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Publikováno v:
Regulatory Peptides. 163:37-42
Neurotensin (NT) is a tridecapeptide distributed in central and peripheral nervous systems, which can behave as a neurotransmitter or neuromodulator at central and peripheral levels. Herein we tested the potential effect of this peptide on quinuclidi
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::9d935785bd6717b9b973ebe9a7e50d67
https://doi.org/10.1016/j.regpep.2010.04.002
https://doi.org/10.1016/j.regpep.2010.04.002
Endobain E, a brain endogenous factor, is present and modulates NMDA receptor in ischemic conditions
Publikováno v:
Life Sciences. 78:245-252
We have isolated from rat cerebral cortex an endogenous Na(+), K(+)-ATPase inhibitor, termed endobain E, which modulates glutamatergic N-methyl-d-aspartate (NMDA) receptor. This endogenous factor allosterically decreases [(3)H]dizocilpine binding to
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::36f62d1b6c3b66f313e0017fe9d55e6a
https://doi.org/10.1016/j.lfs.2005.04.046
https://doi.org/10.1016/j.lfs.2005.04.046
Publikováno v:
Developmental Brain Research. 154:247-254
Ontogenetic studies indicate that inositol phosphate accumulation in rodent brain tissue by cholinergic muscarinic agonists as well as expression of high-affinity neurotensin receptor (NTS1) peak at 7 days after birth. Herein, potential participation
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::19adf3adb08f831852521896071f294b
https://doi.org/10.1016/j.devbrainres.2004.11.003
https://doi.org/10.1016/j.devbrainres.2004.11.003
Publikováno v:
Neurochemical Research. 29:819-825
A brain endogenous factor, termed endobain E, allosterically decreases [3H]dizocilpine binding to NMDA receptor. Such effect depends on receptor activation by the coagonists glutamate and glycine and is interfered by channel blockers, suggesting its
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_________::4b1069b044d389f07c6df4183d1fda68
https://doi.org/10.1023/b:nere.0000018856.99773.71
https://doi.org/10.1023/b:nere.0000018856.99773.71
Publikováno v:
Neurochemical Research. 28:903-910
In the search of Na+,K+-ATPase modulators, we have reported the isolation by gel filtration and HPLC of a brain fraction, termed endobain E, which highly inhibits Na+,K+-ATPase activity. In the present study we compared some properties of endobain E
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_________::1d6d1be010111bdab908c35150905c58
https://doi.org/10.1023/a:1023227510707
https://doi.org/10.1023/a:1023227510707
Publikováno v:
Developmental Brain Research. 138:167-175
The mechanism of action of an endogenous Na(+), K(+)-ATPase inhibitor, termed endobain E, on phosphoinositide hydrolysis was studied in neonatal rat brain cortex and compared with that of ouabain. Lack of additivity for endobain E and glutamate paire
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::471434a79e10df6771d329626a134411
https://doi.org/10.1016/s0165-3806(02)00469-8
https://doi.org/10.1016/s0165-3806(02)00469-8
Publikováno v:
Neurochemistry International. 39:301-310
An endogenous Na + , K + -ATPase inhibitor termed endobain E has been isolated from rat brain which shares several biological properties with ouabain. This cardiac glycoside possesses neurotoxic properties attributable to Na + , K + -ATPase inhibitio
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::f12d6eb50d76e5b890a22bb6f10f7d2b
https://doi.org/10.1016/s0197-0186(01)00034-1
https://doi.org/10.1016/s0197-0186(01)00034-1
Publikováno v:
Neurochemical Research. 26:225-230
Specific ligand binding to rat hippocampal adenosine A1 receptor after administration of the convulsant drug 3-mercaptopropionic acid (MP) was studied by means of a quantitative autoradiographic method. 2-Chloro-N6-[cyclopentyl-2,3,4,5-3H adenosine]
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_________::d7585044813c551259ded8cdb46b4694
https://doi.org/10.1023/a:1010912516299
https://doi.org/10.1023/a:1010912516299
Publikováno v:
Neurochemical Research. 26:1253-1259
The effect of an endogenous Na+, K+-ATPase inhibitor, termed endobain E, on phosphoinositide hydrolysis was studied in rat brain cortical prisms and compared with that of ouabain. As already shown for ouabain, a transient effect was obtained with end
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_________::77ddff0856b5599f1f0273780e9053ef
https://doi.org/10.1023/a:1013923608220
https://doi.org/10.1023/a:1013923608220