Zobrazeno 1 - 10
of 270
pro vyhledávání: '"G Weeda"'
Autor:
Li, Xian1 (AUTHOR) xxt0313@126.com, Li, Danyang1 (AUTHOR), Tian, Ying1 (AUTHOR), Chang, Yaqing1 (AUTHOR) yaqingchang@hotmail.com, Hao, Zhenlin1 (AUTHOR) yaqingchang@hotmail.com
Publikováno v:
Fishes (MDPI AG). Oct2024, Vol. 9 Issue 10, p389. 26p.
Publikováno v:
Medical Education, 31(2), 138-143. Wiley
During their first training period in general practice the authors felt that they did not encounter the balanced workload which is the foundation for learning to be a GP. Previous studies confirmed the existence of differences in overall and specific
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::3db24c0ed24c9afe0f1bb6761d48818e
https://doi.org/10.1111/j.1365-2923.1997.tb02473.x
https://doi.org/10.1111/j.1365-2923.1997.tb02473.x
Autor:
D. Bootsma, Aart G. Jochemsen, A. Bosch, Libin Ma, G. Weeda, A. Westbroek, A. J. Van Der Eb, J. H. J. Hoeijmakers
Publikováno v:
Molecular and Cellular Biology. 14:4126-4134
The human ERCC3 gene, which corrects specifically the nucleotide excision repair defect in human xeroderma pigmentosum group B and cross-complements the repair deficiency in rodent UV-sensitive mutants of group 3, encodes a presumed DNA helicase that
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::3ed66b51ddc966ab0131756b14e4f267
https://doi.org/10.1128/mcb.14.6.4126
https://doi.org/10.1128/mcb.14.6.4126
Publikováno v:
Nucleic Acids Research. 19:6301-6308
The human XPBC/ERCC-3 was cloned by virtue of its ability to correct the excision repair defect of UV-sensitive rodent mutants of complementation group 3. The gene appeared to be in addition implicated in the human, cancer prone repair disorder xerod
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::d9b728be472a3b154944a479fe1480ea
https://doi.org/10.1093/nar/19.22.6301
https://doi.org/10.1093/nar/19.22.6301
Publikováno v:
Carcinogenesis. 12:2361-2368
The human XPBC/ERCC-3 DNA repair gene specifically corrects the repair defect of xeroderma pigmentosum (XP) complementation group B and rodent repair mutant cell lines of group 3. The gene encodes a presumed DNA- and chromatin-binding helicase involv
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::12ce9391af9578f9bd74dda6e770da24
https://doi.org/10.1093/carcin/12.12.2361
https://doi.org/10.1093/carcin/12.12.2361
Autor:
H. Odijk, A. J. Van Der Eb, G. Weeda, D. Bootsma, J. H. J. Hoeijmakers, Andries Westerveld, R. Masurel, J. de Wit, R. C. A. Van Ham
Publikováno v:
Molecular and Cellular Biology. 10:2570-2581
In this report we present the cloning, partial characterization, and preliminary studies of the biological activity of a human gene, designated ERCC-3, involved in early steps of the nucleotide excision repair pathway. The gene was cloned after genom
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::978ca99e87c31dcc546b3ff4dd647c62
https://doi.org/10.1128/mcb.10.6.2570-2581.1990
https://doi.org/10.1128/mcb.10.6.2570-2581.1990
Autor:
J, de Boer, H, van Steeg, R J, Berg, J, Garssen, J, de Wit, C T, van Oostrum, R B, Beems, G T, van der Horst, C F, van Kreijl, F R, de Gruijl, D, Bootsma, J H, Hoeijmakers, G, Weeda
Publikováno v:
Cancer research. 59(14)
Patients with the nucleotide excision repair (NER) disorder xeroderma pigmentosum (XP) are highly predisposed to develop sunlight-induced skin cancer, in remarkable contrast to photosensitive NER-deficient trichothiodystrophy (TTD) patients carrying
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=pmid________::23505fb256a094f0682be13c27176cbd
https://pubmed.ncbi.nlm.nih.gov/10416615
https://pubmed.ncbi.nlm.nih.gov/10416615
Autor:
G, Weeda, J, de Boer, I, Donker, J, de Wit, S B, Winkler, G T, van der Horst, W, Vermeulen, D, Bootsma, J H, Hoeijmakers
Publikováno v:
Recent results in cancer research. Fortschritte der Krebsforschung. Progres dans les recherches sur le cancer. 154
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=pmid________::c46c0da55b884cefd5afaf6e7291c2b6
https://pubmed.ncbi.nlm.nih.gov/10026997
https://pubmed.ncbi.nlm.nih.gov/10026997
Publikováno v:
Environmental and molecular mutagenesis. 31(1)
To study the influence of nucleotide excision repair (NER) on mutagenesis in vivo, ERCC1 +/-, XPA-/-, and wild-type (ERCC1+/+ and XPA+/+, respectively) lambda lacZ-transgenic mice were treated i.p. with N-hydroxy-2-acetylaminofluorene (N-OH-AAF) and
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=pmid________::19689079783b03d5f950648712ee9381
https://pubmed.ncbi.nlm.nih.gov/9464314
https://pubmed.ncbi.nlm.nih.gov/9464314
Autor:
G, Weeda, J H, Hoeijmakers
Publikováno v:
Seminars in cancer biology. 4(2)
Nucleotide excision repair is a versatile process and is one of the best known systems that prevents the deleterious consequences of DNA damage induced by environmental agents and cellular metabolites. Without repair, persisting lesions can interfere
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=pmid________::d712fe33c2e88f843b56855ba24dbb11
https://pubmed.ncbi.nlm.nih.gov/8513146
https://pubmed.ncbi.nlm.nih.gov/8513146