Zobrazeno 1 - 10
of 10
pro vyhledávání: '"Gaia Ameli"'
Autor:
Giulia Marzocchi, Carmela Gurrieri, Elisa Zuffa, Gaia Ameli, Maria Antonella Bardi, Cristina Papayannidis, Rossella Paolini, Nicoletta Testoni, Giovanni Martinelli, Simona Luatti, Laura Bonaldi, Michele Cavo, Antonio Cuneo, Emanuela Ottaviani, Gian Matteo Rigolin, Carmen Baldazzi
Publikováno v:
Genes, Chromosomes and Cancer. 55:375-388
Chromosomal rearrangements involving 3q26 are recurrent findings in myeloid malignancies leading to MECOM overexpression, which has been associated with a very poor prognosis. Other 3q abnormalities have been reported and cryptic MECOM rearrangements
Autor:
Stefania Paolini, Gaia Ameli, Giulia Marzocchi, Nicoletta Testoni, Simona Luatti, Carmen Baldazzi, Michele Cavo, Cristina Papayannidis, Giovanni Martinelli
Publikováno v:
American Journal of Hematology. 91:E14-E16
Autor:
Simona Soverini, Gabriele Gugliotta, Maria Teresa Bochicchio, Simona Luatti, Giulia Marzocchi, Gianantonio Rosti, Nicoletta Testoni, Carmen Baldazzi, Michele Cavo, Mario Tiribelli, Gaia Ameli, Fausto Castagnetti, Michele Baccarani, Giovanni Martinelli
Publikováno v:
Oncotarget
// Simona Luatti 1 , Carmen Baldazzi 1 , Giulia Marzocchi 1 , Gaia Ameli 1 , Maria Teresa Bochicchio 1 , Simona Soverini 1 , Fausto Castagnetti 1 , Mario Tiribelli 2 , Gabriele Gugliotta 1 , Giovanni Martinelli 1 , Michele Baccarani 1 , Michele Cavo
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::24f381b3adf37735bdf80c969baaeb8b
http://hdl.handle.net/11585/596558
http://hdl.handle.net/11585/596558
Autor:
Claudia Crippa, Nicoletta Testoni, Vittorio Montefusco, Iolanda Vincelli, Annalisa Pezzi, Michele Cavo, Carmen Baldazzi, Massimo Offidani, Federica Cavallo, Maria Teresa Petrucci, Simona Luatti, Anders Waage, Barbara Gamberi, Giulia Marzocchi, Elena Zamagni, Pieter Sonneveld, Gaia Ameli, Antonio Palumbo, Francesca Patriarca, F. Di Raimondo, Lucia Pantani, Sonja Zweegman, Alessandro Rambaldi
Publikováno v:
Patriarca, F, Offidani, M, Crippa, C, Vincelli, I, Pantani, L, Marzocchi, G, Cavallo, F, Gamberi, B, Baldazzi, C, Waage, A, Zamagni, E, Rambaldi, A, Testoni, N, Zweegman, S, Montefusco, V, Ameli, G, Petrucci, M T, Palumbo, A, Luatti, S, Sonneveld, P, Di Raimondo, F, Cavo, M & Pezzi, A 2015, ' Rare High Risk IgH Translocations in Newly Diagnosed Multiple Myeloma (MM): Cytogenetic Characterization and Relevance on Prognosis ', Clinical Lymphoma Myeloma and Leukemia, vol. 15, pp. e46-e47 . https://doi.org/10.1016/j.clml.2015.07.181
Clinical Lymphoma Myeloma and Leukemia, 15, e46-e47. Cancer Media Group
Clinical Lymphoma Myeloma and Leukemia, 15, e46-e47. Cancer Media Group
e46 therapies. Activating mutations of KRAS, NRAS and BRAF have been reported in approximately 23%, 20% and 6% of MM cases. Detection of these actionable mutations currently requires bone marrow (BM) aspirates that are invasive and can yield suboptim
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::ffdf70a438986b82ae2af6c76c1e1097
https://research.vumc.nl/en/publications/51507e0f-317b-427f-ba61-2c77e670ac75
https://research.vumc.nl/en/publications/51507e0f-317b-427f-ba61-2c77e670ac75
Autor:
