Zobrazeno 1 - 9
of 9
pro vyhledávání: '"G J, Brunn"'
Autor:
J C, Lawrence, G J, Brunn
Publikováno v:
Progress in molecular and subcellular biology. 26
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=pmid________::d0f9ae73123e6545676bf04f8f4a3380
https://pubmed.ncbi.nlm.nih.gov/11575163
https://pubmed.ncbi.nlm.nih.gov/11575163
Publikováno v:
Progress in cell cycle research. 1
The immunosuppressant drug, rapamycin (RAP), is a potent inhibitor of IL-2-dependent T-cell proliferation. The antiproliferative effect of RAP is mediated through the formation of an active complex with its cytosolic receptor protein, FKBP12. The mol
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=pmid________::1bd92b326afa6f7fc6e0ce0f0a058092
https://pubmed.ncbi.nlm.nih.gov/9552353
https://pubmed.ncbi.nlm.nih.gov/9552353
Publikováno v:
Advances in experimental medicine and biology. 365
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=pmid________::c5b742516465ed3a138a3512dc72feef
https://pubmed.ncbi.nlm.nih.gov/7887304
https://pubmed.ncbi.nlm.nih.gov/7887304
Publikováno v:
The Journal of biological chemistry. 268(30)
The immunosuppressant rapamycin (RAP) is a potent inhibitor of the entry of interleukin (IL)-2-stimulated T cells into S-phase. Earlier results indicated that RAP treatment arrested the growth of the murine IL-2-dependent T cell line CTLL-2 in late G
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=pmid________::1cf2a4e80b339ab26a2f59a517993457
https://pubmed.ncbi.nlm.nih.gov/8226784
https://pubmed.ncbi.nlm.nih.gov/8226784
Publikováno v:
The Journal of biological chemistry. 268(5)
The macrolide rapamycin (RAP) is a potent inhibitor of interleukin-2 (IL-2)-induced T-cell proliferation. Current models suggest that RAP, when complexed to its intracellular receptor, FK506-binding protein, interferes with an IL-2 receptor-coupled s
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=pmid________::a0e7fe382b9d39f9b36dd2c304ab089b
https://pubmed.ncbi.nlm.nih.gov/8429048
https://pubmed.ncbi.nlm.nih.gov/8429048
Publikováno v:
The Journal of pharmacology and experimental therapeutics. 260(3)
The effect of drug-specific antibody Fab' fragment on desipramine (DMI) toxicity was studied in anesthetized rats to determine 1) whether DMI-induced hypotension can be reversed, and 2) whether the effect of this Fab' fragment can be enhanced by the
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=pmid________::b178ce5db50d28145c83b2c6ad098965
https://pubmed.ncbi.nlm.nih.gov/1312169
https://pubmed.ncbi.nlm.nih.gov/1312169
Publikováno v:
Drug metabolism and disposition: the biological fate of chemicals. 19(1)
A monoclonal antibody was used to study the dose-response relationship for antibody-mediated redistribution of tricyclic antidepressants (TCA) in rats. The antibody (anti-TCA) was an IgG1 with Ka = 3.0 x 10(8) M-1 for desipramine (DMI) and 2.2 x 10(8
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=pmid________::dcdd963fde3c598ffa281476ad1e31d2
https://pubmed.ncbi.nlm.nih.gov/1673407
https://pubmed.ncbi.nlm.nih.gov/1673407
Publikováno v:
Annals of Emergency Medicine. 20:1083
The effect of a drug-specific antibody on desipramine (DMI) cardiotoxicity was studied in rats. Animals were pretreated i.v. with 4.2 g/kg of a monoclonal antibody (anti-TCA) followed by DMI HCl 30 mg/kg i.p. (molar ratio of anti-TCA binding sites to
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::03198b07aa80c7bf3e1002bbab680cf3
https://doi.org/10.1016/s0196-0644(05)81358-7
https://doi.org/10.1016/s0196-0644(05)81358-7
Publikováno v:
Annals of Emergency Medicine. 19:629
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_________::e3cae889680979491261cae4fc600827
https://doi.org/10.1016/s0196-0644(05)82455-2
https://doi.org/10.1016/s0196-0644(05)82455-2