The University of Sydney, Australia. Electronic address: lauryn.cavanagh@sydney.edu.au., Fong-Yee C; Bone Research Program, ANZAC Research Institute, The University of Sydney, Australia. Electronic address: colette.fong-yee@sydney.edu.au., Karsten E; Kolling Institute of Medical Research, The University of Sydney, Australia. Electronic address: elisabeth@sanguibio.com., Lin RCY; Centre for Infectious Diseases and Microbiology, Westmead Institute for Medical Research, The University of Sydney, Australia; School of Medical Sciences, University of New South Wales Sydney, Australia. Electronic address: ruby.lin@sydney.edu.au., Cooper MS; Concord Clinical School, The University of Sydney, Australia. Electronic address: mark.cooper@sydney.edu.au., Zhou H; Bone Research Program, ANZAC Research Institute, The University of Sydney, Australia; Concord Clinical School, The University of Sydney, Australia. Electronic address: h.zhou@sydney.edu.au., Seibel MJ; Bone Research Program, ANZAC Research Institute, The University of Sydney, Australia; Concord Clinical School, The University of Sydney, Australia. Electronic address: markus.seibel@sydney.edu.au.

Publikováno v: Molecular metabolism [Mol Metab] 2020 Dec; Vol. 42, pp. 101098. Date of Electronic Publication: 2020 Oct 10.

A retrospective study utilized MIMIC-IV database to explore the potential association between triglyceride-glucose index and mortality in critically ill patients with sepsis.

Autor: Lou, Jiaqi¹ (AUTHOR), Xiang, Ziyi² (AUTHOR), Zhu, Xiaoyu³ (AUTHOR), Fan, Youfen¹ (AUTHOR), Song, Jingyao⁴ (AUTHOR), Cui, Shengyong¹ (AUTHOR), Li, Jiliang¹ (AUTHOR), Jin, Guoying¹ (AUTHOR), Huang, Neng¹ (AUTHOR) 460474225@qq.com

Publikováno v: Scientific Reports. 10/15/2024, Vol. 14 Issue 1, p1-15. 15p.

Metabolic and skeletal homeostasis are maintained in full locus GPRC6A knockout mice.

Autor: Jørgensen CV; Department of Drug Design and Pharmacology, Faculty of Health and Medical Sciences, University of Copenhagen, Universitetsparken 2, DK-2100, Copenhagen, Denmark., Gasparini SJ; Bone Research Program, ANZAC Research Institute, University of Sydney, Sydney, Australia., Tu J; Bone Research Program, ANZAC Research Institute, University of Sydney, Sydney, Australia., Zhou H; Bone Research Program, ANZAC Research Institute, University of Sydney, Sydney, Australia., Seibel MJ; Bone Research Program, ANZAC Research Institute, University of Sydney, Sydney, Australia. markus.seibel@sydney.edu.au., Bräuner-Osborne H; Department of Drug Design and Pharmacology, Faculty of Health and Medical Sciences, University of Copenhagen, Universitetsparken 2, DK-2100, Copenhagen, Denmark. hbo@sund.ku.dk.

Publikováno v: Scientific reports [Sci Rep] 2019 Apr 12; Vol. 9 (1), pp. 5995. Date of Electronic Publication: 2019 Apr 12.