Zobrazeno 1 - 10
of 29
pro vyhledávání: '"G.K. Matheson"'
Publikováno v:
General Pharmacology: The Vascular System. 27:355-361
Chronic buspirone or ipsapirone (3 mg/kg, twice daily) administration to rats for 10 days decreased the sensitivity of inhibition of single-unit activity of serotonergic dorsal raphe neurons to a challenge by each drug. The ED 50 for buspirone was in
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::3a5d0ab39bac8c9af60882f92d30ad51
https://doi.org/10.1016/0306-3623(95)02027-6
https://doi.org/10.1016/0306-3623(95)02027-6
Publikováno v:
Neuropharmacology. 30:79-87
Both BMY-14802 (alpha-(4-fluorophenyl)-4-(5-fluoro-2-pyrimidinyl)-1-piperazine-butanol hydrochloride) and gevotroline (WY-47384; 8-fluoro-2,3,4,5-tetrahydro-2-[3-(3-pyridinyl)-propyl]-1 H -pyrido[4,3- b ]indole hydrochloride) are known to antagonize
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::90e93ae7e12c503c3835d8126a525d0d
https://doi.org/10.1016/0028-3908(91)90046-e
https://doi.org/10.1016/0028-3908(91)90046-e
Publikováno v:
General pharmacology. 25(4)
1. The effects of buspirone, gepirone, ipsapirone and tandospirone on spontaneously discharging serotonergic neurons of the dorsal raphe were determined under the same experimental conditions. 2. Buspirone, gepirone, ipsapirone and tandospirone were
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::c6e7cb27b527c0b6806d8af100df96fa
https://pubmed.ncbi.nlm.nih.gov/7958728
https://pubmed.ncbi.nlm.nih.gov/7958728
Publikováno v:
General Pharmacology: The Vascular System. 18:269-273
1. 1. Several structural analogues of GABA were shown to be inhibitors of GABA A receptor binding in membranes from cat cerebral cortex. 2. 2. These compounds were 3-aminopropanesulfonic acid (APS; IC 50 = 0.04 μ M), imidazoleacetic acid (IMA; IC 50
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::e2e40e5d6c9fc7752a81cfd25ee72a20
https://doi.org/10.1016/0306-3623(87)90010-3
https://doi.org/10.1016/0306-3623(87)90010-3
Publikováno v:
Neuropharmacology. 25:1191-1195
The actions of four analogues of γ-aminobutyric acid (GABA) on blood pressure and heart rate were measured in the cat after intracerebroventricular administration. These compounds were previously found to inhibit binding to GABAA receptors of neuron
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::e2c7e911227d5131447d5ed25eba0be2
https://doi.org/10.1016/0028-3908(86)90135-8
https://doi.org/10.1016/0028-3908(86)90135-8
Publikováno v:
Neuropharmacology. 27(8)
The effects of the anxiolytic agents, buspirone and diazepam, on the hypothalamic-pituitary-adrenal axis (HPAA), indicated by changes in the concentration of corticosterone (CS) in plasma, were studied 1 2 , 1, 2, 4, 6, 8 and 24 hr after administrati
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::e684831febda3103da842de0ae284798
https://pubmed.ncbi.nlm.nih.gov/3216961
https://pubmed.ncbi.nlm.nih.gov/3216961
Autor:
Deanne Guthrie, Janet Rhoades, Deanna Gage-White, Gary W. White, G.K. Matheson, Vanessa Dixon
Publikováno v:
Neuropharmacology. 28(4)
It was found that gepirone and 1-(2-pyrimidinyl)-piperazine (1-PP) increased levels of corticosterone in plasma in the intact rat. Gepirone was more potent and more efficacious than its metabolite, 1-PP. The ED50 was 6.4 μmol/kg for gepirone and 65.
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::bbfe4e2ae7a65b7c61244fe3cf3b94f8
https://pubmed.ncbi.nlm.nih.gov/2747846
https://pubmed.ncbi.nlm.nih.gov/2747846
Publikováno v:
Experimental neurology. 48(2)
Spinal presynaptic inhibition is thought to be mediated by γ-aminobutyric acid (GABA). Because of the relative impermeability of GABA through the blood-brain barrier, the GABA-derivative, β(4-chlorophenyl)-GABA (Lioresal), was used to investigate p
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::08477850d7c4199680522fe51174b443
https://pubmed.ncbi.nlm.nih.gov/168097
https://pubmed.ncbi.nlm.nih.gov/168097
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