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pro vyhledávání: '"Gabriela I. Pereira-López"'
Autor:
Juan Calderón-Colmenero, Felipe Massó, Héctor González-Pacheco, Julio Sandoval, Carlos Guerrero, Jorge Cervantes-Salazar, José A. García-Montes, Araceli Paéz, Gabriela I. Pereira-López, Carlos Zabal-Cerdeira, Juan Pablo Sandoval
Publikováno v:
Frontiers in Pediatrics, Vol 11 (2023)
Endothelial progenitor cells and circulating endothelial cells have been proposed as useful markers of severity and disease progression in certain vascular diseases, including pulmonary arterial hypertension. Our study focused on evaluating the level
Externí odkaz:
https://doaj.org/article/0f6494df565347cca29675cf786e4963
Autor:
Carlos R. Sierra-Fernández, Gabriela I. Pereira-López, Juan Calderón-Colmenero, Pedro Iturralde-Torres, Santiago Nava-Townsend, Alfonso Buendía-Hernández
Publikováno v:
REC: CardioClinics. 56:190-196
Introduction and objectives Heart rhythm disorders are a frequent cause of consultation for pediatric cardiologists all over the world. Arrhythmias can present congenitally in a structurally normal heart, consequence of a congenital heart defect itse
Autor:
Diego B. Ortega‐Zhindón, Juan Calderón‐Colmenero, Gabriela I. Pereira‐López, Juan P. Sandoval, Frida Rivera‐Buendía, Jorge L. Cervantes‐Salazar
Publikováno v:
Journal of cardiac surgeryREFERENCES.
To evaluate the clinical and surgical outcomes among children with bicuspid aortic valve who underwent cardiac surgery.This observational and retrospective study included patients with a diagnosis of bicuspid aortic valve who underwent cardiac surger
Autor:
Miguel A, Alcántara-Ortigoza, Jesús, De Rubens-Figueroa, Miriam E, Reyna-Fabian, Bernardette, Estandía-Ortega, Ariadna, González-del Angel, Bertha, Molina-Álvarez, José A, Velázquez-Aragón, Sandra, Villagómez-Martínez, Gabriela I, Pereira-López, Víctor, Martínez-Cruz, Víctor, Cruz-Martínez, Rosa M, Álvarez-Gómez, Luisa, Díaz-García, Luisa, García-Díaz
Publikováno v:
Pediatric cardiology. 36(4)
Congenital heart defects (CHD) are found in ~50 % of Down syndrome (DS) patients. Genetic variants have been implicated, including CRELD1 mutations, but no previous study has examined the candidate genes, NKX2-5 and GATA4, in DS patients with secundu