Zobrazeno 1 - 10
of 29
pro vyhledávání: '"Fumiko Honda"'
Autor:
Iki Sonn, Fumiko Honda-Ozaki, Sho Yoshimatsu, Satoru Morimoto, Hirotaka Watanabe, Hideyuki Okano
Publikováno v:
Inflammation and Regeneration, Vol 42, Iss 1, Pp 1-20 (2022)
Abstract Background Microglia are innate immune cells that are the only residential macrophages in the central nervous system. They play vital physiological roles in the adult brain and during development. Microglia are particularly in the spotlight
Externí odkaz:
https://doaj.org/article/d5e6ffb2bec6455b8fe94669a3b35aba
Autor:
Naoya Kase, Madoka Terashima, Akira Ohta, Akira Niwa, Fumiko Honda‐Ozaki, Yuri Kawasaki, Tatsutoshi Nakahata, Nobuo Kanazawa, Megumu K. Saito
Publikováno v:
Stem Cells Translational Medicine, Vol 10, Iss 3, Pp 455-464 (2021)
Abstract Nakajo‐Nishimura syndrome (NNS) is an autoinflammatory disorder caused by a homozygous mutations in the PSMB8 gene. The administration of systemic corticosteroids is partially effective, but continuous treatment causes severe side effects.
Externí odkaz:
https://doaj.org/article/ab2d0a8881024d599625298f42662775
Publikováno v:
Inflammation and Regeneration, Vol 39, Iss 1, Pp 1-8 (2019)
Abstract Nakajo-Nishimura syndrome is a proteasome-associated autoinflammatory syndrome with a distinct homozygous mutation in the PSMB8 gene encoding an inducible β5i subunit of the immunoproteasome. Although it is considered that immunoproteasome
Externí odkaz:
https://doaj.org/article/2ea82fdf34084182ab8af4c58321ae06
Autor:
Masakatsu D Yanagimachi, Akira Niwa, Takayuki Tanaka, Fumiko Honda-Ozaki, Seiko Nishimoto, Yuuki Murata, Takahiro Yasumi, Jun Ito, Shota Tomida, Koichi Oshima, Isao Asaka, Hiroaki Goto, Toshio Heike, Tatsutoshi Nakahata, Megumu K Saito
Publikováno v:
PLoS ONE, Vol 8, Iss 4, p e59243 (2013)
Monocytic lineage cells (monocytes, macrophages and dendritic cells) play important roles in immune responses and are involved in various pathological conditions. The development of monocytic cells from human embryonic stem cells (ESCs) and induced p
Externí odkaz:
https://doaj.org/article/7c2afde2d3c24a6297aa184f80948609
Autor:
Nobuo Kanazawa, Naoya Kase, Fumiko Honda-Ozaki, Madoka Terashima, Megumu K. Saito, Yuri Kawasaki, Akira Ohta, Tatsutoshi Nakahata, Akira Niwa
Publikováno v:
Stem Cells Translational Medicine
Stem Cells Translational Medicine, Vol 10, Iss 3, Pp 455-464 (2021)
Stem Cells Translational Medicine, Vol 10, Iss 3, Pp 455-464 (2021)
Nakajo‐Nishimura syndrome (NNS) is an autoinflammatory disorder caused by a homozygous mutations in the PSMB8 gene. The administration of systemic corticosteroids is partially effective, but continuous treatment causes severe side effects. We previ
Autor:
Iki Sonn, Fumiko Honda-Ozaki, Sho Yoshimatsu, Satoru Morimoto, Hirotaka Watanabe, Hideyuki Okano
Publikováno v:
Inflammation and regeneration. 42(1)
Background Microglia are innate immune cells that are the only residential macrophages in the central nervous system. They play vital physiological roles in the adult brain and during development. Microglia are particularly in the spotlight because m
Publikováno v:
Inflammation and Regeneration, Vol 39, Iss 1, Pp 1-8 (2019)
Inflammation and Regeneration
Inflammation and Regeneration
Nakajo-Nishimura syndrome is a proteasome-associated autoinflammatory syndrome with a distinct homozygous mutation in the PSMB8 gene encoding an inducible β5i subunit of the immunoproteasome. Although it is considered that immunoproteasome dysfuncti
Autor:
Yuri Kawasaki, Akira Ohta, Naoya Kase, Tatsutoshi Nakahata, Akira Niwa, Fumiko Honda-Ozaki, Megumu K. Saito, Madoka Terashima, Nabuo Kanazawa
Nakajo-Nishimura syndrome (NNS) is an autoinflammatory disorder caused by a homozygous mutations in PSMB8 gene. The administration of systemic corticosteroids is partially effective, but continuous treatment causes severe side effects. We previously
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_________::d1a73f9e856347ac1143d2ffd031a8ba
https://doi.org/10.1101/2020.06.01.113894
https://doi.org/10.1101/2020.06.01.113894
Autor:
Fumiko Honda-Ozaki, Haruna Ito, Hongmei Lisa Li, Nobuo Kanazawa, Isao Asaka, Ayako Nagahashi, Tatsutoshi Nakahata, Fukumi Furukawa, Akitsu Hotta, Yuri Kawasaki, Megumu K. Saito, Masakatsu Yanagimachi, Norikazu Saiki, Koichi Igura, Madoka Terashima, Akira Niwa
Publikováno v:
Stem Cell Reports
Summary Nakajo-Nishimura syndrome (NNS) is an immunoproteasome-associated autoinflammatory disorder caused by a mutation of the PSMB8 gene. Although dysfunction of the immunoproteasome causes various cellular stresses attributed to the overproduction
Publikováno v:
The Journal of Dermatology. 46