Zobrazeno 1 - 10
of 21
pro vyhledávání: '"Gail Kent"'
Publikováno v:
Journal of Neuroinflammation, Vol 16, Iss 1, Pp 1-14 (2019)
Abstract Background IL-10 knockout (KO) mice are protected from experimental autoimmune encephalomyelitis (EAE) with low-dose estrogen (E2) treatment similar to wild-type (WT) mice. Previous studies have demonstrated a decrease in tumor necrosis fact
Externí odkaz:
https://doaj.org/article/02ddf755ab684271af1209d868c71355
Autor:
Gil Benedek, Priya Chaudhary, Roberto Meza-Romero, Evan Calkins, Gail Kent, Halina Offner, Dennis Bourdette, Arthur A. Vandenbark
Publikováno v:
Journal of Neuroinflammation, Vol 14, Iss 1, Pp 1-11 (2017)
Abstract Background One of the main challenges in treating multiple sclerosis (MS) is reversing the effects of accumulated damage in the central nervous system (CNS) of progressive MS subjects. While most of the available drugs for MS subjects are an
Externí odkaz:
https://doaj.org/article/b3fbe08b80e14821a1f3485230bafc9a
Autor:
Chelsey D. Kline, Madeleine Anderson, John W. Bassett, Gail Kent, Rachel Berryman, Matthew Honeggar, Shosuke Ito, Kazumasa Wakamatsu, Arup K. Indra, Philip J. Moos, Sancy A. Leachman, Pamela B. Cassidy
Publikováno v:
Cancers; Volume 14; Issue 20; Pages: 5011
TR1 and other selenoproteins have paradoxical effects in melanocytes and melanomas. Increasing selenoprotein activity with supplemental selenium in a mouse model of UV-induced melanoma prevents oxidative damage to melanocytes and delays melanoma tumo
Autor:
Arthur A Vandenbark, Roberto Meza-Romero, Jack Wiedrick, Grant Gerstner, Hilary Seifert, Gail Kent, Marta Piechycna, Gil Benedek, Richard Bucala, Halina Offner
Publikováno v:
Cell Immunol
Our previous studies demonstrated increased serum levels of macrophage migration inhibitory factor (MIF-1) and its homologue, MIF-2, in males during MS progression; and that genetically high-MIF-expressing male subjects with relapsing multiple sclero
Autor:
Roberto Meza-Romero, Jack Wiedrick, Arthur A. Vandenbark, Ashley Headrick, Richard Bucala, Gail Kent, Gil Benedek, Halina Offner, Grant Gerstner, Hilary A. Seifert
Publikováno v:
Cell Immunol
Macrophage migration inhibitory factor (MIF-1) and its homologue d -dopachrome tautomerase (MIF-2) share the common CD74 receptor and function innately to enhance severity of multiple sclerosis (MS) as well as the experimental autoimmune encephalomye
Autor:
Zhiping Wang, Brett Cutler, Christina Topham, Yuangang Liu, Teri M. Greiling, Gail Kent, Doris Yang, Jennifer L. Strunck, Dylan Haynes, Pamela B. Cassidy, Barik Rajpal, Alex G. Ortega-Loayza
Publikováno v:
Journal of Investigative Dermatology. 142:235-239.e1
Autor:
Gail Kent, Roberto Meza-Romero, Arthur A. Vandenbark, Gil Benedek, Ha Nguyen, Halina Offner, Grant Gerstner
Publikováno v:
Metabolic Brain Disease. 34:153-164
Multiple sclerosis (MS) is a demyelinating and degenerative disease of the central nervous system (CNS) with a strong inflammatory component that affects more than 2 million people worldwide (and at least 400,000 in the United States). In MS, macroph
Autor:
Gail Kent, Jun Zhang, Halina Offner, Hilary A. Seifert, Ha Nguyen, Arthur A. Vandenbark, Gil Benedek
Publikováno v:
Journal of Neuroimmunology. 305:59-67
Immunoregulatory sex hormones, including estrogen and estriol, may prevent relapses in multiple sclerosis during pregnancy. Our previous studies have demonstrated that regulatory B cells are crucial for estrogen-mediated protection against experiment
Publikováno v:
Journal of Neuroinflammation
Journal of Neuroinflammation, Vol 16, Iss 1, Pp 1-14 (2019)
Journal of Neuroinflammation, Vol 16, Iss 1, Pp 1-14 (2019)
Background IL-10 knockout (KO) mice are protected from experimental autoimmune encephalomyelitis (EAE) with low-dose estrogen (E2) treatment similar to wild-type (WT) mice. Previous studies have demonstrated a decrease in tumor necrosis factor in all
Autor:
Arthur A. Vandenbark, Halina Offner, Roberto Meza-Romero, Gail Kent, Ashley Headrick, Hilary A. Seifert, Grant Gerstner, Priya Chaudhary, Jack Wiedrick, Ashley Maestas
Publikováno v:
Cell Immunol
Experimental autoimmune encephalomyelitis (EAE) is commonly used as an animal model for evaluating clinical, histological and immunological processes potentially relevant to the human disease multiple sclerosis (MS), for which the mode of disease ind