Zobrazeno 1 - 4
of 4
pro vyhledávání: '"G L Ivey"'
Publikováno v:
The Yale Journal of Biology and Medicine
In normal individuals, the main route for tyrosine degradation is the hepatic pathway tyrosine→4-hydroxyphenylpyruvic acid→homogentisic acid→CO2. Quantitatively minor pathways, in large part extrahepatic, are: tyrosine→tyramine→octopamine a
Autor:
B M, Nordlinger, J T, Fulenwider, G L, Ivey, B A, Faraj, F M, Ali, M, Kutner, J M, Henderson, D, Rudman
Publikováno v:
The Journal of laboratory and clinical medicine. 94(6)
The aim of this study was to define the enzyme defect responsible for tyrosinemia in cirrhotic patients. The principal hepatic degradation pathway for tyrosine, tyrosine leads to p-hydroxyphenylpyruvic acid equilibrium homogentisic acid leads to CO2
Autor:
D Rudman, Bernard M. Nordlinger, G L Ivey, Robert D. Jansen, Farouk M. Ali, Michael Kutner, J T Fulenwider, Bahjat A. Faraj, Vernon M. Camp, E B Rypins, F. Schmidt
Hypertyraminemia is common in hepatic cirrhosis and correlates in severity with encephalopathy. The mechanism of cirrhotic hypertyraminemia has not been established. The alternative possibilities are increased production from tyrosine and impaired de
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::a289a07ea55dff07e4a286d25b12f9d5
https://europepmc.org/articles/PMC372134/
https://europepmc.org/articles/PMC372134/
Publikováno v:
Gastroenterology. 78(5 Pt 1)
Physiologic similarities between cirrhotic and septic patients have implicated systemic endotoxemia as a possible mediator of the hemodynamic, neurologic, and hematologic complications observed in patients with cirrhosis of the liver. The recently re