Gaia Ameli, Carolina Terragna, Nicoletta Testoni, Marina Martello, Vincenza Solli, Rosalinda Termini, Michele Cavo, Beatrice Anna Zannetti, Barbara Santacroce, Giulia Marzocchi, Lucia Pantani, Giovanni Martinelli, Angela Flores Dico, Enrica Borsi, Elena Zamagni, Katia Mancuso, Paola Tacchetti
Publikováno v:
Cancer Research. 77:3936-3936
Intro: Multiple Myeloma (MM) is a biologically complex disease, whose genetic plasticity favors the coexistence of genetically heterogeneous subclones, selected in a Darwinian fashion throughout the disease course. Therapy might represent a major sel
Autor:
Flores Dico, Giovanni Martinelli, Vincenza Solli, Nicoletta Testoni, Marina Martello, Michele Cavo, Giulia Marzocchi, Gaia Ameli, Elena Zamagni, Lucia Pantani, Paola Tacchetti, Carolina Terragna, Beatrice Anna Zannetti, Barbara Santacroce, Enrica Borsi, Rosalinda Termini, Katia Mancuso
Publikováno v:
Clinical Lymphoma Myeloma and Leukemia. 17:e12
Autor:
Lucia Pantani, Michele Cavo, Vincenza Solli, Elena Zamagni, Carolina Terragna, Gaia Ameli, Marina Martello, Rosalinda Termini, Giulia Marzocchi, Giovanni Martinelli, Katia Mancuso, Enrica Borsi, Flores Dico, Beatrice Anna Zannetti, Barbara Santacroce, Paola Tacchetti, Nicoletta Testoni
Publikováno v:
ResearcherID
Background Multiple Myeloma (MM) is a biologically complex disease, whose genetic plasticity favors the coexistence of genetically heterogeneous subclones, selected in a Darwinian fashion throughout the disease course. Based on a disease evolution mo
Autor:
Giulia Marzocchi, Pieter Sonneveld, Anders Waage, Barbara Gamberi, Antonio Palumbo, Vittorio Montefusco, Nicoletta Testoni, Massimo Offidani, Claudia Crippa, Michele Cavo, Alessandro Rambaldi, Maria Teresa Petrucci, Annalisa Pezzi, Francesca Patriarca, Sonja Zweegman, Francesco Di Raimondo, Gaia Ameli, Iolanda Vincelli, Elena Zamagni, Federica Cavallo
Publikováno v:
Blood. 124:2042-2042
INTRODUCTION: Cytogenetic abnormalities (CA) are one of the major factor for risk stratification in Multiple Myeloma (MM). Approximately 50% of MM patients (pts) harbour a translocation involving the Immunoglobulin Heavy chain locus (IgH). Each trans
Autor:
Daniel Remondini, Lucia Pantani, Gaia Ameli, Carolina Terragna, Serena Rocchi, Annalisa Pezzi, Giovanni Martinelli, Giulia Marzocchi, Marina Martello, Angela Flores Dico, Nicoletta Testoni, Paola Tacchetti, Anna Beatrice Zannetti, Enrica Borsi, Elena Zamagni, Michele Cavo, Katia Mancuso, Annamaria Brioli, Barbara Santacroce
Publikováno v:
ResearcherID
Background Although remarkable advances have been achieved in MM therapy, mainly thanks to the introduction of novel-agent-based regimens, the disease remains incurable. Neoplastic CD138+ plasma cells are the hallmark of MM: both their expansion in t
Autor:
Paola Tacchetti, Serena Rocchi, Torsten Haferlach, Gaia Ameli, Carolina Terragna, Giulia Marzocchi, Marina Martello, Annalisa Pezzi, Flavio Mignone, Giovanni Martinelli, Lucia Pantani, Angela Flores Dico, Michele Cavo, Annamaria Brioli, Katia Mancuso, Enrica Borsi, Igor Saggese, Elena Zamagni, Beatrice Anna Zannetti, Barbara Santacroce, Nicoletta Testoni
Publikováno v:
ResearcherID
Introduction. The role of TP53 tumor-suppressor gene in mediating cellular basic tumor suppressive mechanisms is crucial: its utmost importance is underscored by a 50% mutational rate among most human cancers. In MM, TP53 is rarely reported as being