Zobrazeno 1 - 7
of 7
pro vyhledávání: '"Gabriel Casella"'
Autor:
Maryellen Giger, Marcus Clark, Madeleine Durkee, Rebecca Abraham, Junting Ai, Gabriel Casella, Deepjyoti Ghosh, Thao Cao
Publikováno v:
Lupus Science and Medicine, Vol 9, Iss Suppl 3 (2022)
Externí odkaz:
https://doaj.org/article/36ada8f561a44278a530cadc480d6a73
Autor:
Rebecca Abraham, Madeleine S. Durkee, Junting Ai, Margaret Veselits, Gabriel Casella, Yuta Asano, Anthony Chang, Kichul Ko, Charles Oshinsky, Emily Peninger, Maryellen L. Giger, Marcus R. Clark
Publikováno v:
The Journal of Clinical Investigation, Vol 132, Iss 13 (2022)
BACKGROUND In human lupus nephritis (LN), tubulointerstitial inflammation (TII) on biopsy predicts progression to end-stage renal disease (ESRD). However, only about half of patients with moderate-to-severe TII develop ESRD. We hypothesized that this
Externí odkaz:
https://doaj.org/article/48c07970fe1844bdb97aea821879975f
Autor:
Madeleine S. Durkee, Nevaeh Petrie, Kyle Lleras, Junting Ai, Rebecca Abraham, J. Cy Chittenden, Chasity Kasir, Fiona Clark, Gabriel Casella, Marcus R. Clark, Maryellen L. Giger
Publikováno v:
Medical Imaging 2023: Image Perception, Observer Performance, and Technology Assessment.
Autor:
Marcus Clark, Madeleine Durkee, Rebecca Abraham, Gabriel Casella, Junting Ai, Deepjyoti Ghosh, Thao Cao, Maryellen Giger
Publikováno v:
Lupus Nephritis.
Autor:
Rebecca Abraham, Madeleine S. Durkee, Junting Ai, Margaret Veselits, Gabriel Casella, Yuta Asano, Anthony Chang, Kichul Ko, Charles Oshinsky, Emily Peninger, Maryellen L. Giger, Marcus R. Clark
Publikováno v:
The Journal of clinical investigation. 132(13)
BACKGROUNDIn human lupus nephritis (LN), tubulointerstitial inflammation (TII) on biopsy predicts progression to end-stage renal disease (ESRD). However, only about half of patients with moderate-to-severe TII develop ESRD. We hypothesized that this
Autor:
Gabriel Casella, Rachel Munk, Myriam Gorospe, Yulan Piao, Supriyo De, Kotb Abdelmohsen, Kyoung Mi Kim
Publikováno v:
Nucleic Acids Research. 47:7294-7305
Cellular senescence, an integral component of aging and cancer, arises in response to diverse triggers, including telomere attrition, macromolecular damage and signaling from activated oncogenes. At present, senescent cells are identified by the comb
Publikováno v:
Wiley Interdiscip Rev RNA
Cellular senescence, a developmental program central to normal aging and aging pathologies, is robustly regulated at the post-transcriptional level. This regulation involves the interaction of RNA-binding proteins and noncoding RNAs with senescence-a