Zobrazeno 1 - 10
of 38
pro vyhledávání: '"Fumi Kuriiwa"'
Publikováno v:
Journal of Pharmacological Sciences, Vol 146, Iss 1, Pp 29-32 (2021)
Hydroxyl radical (•OH) production in the rat striatum during carbon monoxide (CO) poisoning, which inhibits complex IV, was enhanced synergistically by malonate, a mitochondrial complex II inhibitor, but not N-methyl-4-phenylpyridinium or NaCN, com
Externí odkaz:
https://doaj.org/article/92f67a62b83a49b9a7b6593aba4cd805
Publikováno v:
Genomics Data, Vol 12, Iss C, Pp 74-75 (2017)
Carbon monoxide (CO) poisoning causes brain damage, which is attenuated by treatment with hydrogen [1,2], a scavenger selective to hydroxyl radical (·≡OH) [3]. This suggests a role of ·≡OH in brain damage due to CO poisoning. Studies have shown
Externí odkaz:
https://doaj.org/article/98e67c316a764200a5a5385d9b8e46d5
Publikováno v:
Journal of Pharmacological Sciences, Vol 104, Iss 1, Pp 90-93 (2007)
Extracellular cGMP in the striatum of rats exposed to 3000 ppm carbon monoxide (CO) or 8% O2 was decreased during the early period of exposure. Thereafter, extracellular cGMP in rats exposed to CO, but not 8% O2, was transiently increased. A nitric o
Externí odkaz:
https://doaj.org/article/3ff2bd2faf884431add46ac2e1392362
Publikováno v:
Scientific Reports, Vol 10, Iss 1, Pp 1-10 (2020)
Scientific Reports
Scientific Reports
Oxidative stress has been suggested to play a role in brain damage during carbon monoxide (CO) poisoning. Severe poisoning induced by CO at 3000 ppm, but not 1000 ppm, enhances hydroxyl radical (˙OH) production in the rat striatum, which might be me
Publikováno v:
Toxicology. 394
Severe poisoning induced by carbon monoxide (CO) at 3000 ppm, but not 1000 ppm, enhances hydroxyl radical ( OH) production in rat striatum, which is greatly susceptible to inhibitors of NADPH oxidase (NOX), including diphenyleneiodonium (DPI), but no
Publikováno v:
Free Radical Research. 48:1322-1333
Previous studies have suggested that carbon monoxide (CO) poisoning stimulates cAMP production via purine P2Y11-like receptors in the rat striatum, activating cAMP signaling pathways, resulting in hydroxyl radical ((•)OH) production. Extracellular
Autor:
Ken-ichi Yoshida, Hajime Mizukami, Yoshifumi Takata, Fumi Kuriiwa, Masamune Kobayashi, Yoshinari Goseki, Katshuyuki Fukutake, Shuichi Hara
Publikováno v:
International Journal of Cardiology. 179:421-423
Autor:
Masamune Kobayashi, Hajime Mizukami, Shinjiro Mori, Fumi Kuriiwa, Tatsushige Fukunaga, Shuichi Hara
Publikováno v:
Legal Medicine. 15:91-95
The autopsy findings of a 30-year-old woman who died of cerebral hemorrhage induced by bilateral adrenal pheochromocytoma are presented. The cerebral hemorrhage was shown on the left cerebral hemisphere widely. Her both adrenal glands were severe swe
Publikováno v:
Free Radical Biology and Medicine. 52:1086-1092
Studies have suggested that cAMP signaling pathways may be associated with the production of reactive oxygen species. In this study, we examined how modifications in cAMP signaling affected the production of hydroxyl radicals in rat striatum using mi
Publikováno v:
Toxicology. 288:49-55
We examined the effect of carbon monoxide (CO) poisoning on the production of cAMP, an intracellular second messenger, in rat striatum in terms of extracellular cAMP, which is highly correlated with intracellular cAMP, by using microdialysis. Severe