Výsledky vyhledávání

SLC44A2 deficient mice have a reduced response in stenosis but not in hypercoagulability driven venous thrombosis

Autor: Daniëlle Coenen, Sophie Dólleman, Annemarie M. van Oeveren-Rietdijk, Bart J.M. van Vlijmen, Judith M.E.M. Cosemans, Gaia Zirka, Hetty C. de Boer, Julia Tilburg, Pierre Morange, Mieke F.A. Karel, Grace M. Thomas, Henri H. Versteeg, Chrissta X. Maracle

Publikováno v: Journal of Thrombosis and Haemostasis, 18(7), 1714-1727. Wiley
Journal of Thrombosis and Haemostasis, 18(7), 1714-1727. WILEY
Journal of Thrombosis and Haemostasis
Journal of Thrombosis and Haemostasis, 2020, 18 (7), pp.1714-1727. ⟨10.1111/jth.14835⟩
Journal of Thrombosis and Haemostasis, Wiley, 2020, 18 (7), pp.1714-1727. ⟨10.1111/jth.14835⟩

Background Genome wide association studies (GWAS) identified SLC44A2 as a novel susceptibility gene for venous thrombosis (VT) and previous work established that SLC44A2 contributed to clot formation upon vascular injury.Objective To further investig

Externí odkaz: https://explore.openaire.eu/search/publication?articleId=doi_dedup___::03b97e5fdd2cea895cabd20960869551
https://cris.maastrichtuniversity.nl/en/publications/65410a1a-458e-4e26-b74c-59a5714ec1f9

Zobrazit plný text záznamu

Neutrophils recruited by leukotriene B4 induce features of plaque destabilization during endotoxaemia

Autor: Farid Slimani, Jean-Luc Vonesch, Xavier Norel, Anne-Laure Charles, Peggy Tilly, Marie-Anne Mawhin, Magnus Bäck, Jean-Baptiste Michel, Jean-Etienne Fabre, Gaia Zirka

Publikováno v: Cardiovascular Research
Cardiovascular Research, 2018, 114 (12), pp.1656-1666. ⟨10.1093/cvr/cvy130⟩

Aims: Both leukotrienes and neutrophils have been linked to plaque destabilization. Despite being evoked, the role of leukotriene B4 (LTB4) in neutrophil recruitment to plaques and the concomitant effects of these two actors on plaque stability remai

Externí odkaz: https://explore.openaire.eu/search/publication?articleId=doi_dedup___::ca3ef51a09bdde9858e7195db5ff42d1
https://doi.org/10.1093/cvr/cvy130

Zobrazit plný text záznamu

Abstract 090: Slc44a2 Deficient Mice Exhibit Less Severity of Thrombosis in a Stenosis Model of Deep Vein Thrombosis

Autor: Bart J.M. van Vlijmen, Julia Tilburg, Gaia Zirka, Grace M. Thomas, Lieke van den Heijkant, Pierre Morange, Chrissta X Maracle

Publikováno v: Arteriosclerosis, Thrombosis, and Vascular Biology. 38

Background/Objective: Recent genome wide association studies identified SLC44A2 as a novel susceptibility locus for venous thromboembolism (VTE), a region encoding the solute carrier family 44 member 2 protein (SLC44A2). Here we utilize Slc44a2 defic

Externí odkaz: https://explore.openaire.eu/search/publication?articleId=doi_________::3ce91a164e93366d99d2e1c7175799cd
https://doi.org/10.1161/atvb.38.suppl_1.090

Zobrazit plný text záznamu

Vyhledávací nástroje:

Upřesnit hledání