Zobrazeno 1 - 3
of 3
pro vyhledávání: '"Gabriel Ocana-Santero"'
Autor:
Serge Ducommun, Paulo R. Jannig, Igor Cervenka, Marta Murgia, Melanie J. Mittenbühler, Ekaterina Chernogubova, José M. Dias, Baptiste Jude, Jorge C. Correia, Jonathan G. Van Vranken, Gabriel Ocana-Santero, Margareta Porsmyr-Palmertz, Sarah McCann Haworth, Vicente Martínez-Redondo, Zhengye Liu, Mattias Carlström, Matthias Mann, Johanna T. Lanner, Ana I. Teixeira, Lars Maegdefessel, Bruce M. Spiegelman, Jorge L. Ruas
Publikováno v:
Molecular Metabolism, Vol 82, Iss , Pp 101912- (2024)
Objective: Skeletal muscle plasticity and remodeling are critical for adapting tissue function to use, disuse, and regeneration. The aim of this study was to identify genes and molecular pathways that regulate the transition from atrophy to compensat
Externí odkaz:
https://doaj.org/article/2abd30b26178418fbe5ccf30fb438f44
Publikováno v:
International Journal of Molecular Sciences, Vol 22, Iss 4, p 1815 (2021)
Friedreich’s ataxia is an autosomal recessive neurogenetic disease that is mainly associated with atrophy of the spinal cord and progressive neurodegeneration in the cerebellum. The disease is caused by a GAA-expansion in the first intron of the fr
Externí odkaz:
https://doaj.org/article/670d199b0a4c40419ef372937d2bf54f
Publikováno v:
Biblos-e Archivo. Repositorio Institucional de la UAM
instname
International Journal of Molecular Sciences
International Journal of Molecular Sciences, Vol 22, Iss 1815, p 1815 (2021)
Digital.CSIC. Repositorio Institucional del CSIC
instname
International Journal of Molecular Sciences
International Journal of Molecular Sciences, Vol 22, Iss 1815, p 1815 (2021)
Digital.CSIC. Repositorio Institucional del CSIC
Friedreich’s ataxia is an autosomal recessive neurogenetic disease that is mainly associated with atrophy of the spinal cord and progressive neurodegeneration in the cerebellum. The disease is caused by a GAA-expansion in the first intron of the fr
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::ed0713b84f4fdaaf931f8330c0752373
http://hdl.handle.net/10486/701710
http://hdl.handle.net/10486/701710