Zobrazeno 1 - 10
of 28
pro vyhledávání: '"G. W. Forsyth"'
Publikováno v:
Scopus-Elsevier
Gaspar, K. J., K. J. Racette, J. R. Gordon, M. E. Loewen, and G. W. Forsyth. Cloning a chloride conductance mediator from the apical membrane of porcine ileal enterocytes. Physiol Genomics 3: 101–111, 2000.—Attempts to attribute ileal brush-borde
Autor:
G W Forsyth, S E Gabriel
Publikováno v:
Journal of Biological Chemistry. 266:17764-17769
Conductive transport of chloride ion is important in controlling ion and fluid secretion by exocrine tissues. The current study was directed at identifying proteins in the intestinal brush-border membrane that may be involved with conductive chloride
Autor:
S E, Gabriel, G W, Forsyth
Publikováno v:
The Journal of biological chemistry. 266(27)
Conductive transport of chloride ion is important in controlling ion and fluid secretion by exocrine tissues. The current study was directed at identifying proteins in the intestinal brush-border membrane that may be involved with conductive chloride
Autor:
G W, Forsyth, S E, Gabriel
Publikováno v:
American journal of veterinary research. 51(10)
Compounds that prevent chloride transport in membrane vesicles have been tested for in vivo activity against the effects of intestinal secretory agents. Chloride channel blockers including diphenylamine-2-carboxylate, 4-acetamido-4'-isothiocyanostilb
Publikováno v:
The Journal of Membrane Biology. 96:243-249
The physiological relevance of an apparent ionophore activity of cholera toxin towards Ca2+ has been examined in several different systems designed to measure affinity, specificity, rates of ion transfer, and effects on intracellular ion concentratio
Publikováno v:
Infection and Immunity. 21:373-380
The effects of cholera toxin on mucosal cyclic nucleotide concentrations and on net fluid secretion in the porcine small intestine are reported. Cholera toxin causes net secretion of fluid into the small intestine of weanling pigs, and secretory rate
Publikováno v:
Canadian Journal of Biochemistry. 56:280-286
Cholera toxin is thought to cause intestinal secretion by activating adenylate cyclase and increasing intracellular 3′,5′-cyclic AMP concentrations in intestinal mucosa. Cholera toxin causes profuse secretion of fluid into ligated intestinal loop
Publikováno v:
American Journal of Physiology-Gastrointestinal and Liver Physiology. 241:G227-G234
The effects of several weak acids on the secretory actions of cholera toxin and the heat-stable enterotoxin of Escherichia coli (ST) have been examined in ligated jejunal loops in weanling pigs. Ascorbic and acetic acids had no effect, but L-lactic a
Publikováno v:
Canadian Journal of Physiology and Pharmacology. 57:1004-1010
The feasibility of reducing intestinal secretion by the use of agents which decrease intestinal mucosal cAMP concentration has been investigated in the weanling pig and the rabbit. Three different agents for decreasing mucosal cAMP concentration were
Publikováno v:
Canadian Journal of Physiology and Pharmacology. 58:772-777
Fluid secretory responses and associated changes in cyclic nucleotide concentration caused by the heat-stable enterotoxin of Escherichia coli (ST) have been investigated in the jejunum of weanling pigs. Effects associated with ST treatment have been