Zobrazeno 1 - 10
of 49
pro vyhledávání: '"Gabriela Calounova"'
Autor:
Michael Welsh, Christer Betsholtz, Ying Sun, Erik Larsson, Jaroslav Mares, Björn Åkerblom, Gabriela Calounova, Guangxiang Zang, Vitezslav Kriz, Nina S. Funa
Supplementary Figure 3 from Dysfunctional Microvasculature as a Consequence of Shb Gene Inactivation Causes Impaired Tumor Growth
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::2e635d92c8d24140a341f43303e49bfd
https://doi.org/10.1158/0008-5472.22379607.v1
https://doi.org/10.1158/0008-5472.22379607.v1
Autor:
Michael Welsh, Christer Betsholtz, Ying Sun, Erik Larsson, Jaroslav Mares, Björn Åkerblom, Gabriela Calounova, Guangxiang Zang, Vitezslav Kriz, Nina S. Funa
Supplementary Figure Legends 1-7 from Dysfunctional Microvasculature as a Consequence of Shb Gene Inactivation Causes Impaired Tumor Growth
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::2e6713a04b4f5f72a1cfcc914efe0d1c
https://doi.org/10.1158/0008-5472.22379601.v1
https://doi.org/10.1158/0008-5472.22379601.v1
Autor:
Michael Welsh, Christer Betsholtz, Ying Sun, Erik Larsson, Jaroslav Mares, Björn Åkerblom, Gabriela Calounova, Guangxiang Zang, Vitezslav Kriz, Nina S. Funa
Supplementary Table 2 Legend from Dysfunctional Microvasculature as a Consequence of Shb Gene Inactivation Causes Impaired Tumor Growth
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::061c6d5d9fb8d43d04c6ed2562c1a251
https://doi.org/10.1158/0008-5472.22379592.v1
https://doi.org/10.1158/0008-5472.22379592.v1
Autor:
Michael Welsh, Christer Betsholtz, Ying Sun, Erik Larsson, Jaroslav Mares, Björn Åkerblom, Gabriela Calounova, Guangxiang Zang, Vitezslav Kriz, Nina S. Funa
Shb (Src homology 2 protein B) is an adapter protein downstream of the vascular endothelial growth factor receptor receptor-2 (VEGFR-2). Previous experiments have suggested a role for Shb in endothelial cell function. Recently, the Shb gene was inact
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_________::cd698ed6ec874da0761dd6556fe0476f
https://doi.org/10.1158/0008-5472.c.6499290.v1
https://doi.org/10.1158/0008-5472.c.6499290.v1
Autor:
Michael Welsh, Christer Betsholtz, Ying Sun, Erik Larsson, Jaroslav Mares, Björn Åkerblom, Gabriela Calounova, Guangxiang Zang, Vitezslav Kriz, Nina S. Funa
Supplementary Table 2 from Dysfunctional Microvasculature as a Consequence of Shb Gene Inactivation Causes Impaired Tumor Growth
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::6aaa61482e28545c876e003661e61c73
https://doi.org/10.1158/0008-5472.22379589
https://doi.org/10.1158/0008-5472.22379589
Autor:
Michael Welsh, Christer Betsholtz, Ying Sun, Erik Larsson, Jaroslav Mares, Björn Åkerblom, Gabriela Calounova, Guangxiang Zang, Vitezslav Kriz, Nina S. Funa
Supplementary Figure 2 from Dysfunctional Microvasculature as a Consequence of Shb Gene Inactivation Causes Impaired Tumor Growth
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::172db5f2e871c0b26d2fa95886878427
https://doi.org/10.1158/0008-5472.22379610
https://doi.org/10.1158/0008-5472.22379610
Autor:
Michael Welsh, Christer Betsholtz, Ying Sun, Erik Larsson, Jaroslav Mares, Björn Åkerblom, Gabriela Calounova, Guangxiang Zang, Vitezslav Kriz, Nina S. Funa
Supplementary Table 1 from Dysfunctional Microvasculature as a Consequence of Shb Gene Inactivation Causes Impaired Tumor Growth
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::36b5fba970ddd2cba8eeb62224998554
https://doi.org/10.1158/0008-5472.22379595
https://doi.org/10.1158/0008-5472.22379595
Autor:
Michael Welsh, Christer Betsholtz, Ying Sun, Erik Larsson, Jaroslav Mares, Björn Åkerblom, Gabriela Calounova, Guangxiang Zang, Vitezslav Kriz, Nina S. Funa
Supplementary Figure 1 from Dysfunctional Microvasculature as a Consequence of Shb Gene Inactivation Causes Impaired Tumor Growth
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::169587e73d34a875c18e118c7485fce1
https://doi.org/10.1158/0008-5472.22379613.v1
https://doi.org/10.1158/0008-5472.22379613.v1
Autor:
Thomas Lind, Caroline Öhman, Gabriela Calounova, Annica Rasmusson, Göran Andersson, Gunnar Pejler, Håkan Melhus
Publikováno v:
PLoS ONE, Vol 12, Iss 4, p e0176217 (2017)
Calvarial thinning and skull bone defects have been reported in infants with hypervitaminosis A. These findings have also been described in humans, mice and zebrafish with loss-of-function mutations in the enzyme CYP26B1 that degrades retinoic acid (
Externí odkaz:
https://doaj.org/article/f0636ddbc9cc4df08fb826b3585ce33f
Autor:
Thomas Lind, Ann-Marie Gustafson, Gabriela Calounova, Lijuan Hu, Annica Rasmusson, Kenneth B Jonsson, Sara Wernersson, Magnus Åbrink, Göran Andersson, Sune Larsson, Håkan Melhus, Gunnar Pejler
Publikováno v:
PLoS ONE, Vol 11, Iss 12, p e0167964 (2016)
Here we addressed the potential impact of chymase, a mast-cell restricted protease, on mouse bone phenotype. We show that female mice lacking the chymase Mcpt4 acquired a persistent expansion of diaphyseal bone in comparison with wild type controls,
Externí odkaz:
https://doaj.org/article/f44d1ff4d0f847a495a8ab062f799a9d