Zobrazeno 1 - 10
of 31
pro vyhledávání: '"Garrett P. Larson"'
Autor:
Jaya Prakash Chalise, Ali Ehsani, Mengistu Lemecha, Yu-Wen Hung, Guoxiang Zhang, Garrett P. Larson, Keiichi Itakura
Publikováno v:
Frontiers in Immunology, Vol 14 (2023)
During B cell development in bone marrow, large precursor B cells (large Pre-B cells) proliferate rapidly, exit the cell cycle, and differentiate into non-proliferative (quiescent) small Pre-B cells. Dysregulation of this process may result in the fa
Externí odkaz:
https://doaj.org/article/74b33e895d8741968fbb75b5886ae35b
Autor:
Vincent M. Groner, Garrett E. Larson, Yuwei Kan, Mark F. Roll, James G. Moberly, Kristopher V. Waynant
Publikováno v:
Acta Crystallographica Section E: Crystallographic Communications, Vol 75, Iss 9, Pp 1394-1398 (2019)
The structure of the title complex, [Ag(C11H15N3S)2]PF6, has monoclinic (P21/c) symmetry, and the silver atom has a distorted square-planar geometry. The coordination complex crystallized from mixing silver hexafluoridophosphate with a concentrated t
Externí odkaz:
https://doaj.org/article/6a83f45e46c542a9a4f58171e4dd18ef
Autor:
Garrett P. Larson, David Johnson, James A. Stewart, Rebecca Sutphen, Peter J. O'Dwyer, John M. Kirkwood, Al B. Benson, Mary B. Daly, Jeffrey Weitzel, Theodore G. Krontiris, John J. Rossi, Jessica Alluin, Guillermo E. Rivas, Karim Majzoub, Laurent F. Thomas, David Smith, Sierra M. Li, Jacob Biesinger, Pål Sætrom
Supplementary Figure and Table Legends from A Risk Variant in an miR-125b Binding Site in BMPR1B Is Associated with Breast Cancer Pathogenesis
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::3a23044f59441af4e1582e00d9b8d275
https://doi.org/10.1158/0008-5472.22379138
https://doi.org/10.1158/0008-5472.22379138
Autor:
Garrett P. Larson, David Johnson, James A. Stewart, Rebecca Sutphen, Peter J. O'Dwyer, John M. Kirkwood, Al B. Benson, Mary B. Daly, Jeffrey Weitzel, Theodore G. Krontiris, John J. Rossi, Jessica Alluin, Guillermo E. Rivas, Karim Majzoub, Laurent F. Thomas, David Smith, Sierra M. Li, Jacob Biesinger, Pål Sætrom
MicroRNAs regulate diverse cellular processes and play an integral role in cancer pathogenesis. Genomic variation within miRNA target sites may therefore be important sources for genetic differences in cancer risk. To investigate this possibility, we
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_________::5b09c0dcda1f3a990eb10c06cdb0897a
https://doi.org/10.1158/0008-5472.c.6499148.v1
https://doi.org/10.1158/0008-5472.c.6499148.v1
Autor:
Garrett P. Larson, David Johnson, James A. Stewart, Rebecca Sutphen, Peter J. O'Dwyer, John M. Kirkwood, Al B. Benson, Mary B. Daly, Jeffrey Weitzel, Theodore G. Krontiris, John J. Rossi, Jessica Alluin, Guillermo E. Rivas, Karim Majzoub, Laurent F. Thomas, David Smith, Sierra M. Li, Jacob Biesinger, Pål Sætrom
Supplementary Methods, Tables 1-3, Figure 1 from A Risk Variant in an miR-125b Binding Site in BMPR1B Is Associated with Breast Cancer Pathogenesis
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::8c7d008071b75bb20f98f0256f0f0255
https://doi.org/10.1158/0008-5472.22379135
https://doi.org/10.1158/0008-5472.22379135
Publikováno v:
Mol Cell Endocrinol
Mice with global deletion of Arid5b expression are lean and resistant to diet-induced obesity, and Arid5b is required for adipogenesis in a variety of in vitro models. To determine whether the lean phenotype of Arid5b(−/−) mice can be explained b
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::18b26924342ac706c4a61c51b9eba581
https://europepmc.org/articles/PMC8956154/
https://europepmc.org/articles/PMC8956154/
Publikováno v:
PLoS ONE, Vol 5, Iss 12, p e14318 (2010)
We describe three statistical results that we have found to be useful in case-control genetic association testing. All three involve combining the discovery of novel genetic variants, usually by sequencing, with genotyping methods that recognize prev
Externí odkaz:
https://doaj.org/article/8cf51c9bb8be40189211d4b4a823aef2
Publikováno v:
Medicines
Volume 6
Issue 4
Medicines, Vol 6, Iss 4, p 104 (2019)
Volume 6
Issue 4
Medicines, Vol 6, Iss 4, p 104 (2019)
Background: Tumor necrosis factor (TNF)-related apoptosis-inducing ligand (TRAIL) selectively kills tumor cells in cancer patients. However, patients often develop TRAIL resistance
thus, agents that can sensitize cells to TRAIL therapy would be
thus, agents that can sensitize cells to TRAIL therapy would be
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::c832faa5fbd98aa1e51475eb494e2b4f
https://doi.org/10.3390/medicines6040104
https://doi.org/10.3390/medicines6040104
Autor:
Chelsy R. Jungbluth, Danielle Port, Josef Herzog, Jeffrey N. Weitzel, Esther M. John, Raquel Ogaz, Jessica Clague, Deborah I. Barragan, Sharon Sand, Mary S. Beattie, Susan L. Neuhausen, J. Salvador Saldivar, Gary Unzeitig, Garrett P. Larson, Paul Duncan, Arelis E. Martir-Negron, Charité Ricker, Nancy Feldman, Cheryl Cina
Publikováno v:
Journal of Clinical Oncology. 31:210-216
Purpose To determine the prevalence and type of BRCA1 and BRCA2 (BRCA) mutations among Hispanics in the Southwestern United States and their potential impact on genetic cancer risk assessment (GCRA). Patients and Methods Hispanics (n = 746) with a pe
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::1ef2a79a68e13135bc4e68cbdaa57981
https://doi.org/10.1200/jco.2011.41.0027
https://doi.org/10.1200/jco.2011.41.0027
Autor:
Garrett P. Larson, R. Steven Esworthy, Min Li, David D. Smith, M.L. Richard Yip, Fong-Fong Chu, Byung-Wook Kim
Publikováno v:
Inflammatory Bowel Diseases. 17:1373-1386
Genetic background has a profound effect on inflammatory bowel disease. The Gpx1 and Gpx2 double knockout (GPX1/2-DKO) mice on a mixed C57BL/6 (B6) and 129S1/SvimJ (129) background exhibit spontaneous ileocolitis. The DKO mice on a B6 background have
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::10eb38b28ca7e0c18abd418f68cb27f2
https://doi.org/10.1002/ibd.21479
https://doi.org/10.1002/ibd.21479