Zobrazeno 1 - 10
of 23
pro vyhledávání: '"Fumiko DATE"'
Autor:
Yuko Omori, Yusuke Ono, Takashi Kokumai, Fumiko Date, Ryota Higuchi, Goro Honda, Yusuke Mizukami, Takanori Morikawa, Michiaki Unno, Toru Furukawa
Publikováno v:
Cancer Research. 82:A081-A081
Objective: Morphological subtyping of intraductal papillary mucinous neoplasms of the pancreas (IPMNs) into pancreatobiliary-type, intestinal-type, or gastric-type is a prognostic factor, however, molecular foundations explaining the difference of cl
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_________::6d4557b62c6f2ed49a13273eacf698e7
https://doi.org/10.1158/1538-7445.panca22-a081
https://doi.org/10.1158/1538-7445.panca22-a081
Publikováno v:
Autoimmunity. 46:231-237
Although costimulatory molecules have been shown to play crucial roles in the immune response, their involvement in the pathogenesis of Sjögren's syndrome is incompletely understood. In this study, we evaluated the relationship between the severity
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::be3d9627a58c9efad7a9b351a2c38e91
https://doi.org/10.3109/08916934.2012.757307
https://doi.org/10.3109/08916934.2012.757307
Autor:
Takashi Sawai, Hiroshi Miki, Kohei Yamauchi, Shinobu Sato, Noriyuki Iwama, Masahiro Yamashita, Tohru Takahashi, Naoko Shibata, Fumiko Date, Masao Ono
Publikováno v:
Human Pathology. 40:1553-1563
Summary An abnormal healing process is believed to be involved in lung tissue damage in pulmonary fibrosis. Lymphangiogenesis has been determined to play a role in structural remodeling in diffuse alveolar damage. However, the mechanism of lymphangio
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::3a9823262e9d86a4c786ab6229fcd0ca
https://doi.org/10.1016/j.humpath.2009.03.021
https://doi.org/10.1016/j.humpath.2009.03.021
Autor:
Kazumichi Furuyama, Yotaro Shinozawa, Kazunobu Ishikawa, Satoru Yokoyama, Fumiko Date, Shigeki Shibahara, Masao Ono, Kazuhisa Takeda, Feng Han
Publikováno v:
Journal of Biochemistry. 147:143-151
Heme oxygenase (HO) catalyzes oxidative breakdown of heme, and constitutes two isozymes, HO-1 and HO-2. Here, we explored the tissue-specific regulation of expression of HO-1 and HO-2 under hypoxemia. There was no significant change in the overall ex
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::7cd28900006deb1847a6fe113f1f5fcf
https://doi.org/10.1093/jb/mvp153
https://doi.org/10.1093/jb/mvp153
Autor:
Shinobu Sato, Kohei Yamauchi, Juha Risteli, Fumiko Date, Masao Ono, Noriyuki Iwama, Ryoji Chiba, Tohru Takahashi, Masahiro Yamashita, Naoko Shibata, Hiroyuki Kumagai
Publikováno v:
Human Pathology. 40:1278-1287
There is limited information regarding the process of tissue remodeling in fibroblastic foci associated with idiopathic pulmonary fibrosis. The aim of this study was to identify the different pathologic stages of tissue remodeling in fibroblastic foc
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::0830adb7739d05b51693c2e2f0e53f55
https://doi.org/10.1016/j.humpath.2009.01.014
https://doi.org/10.1016/j.humpath.2009.01.014
Publikováno v:
Annals of the Rheumatic Diseases. 66(2):242-245
Background: The incidence and characteristics of spontaneous ankylosis in the ankle of specific F 1 mice descended from two Fas -deficient strains were reported. Here the coincidence of synovial proliferation and ankylosis in the descendent F 2 mice
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::99fdd0976bfe4896bd9b58a804d61c4b
http://hdl.handle.net/10097/51627
http://hdl.handle.net/10097/51627
Autor:
Masao Ono, Naoko Tanda, Mingcai Zhang, Fumiko Date, Shiro Mori, Hiroshi Furukawa, Masato Nose
Publikováno v:
Annals of the Rheumatic Diseases. 65(10):1273-1278
Background: The spontaneous onset of macroscopic arthropathy in the ankle of the particular F 1 mice descended from two Fas -deficient strains of mice; a mutant substrain of MRL/Mp. Fas lpr (MRL/rpl) and C3H/He. Fas lpr (C3H/lpr) was recently observe
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::a29d037a9f3d94da297d868c97959405
http://hdl.handle.net/10097/51628
http://hdl.handle.net/10097/51628
Autor:
Shigeki Shibahara, Kazumichi Furuyama, Takayuki Masuda, Wataru Hida, Hayato Matsumoto, Tomomi Kitamuro, Kazunobu Ishikawa, Hiromasa Ogawa, Kazuhiro Ogawa, Kazuhisa Takeda, Fumiko Date, Yukio Maruyama, Yongzhao Zhang, Tetsuya Adachi
Publikováno v:
Biochemical and Biophysical Research Communications. 320:514-522
Heme oxygenase (HO) catalyzes physiological heme degradation and consists of two structurally related isozymes, HO-1 and HO-2. Here we show that HO-2-deficient (HO-2−/−) mice exhibit hypoxemia and hypertrophy of the pulmonary venous myocardium as
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::607908a7e124985f19ae08882b44abcf
https://doi.org/10.1016/j.bbrc.2004.05.195
https://doi.org/10.1016/j.bbrc.2004.05.195
Autor:
Andrew D. Darnel, Chika Kaneko, Takuya Moriya, Masao Nakabayashi, Fumiko Date, Kazuhito Totsune, Kazuhiro Takahashi, Takashi Suzuki, Hironobu Sasano, Junji Takeyama, Yasunari Muramatsu
Publikováno v:
Molecular and Cellular Endocrinology. 205:43-50
Orexin-A is a neuropeptide present in the brain and is known to regulate feeding and sleeping. In this study, we examined the systemic distribution of orexin-A in human tissues. Immunoreactivity for orexin-A was detected in ganglion cells of the thor
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::7553f7491878d23cae09fd37417df90f
https://doi.org/10.1016/s0303-7207(03)00206-5
https://doi.org/10.1016/s0303-7207(03)00206-5
Publikováno v:
Autoimmunity. 48(4)
Ankylosis is a major pathological manifestation of spondyloarthropathy. The aim of this study was to evaluate the effects of anti-IL-17 therapy on spontaneous ankylosing enthesitis in mice. In this study, we used male DBA/1 mice as a spontaneous anky
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::856ea32576701f01dd8157e4c7434829
https://pubmed.ncbi.nlm.nih.gov/25352178
https://pubmed.ncbi.nlm.nih.gov/25352178