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FOXO3 targets are reprogrammed as Huntington's disease neural cells and striatal neurons face senescence with p16 INK4a increase

Autor: Voisin, Jessica, Farina, Francesca, Naphade, Swati, Fontaine, Morgane, Tshilenge, Kizito‐Tshitoko, Galicia Aguirre, Carlos, Lopez‐Ramirez, Alejandro, Dancourt, Julia, Ginisty, Aurélie, Sasidharan Nair, Satish, Lakshika Madushani, Kuruwitage, Zhang, Ningzhe, Lejeune, François‐Xavier, Verny, Marc, Campisi, Judith, Ellerby, Lisa M., Neri, Christian

Publikováno v: Aging Cell
Aging Cell, Wiley Open Access, 2020, 19 (11), pp.e13226. ⟨10.1111/acel.13226⟩
Aging Cell, 2020, 19 (11), pp.e13226. ⟨10.1111/acel.13226⟩
Aging cell, vol 19, iss 11

Neurodegenerative diseases (ND) have been linked to the critical process in aging—cellular senescence. However, the temporal dynamics of cellular senescence in ND conditions is unresolved. Here, we show senescence features develop in human Huntingt

Externí odkaz: https://explore.openaire.eu/search/publication?articleId=pmid_dedup__::3073ced5c4e9ac2359c3c3fd8e35bdae
https://hal.sorbonne-universite.fr/hal-03020775/file/acel.13226.pdf