Zobrazeno 1 - 10
of 11
pro vyhledávání: '"Funda F. Bölükbaşı Hatip"'
Autor:
Shinji Ouma, Yoshio Tsuboi, Takayasu Mishima, Yoichi Matsunaga, Izzettin Hatip-Al-Khatib, Shinsuke Fujioka, Hiromu Ogura, Midori Suenaga, Funda F. Bölükbaşı Hatip
Publikováno v:
eNeurologicalSci
eNeurologicalSci, Vol 25, Iss, Pp 100369-(2021)
eNeurologicalSci, Vol 25, Iss, Pp 100369-(2021)
Background and purpose There is sufficient evidence to support vitamin D's noncalcemic effects and the role of vitamin D deficiency in the development of a wide range of neurological disorders. This study aimed to evaluate whether serum 25(OH)D and 1
Autor:
Izzettin Hatip-Al-Khatib, Funda F. Bölükbaşı Hatip, Junichi Matsumoto, Fuyuko Takata, Takashi Machida, Yasufumi Kataoka, Shinya Dohgu, Atsushi Yamauchi
Publikováno v:
Brain Research. 1692:34-44
Interleukin (IL)-6 is an important mediator of neurovascular dysfunction, neurodegeneration and/or neuroinflammation. We previously reported that brain pericytes released higher levels of IL-6 than did glial cells (astrocytes and microglia) in respon
Autor:
Atsushi Yamauchi, Öznur Açikalin, Izzettin Hatip-Al-Khatib, Funda F. Bölükbaşı Hatip, Yasufumi Kataoka, Ruyal F Tan
Nicotinic acetylcholine receptors (nAChRs) are implicated in the pathogenesis of Parkinson's disease (PD). Varenicline tartrate is a partial agonist at ?4ß2 and full agonist at ?7 neuronal nAChR subunits. A unilateral lesion of the substantia nigra
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::8732f5c1fc8d08e5e7cf9cb229d499d1
http://acikerisim.pau.edu.tr:8080/xmlui/handle/11499/10973
http://acikerisim.pau.edu.tr:8080/xmlui/handle/11499/10973
Autor:
Kotaro Takasaki, Izzettin Hatip-Al-Khatib, Kaori Kubota, Katsunori Iwasaki, Funda F. Bölükbaşı Hatip, Risako Fujikawa, Ai Nogami-Hara, Nobuaki Egashira, Shutaro Katsurabayashi, Takuya Watanabe, Masaki Nagao
Ethnopharmacological relevance Japanese Angelica acutiloba root (Angelica root) is included in several Kampo medicines including Yokukansan (YKS). Angelica root and YKS are used for the treatment of a variety of psychological and neurodegenerative di
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::3771c8d5ca96911db18dd35a2c655c05
https://doi.org/10.1016/j.jep.2017.12.025
https://doi.org/10.1016/j.jep.2017.12.025
Autor:
Yoichi Matsunaga, Izzettin Hatip-Al-Khatib, Yoshio Tsuboi, Midori Suenaga, Funda F. Bölükbaşı Hatip, Shinji Ouma
Publikováno v:
Brain and Behavior
Objectives: To determine the relevance of Mini-Mental State Examination (MMSE), serum 25-hydroxyvitamin D (25(OH)D3), and 1,25(OH)2D3 concentrations to mild cognitive impairment (MCI) and various stages of Alzheimer's disease (AD). Materials and Meth
Publikováno v:
Life Sciences. 92:228-236
Aims Alzheimer's disease (AD) is characterized by vascular dysfunction, in addition to memory impairment. Previously we found that β-sheet breaker peptides (βSBPs) improved memory impairment induced by amyloid β-peptide Aβ40. In this study we inv
Publikováno v:
Current Alzheimer Research. 7:602-614
Amyloid β-protein (Aβ) assembly into toxic fibrillar structures is seminal in development of senile plaques, the pathological hallmark of Alzheimers disease. Blocking this process could have a therapeutic value. β-sheet breaker peptides (βSBP) de
Publikováno v:
British Journal of Pharmacology. 158:1165-1172
Background and purpose: Aggregates of the protein amyloid-beta (Aβ) play a crucial role in the pathogenesis of Alzheimer's disease (AD). Most therapeutic approaches to AD do not target Aβ, so determination of the factor(s) that facilitate aggregati
Publikováno v:
Pharmacology. 97(5-6)
Altered vascular reactivity due to endothelial dysfunction, consequent to vascular damage, is observed in rheumatoid arthritis. We investigated the effect of angiotensin (Ang)-(1-7) on vasculature changes in arthritis induced by complete Freund's adj
Autor:
Saadettin Eskicorapci, Ali Ersin Zumrutbas, Ismail Cenk Acar, Funda F. Bölükbaşı Hatip, Vural Kucukatay, Mehmet Babaoglu, Zafer Aybek
Purpose: In this study, gap junction expression and the effects of estrogen deficiency and gap junction inhibitors were investigated in overactive bladder models which were created by bladder outlet obstruction. Methods: In our study, we created four
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::91e92576c7c302d1c365bf9a8f003c6a
http://acikerisim.pau.edu.tr:8080/xmlui/handle/11499/8038
http://acikerisim.pau.edu.tr:8080/xmlui/handle/11499/8038