Výsledky vyhledávání - "G. S. Burgess"

Regulation of the c-jun gene in p210 BCR-ABL transformed cells corresponds with activity of JNK, the c-jun N-terminal kinase

Autor: G S, Burgess, E A, Williamson, L D, Cripe, S, Litz-Jackson, J A, Bhatt, K, Stanley, M J, Stewart, A S, Kraft, H, Nakshatri, H S, Boswell

Publikováno v: Blood. 92(7)

Activity of the c-jun N-terminal kinase (JNK) has been shown in hematopoietic cells transformed by p210 BCR-ABL. However, analysis has not been reported for hematopoietic cells on the consequences of this activity for c-jun promoter regulation within

Externí odkaz: https://explore.openaire.eu/search/publication?articleId=pmid________::bd185e152a8952c00b840ccd3cdd38c6
https://pubmed.ncbi.nlm.nih.gov/9746785

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Role for E2F1 in p210 BCR-ABL downstream regulation of c-myc transcription initiation. Studies in murine myeloid cells

Autor: M J, Stewart, S, Litz-Jackson, G S, Burgess, E A, Williamson, D S, Leibowitz, H S, Boswell

Publikováno v: Leukemia. 9(9)

Experiments were performed to elucidate the mechanism through which p210 BCR-ABL, by its downstream signals, regulates c-myc messenger RNA expression in hematopoietic cells. We studied a model system in which stable expression of p210 BCR-ABL in inte

Externí odkaz: https://explore.openaire.eu/search/publication?articleId=pmid________::327ba907549bb43403e6fdf4647c9085
https://pubmed.ncbi.nlm.nih.gov/7658719

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p210 bcr-abl confers overexpression of inosine monophosphate dehydrogenase: an intrinsic pathway to drug resistance mediated by oncogene

Autor: K, Gharehbaghi, G S, Burgess, F R, Collart, S, Litz-Jackson, E, Huberman, H N, Jayaram, H S, Boswell

Publikováno v: Leukemia. 8(8)

The p210 bcr-abl fusion protein tyrosine kinase oncogene has been implicated in the pathogenesis of chronic granulocytic leukemia (CGL). Specific intracellular functions performed by p210 bcr-abl have recently been delineated. We considered the possi

Externí odkaz: https://explore.openaire.eu/search/publication?articleId=pmid________::9dfc79eff0cce65c33f8e4965c13f1b1
https://pubmed.ncbi.nlm.nih.gov/7520100

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