Zobrazeno 1 - 10
of 35
pro vyhledávání: '"Fumiyo Matsuda"'
Autor:
Rika Yatsushiro, Fumiyo Matsuda, Bo Causer, Kazuhiro Inoue, Junne Kikata, Naoki Miura, Kumiko Hashimoto, Yasuaki Akasaki, Keiko Unedaya
Publikováno v:
Frontiers in Education, Vol 9 (2024)
IntroductionThe Mishima Islands in Kagoshima Prefecture, Japan, are remote islands designated as medically underserved. Supporting the people living in such remote islands requires a multifaceted examination of the livelihoods of the residents, inclu
Externí odkaz:
https://doaj.org/article/0d291fe2a40d4fdc8750cbb2799dc17f
Autor:
Harutoshi Sakakima, Kosei Ijiri, Fumiyo Matsuda, Hiroyuki Tominaga, Takanori Biwa, Kazunori Yone, Yoshiyuki Sankai
Publikováno v:
Case Reports in Orthopedics, Vol 2013 (2013)
Externí odkaz:
https://doaj.org/article/1f411442239e469e9d8eeff97df58a02
Publikováno v:
British Journal of Pharmacology
Midkine (MK) is a growth factor with neurotrophic and neurite outgrowth activities. It was expressed in the peri-ischaemic area in the acute phase of cerebral infarction in rat brains. Astrocytes were the origin of MK in this occasion. MK has been as
Autor:
Ko-ichi Kawahara, Shinichiro Arimura, Fumiyo Matsuda, Ryuichi Maenosono, Yuko Nawa, Yoko Morimoto, Noboru Arimura, Minoru Shigemori, Kiyoshi Kikuchi, Xiaojie Meng, Binita Shrestha, Kazunori Takenouchi, Masahiro Iwata, Teruto Hashiguchi, Ikuro Maruyama, Yoshiko Ohno, Terukazu Kuramoto, Hisayo Sameshima, Kentaro Mera, Takashi Ito, Yutaka Tajima, Salunya Tancharoen, Yoko Oyama, Yoshihiro Yoshida, Kenji Nakayama, Naoki Miura, Kamal Krishna Biswas, Hisaaki Uchikado
Publikováno v:
Biochemical and Biophysical Research Communications. 390:1121-1125
Aquaporin-4 (AQP4) plays a role in the generation of post-ischemic edema. Pharmacological modulation of AQP4 function may thus provide a novel therapeutic strategy for the treatment of stroke, tumor-associated edema, epilepsy, traumatic brain injury,
Autor:
Avtar K. Singh, Balasubramaniam Annamalai, Fumiyo Matsuda, Inderjit Singh, Tejbir Singh Dhindsa, Tajinder S Dhammu, Mushfiquddin Khan
Publikováno v:
Brain research. 1630
Traumatic brain injury (TBI) derails nitric oxide (NO)-based anti-inflammatory and anti-excitotoxicity mechanisms. NO is consumed by superoxide to form peroxynitrite, leading to decreased NO bioavailability for S-nitrosoglutathione (GSNO) synthesis a
Autor:
Megumi Sakasegawa, Kosei Ijiri, Takao Setoguchi, Satoshi Yamasaki, Lin Xu, Fumiyo Matsuda, Hirofumi Shimada, Miguel Otero, Akihide Tanimoto, Setsuro Komiya, Harutoshi Sakakima, Mary B. Goldring, Kaneyuki Tsuchimochi
Publikováno v:
Pathology, research and practice. 212(4)
Osteoarthritis (OA) is a whole joint disease characterized by cartilage degradation, which causes pain and disability in older adults. Our previous work showed that growth arrest and DNA damage-inducible protein 45 β (GADD45β) is upregulated in cho
Autor:
Mauhammad Baarine, Mushfiquddin Khan, Tajinder S Dhammu, Tejbir Singh Dhindsa, Inderjit Singh, Fumiyo Matsuda, Avtar K. Singh
Publikováno v:
Drug Design, Development and Therapy
Mushfiquddin Khan,1 Tajinder S Dhammu,1 Fumiyo Matsuda,1,2 Mauhammad Baarine,3 Tejbir Singh Dhindsa,1 Inderjit Singh,1 Avtar K Singh3,4 1Department of Pediatrics, Medical University of South Carolina, Charleston, SC, USA; 2School of Health Sciences,
Publikováno v:
BMC Neuroscience
Background Stroke immediately sets into motion sustained excitotoxicity and calcium dysregulation, causing aberrant activity in neuronal nitric oxide synthase (nNOS) and an imbalance in the levels of nitric oxide (NO). Drugs targeting nNOS-originated
Autor:
Harutoshi Sakakima, Kosei Ijiri, Tomomi Kamizono, Yoshihiro Yoshida, Fumiyo Matsuda, Kimiko Izumo
Publikováno v:
Acta Histochemica. 108:357-364
Midkine (MK) is a multifunctional cytokine and heparin-binding growth factor with neurotrophic activity. MK and its receptor were examined for up to 14 days in a chemically injured rat muscle regeneration process caused by the injection of bupivacain
Publikováno v:
Stroke. 45
Background: In stroke patients, the stimulation of neurorepair mechanisms is necessary to reduce morbidity and disability. Our studies on brain and spinal cord trauma show that an exogenous treatment with the S-nitrosylating agent S-nitrosoglutathion