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pro vyhledávání: '"GBR-12935"'
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Akademický článek
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Autor:
Tamás Ollmann, Anita Kovács, Veronika Kállai, Attila Toth, Beáta Berta, László Péczely, Zoltán Petykó, Rita Gálosi, László Lénárd
Publikováno v:
Behavioural Brain Research. 344:57-64
Effects of destroyed noradrenergic (NE) innervation in the medial prefrontal cortex (mPFC) were examined on dopamine (DA) content and metabolism. Six-hydroxy-DOPA (6-OHDOPA) or 6-hydroxy-dopamine (6-OHDA) in combination with a potent DA reuptake inhi
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::c3b089a2a7ad2d250573fb7882ba4cee
https://doi.org/10.1016/j.bbr.2018.02.016
https://doi.org/10.1016/j.bbr.2018.02.016
Akademický článek
Tento výsledek nelze pro nepřihlášené uživatele zobrazit.
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Publikováno v:
Addi. Archivo Digital para la Docencia y la Investigación
instname
PLoS ONE, Vol 11, Iss 9, p e0162416 (2016)
PLoS ONE
instname
PLoS ONE, Vol 11, Iss 9, p e0162416 (2016)
PLoS ONE
Dopamine can influence NMDA receptor function and regulate glutamate-triggered long-term changes in synaptic strength in several regions of the CNS. In spinal cord, regulation of the threshold of synaptic plasticity may determine the proneness to und
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::c79052472093cce78100b383c0e89d23
http://hdl.handle.net/10810/32527
http://hdl.handle.net/10810/32527
Publikováno v:
Neuropsychopharmacology. 34:1233-1244
It is well documented that somatodendritically released dopamine is important in the excitability and synaptic transmission of midbrain dopaminergic neurons. Recently we showed that in midbrain slices, acute ethanol exposure facilitates glutamatergic
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::2eaaab82bf56fcbcbbb9970f24c80c9c
https://doi.org/10.1038/npp.2008.143
https://doi.org/10.1038/npp.2008.143
Publikováno v:
Journal of Neurochemistry. 62:338-341
The binding of the dopamine uptake inhibitor [3H]GBR-12935 to 16 regions of the human brain was investigated in competition experiments with increasing concentrations of GBR-12909, mazindol, and dopamine. The methodology used included a relatively hi
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_________::3e235836981482932dbdfbb0e37258c2
https://doi.org/10.1046/j.1471-4159.1994.62010338.x
https://doi.org/10.1046/j.1471-4159.1994.62010338.x
Publikováno v:
Journal of Pharmacology and Experimental Therapeutics. 325:165-174
Anesthesiologists among physicians are on the top of the drug abuse list, and the mechanism is unclear. Recent studies suggest occupation-related second-hand exposure to i.v. drugs, including propofol, may play a role. Growing evidence indicates that
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::cd340b75bae5cc5c0447aad50a869884
https://doi.org/10.1124/jpet.107.132472
https://doi.org/10.1124/jpet.107.132472
Publikováno v:
European Journal of Pharmacology. 554:128-136
In previous in vivo voltammetry studies, acute nicotine administration increased striatal dopamine clearance. The current study aimed to determine whether nicotine also increases [(3)H]dopamine uptake across the time course of the previous voltammetr
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::679709ca6294a5ae905c8fc74f1ca83b
https://doi.org/10.1016/j.ejphar.2006.09.074
https://doi.org/10.1016/j.ejphar.2006.09.074
Autor:
Brett J. Stanley, Carmela M. Reichel, Sanders A. McDougall, Cristal M. Farley, Cynthia A. Crawford, Jennifer J. Wacan
Publikováno v:
Neurotoxicology and Teratology. 28:323-332
In the present study, we examined whether exposing rats to manganese (Mn) during the preweanling period would affect basal or cocaine-induced locomotor activity in adulthood and reduce the number of striatal dopamine transporter binding sites. On pos
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::53d8a9bcceba43ccdaf4f25cf206ac65
https://doi.org/10.1016/j.ntt.2006.02.002
https://doi.org/10.1016/j.ntt.2006.02.002