Zobrazeno 1 - 3
of 3
pro vyhledávání: '"Gabrielė Vydmantaitė"'
Autor:
Deimantė Narauskaitė, Gabrielė Vydmantaitė, Justina Rusteikaitė, Revathi Sampath, Akvilė Rudaitytė, Gabija Stašytė, María Isabel Aparicio Calvente, Aistė Jekabsone
Publikováno v:
Pharmaceuticals, Vol 14, Iss 8, p 811 (2021)
Each year, millions of individuals suffer from a non-healing wound, abnormal scarring, or injuries accompanied by an infection. For these cases, scientists are searching for new therapeutic interventions, from which one of the most promising is the u
Externí odkaz:
https://doaj.org/article/4c2f622d5d6c489495aa0cf0ee249112
Autor:
María Isabel Aparicio Calvente, Revathi Sampath, Justina Rusteikaitė, Aistė Jekabsone, Deimantė Narauskaitė, Akvilė Rudaitytė, Gabija Stašytė, Gabrielė Vydmantaitė
Publikováno v:
Pharmaceuticals
Pharmaceuticals, Vol 14, Iss 811, p 811 (2021)
Pharmaceuticals, Vol 14, Iss 811, p 811 (2021)
Each year, millions of individuals suffer from a non-healing wound, abnormal scarring, or injuries accompanied by an infection. For these cases, scientists are searching for new therapeutic interventions, from which one of the most promising is the u
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::ca93ddb94a2d90042064599295d63723
Autor:
Marco Corazzari, Laura Tapella, Giulia Dematteis, Carla Distasi, Ramunė Morkūnienė, Marcello Manfredi, Elettra Barberis, Federico Alessandro Ruffinatti, Mariagrazia Grilli, Elena Grossini, Serena Farruggio, Aistė Jekabsone, Dmitry Lim, Armando A. Genazzani, Eleonora Ferrari, Emilio Marengo, Malak Chahin, Gabrielė Vydmantaitė
Publikováno v:
Cell Death & Disease, London : Nature Publishing Group, 2020, vol. 11, no. 8, 645, p. 1-6
Cell Death & Disease
Cell Death and Disease, Vol 11, Iss 8, Pp 1-16 (2020)
Cell Death & Disease
Cell Death and Disease, Vol 11, Iss 8, Pp 1-16 (2020)
The pathogenesis of Alzheimer’s disease (AD), a slowly-developing age-related neurodegenerative disorder, is a result of the action of multiple factors including deregulation of Ca2+ homeostasis, mitochondrial dysfunction, and dysproteostasis. Inte