JIP3 interacts with dynein and kinesin-1 to regulate bidirectional organelle transport

Autor: Celestino, Ricardo, Gama, José B, Castro-Rodrigues, Artur F., Barbosa, Daniel J., Rocha, Helder, d'Amico, Ennio A., Musacchio, Andrea, Carvalho, Ana Xavier, Morais-Cabral, João H, Gassmann, Reto, d’Amico, Ennio A.

Publikováno v: Journal of Cell Biology. 221

The MAP kinase and motor scaffold JIP3 prevents excess lysosome accumulation in axons of vertebrates and invertebrates. How JIP3’s interaction with dynein and kinesin-1 contributes to organelle clearance is unclear. We show that human dynein light

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https://doi.org/10.1083/jcb.202110057

Proteomic Analysis of the Action of the Mycobacterium ulcerans Toxin Mycolactone: Targeting Host Cells Cytoskeleton and Collagen.

Autor: Gama, José B.^1,2 (AUTHOR), Ohlmeier, Steffen³ (AUTHOR), Martins, Teresa G.^1,2 (AUTHOR), Fraga, Alexandra G.^1,2 (AUTHOR), Sampaio-Marques, Belém^1,2 (AUTHOR), Carvalho, Maria A.⁴ (AUTHOR), Proença, Fernanda⁴ (AUTHOR), Silva, Manuel T.⁵ (AUTHOR), Pedrosa, Jorge^1,2 (AUTHOR) jpedrosa@ecsaude.uminho.pt, Ludovico, Paula^1,2 (AUTHOR)

Publikováno v: PLoS Neglected Tropical Diseases. 8/7/2014, Vol. 8 Issue 8, p1-14. 14p.

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Citopatogénese molecular induzida pela toxina do Mycobacterium ulcerans, micolactona

Autor: Gama, José B.

Publikováno v: Repositório Científico de Acesso Aberto de Portugal
Repositório Científico de Acesso Aberto de Portugal (RCAAP)
instacron:RCAAP
Ciência Vitae

Tese de doutoramento em Ciências da Saúde
Buruli ulcer (BU) is a tropical neglected, necrotizing skin disease caused by Mycobacterium ulcerans infection. The tissue damage that characterizes the disease is driven by the lipidic exotoxin mycola

Externí odkaz: https://explore.openaire.eu/search/publication?articleId=dedup_wf_001::d1026373c42c387febdd4bda3bef0211

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