Zobrazeno 1 - 10
of 14
pro vyhledávání: '"G R Jalali"'
Autor:
Louise Harewood, G R Jalali, Christine J. Harrison, Rachel L. Harris, Chris Mitchell, Anthony V. Moorman, N. Sumption, Hazel M. Robinson, M Martineau, Susan M. Richards, M. S. Jabbar Al-Obaidi
This study identifies multiple copies of the AML1 gene on a duplicated chromosome 21, dup(21), as a recurrent abnormality in acute lymphoblastic leukemia (ALL). Clusters of AML1 signals were visible at interphase by fluorescence in situ hybridization
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::e2c1397503a146d74c4936fef16a647e
https://doi.org/10.1038/sj.leu.2402849
https://doi.org/10.1038/sj.leu.2402849
Autor:
Anthony M. Ford, G R Jalali, Mel Greaves, Richard F. Stevens, Christine J. Harrison, M Martineau
Publikováno v:
Leukemia. 17:993-995
Publikováno v:
2007 IEEE International Conference on Industrial Engineering and Engineering Management.
The direct application of the theory of constraints (TOC) in project management is known as critical chain project management (CCPM) which is introduced by Goldratt. It is considered to be one of the popular approaches to project management. One of t
Publikováno v:
Genetic counseling (Geneva, Switzerland). 18(3)
A de novo complex chromosome rearrangement (CCR) found in a phenotypically abnormal boy was characterized by G-bands, FISH with subtelomere probes, and M-FISH. The G-banding analysis revealed involvement of chromosomes 1, 2, 3, 15, and 18 with (at le
Autor:
Qian An, Christine J. Harrison, Helen Worley, Sarah L. Wright, M Martineau, Zoe J. Konn, Jonathan C. Strefford, G R Jalali
Publikováno v:
Leukemia. 22(1)
We describe four cases of childhood B-cell progenitor acute lymphoblastic leukaemia (BCP-ALL) and one of T-cell (T-ALL) with unexpected numbers of interphase signals for ETV6 with an ETV6–RUNX1 fusion probe. Three fusion negative cases each had a t
Autor:
Kan Luk Cheung, Zoë J. Broadfield, Kerry E. Barber, G R Jalali, Anthony V. Moorman, Hazel M. Robinson, Jonathan C. Strefford, Rachel L. Harris, Christine J. Harrison, M Martineau
Publikováno v:
Leukemia. 19(4)
Deletions from the derivative chromosome 9, der(9), of the translocation, t(9;22)(q34;q11), at the site of the ABL/BCR fusion gene, have been demonstrated by fluorescence in situ hybridisation (FISH), in both Philadelphia chromosome (Ph)-positive chr
Autor:
Jonathan C. Strefford, Monica Stewart, Hazel M. Robinson, S Rutherford, Kerry E. Barber, G R Jalali, Christine J. Harrison, T D Allen, Kan Luk Cheung, Rachel L. Harris, Zoë J. Broadfield, M Martineau, Anthony V. Moorman, Ewan R. Cameron, Louise Harewood
Publikováno v:
Leukemia. 18(6)
Autor:
C. F. Bennett, Christine J. Harrison, Louise Harewood, Kathryn G. Roberts, Anthony H. Goldstone, G R Jalali, Susan M. Richards, H. G. Prentice, M. S. Jabbar Al-Obaidi, M Martineau, Ian M. Franklin
Publikováno v:
Leukemia. 16(4)
Dual-color interphase fluorescence in situ hybridization (FISH) with ETV6 and AML1 probes was used for the first time on a series of 159 adult patients with acute lymphoblastic leukemia (ALL), for detection of the t(12;21)(p13;q22) translocation. Sev
Autor:
R Codrington, Christine J. Harrison, SM Hart, H. G. Prentice, Letizia Foroni, G R Jalali, A. V. Hoffbrand, Michael Potter, H E O'Connor, Paola Carrara, Mary Papaioannou
Publikováno v:
British journal of haematology. 111(4)
The t(12;21)(p13;q22) translocation, resulting in the fusion of the ETV6 and AML1 genes, occurs in 20-25% of paediatric B-lineage acute lymphoblastic leukaemias (ALL). The identification of the fusion product has important prognostic and therapeutic
Autor:
Louise Harewood, Christine J. Harrison, Kan Luk Cheung, G R Jalali, Zoë J. Broadfield, Susan M. Richards, Chris Mitchell, Anthony V. Moorman, M Martineau, Rachel L. Harris, Kerry E. Taylor, Hazel M. Robinson
Publikováno v:
Leukemia. 17:2249-2250
A total of 28 children and nine adults with relapsed T-ALL were analyzed for the configuration of their T-cell receptor (TCR) and TAL1 genes at diagnosis and relapse to evaluate their stability throughout the disease course. A total of 150 clonal TCR