Zobrazeno 1 - 10
of 321
pro vyhledávání: '"Fujio SUZUKI"'
Autor:
Akira Asai, Yusuke Tsuchimoto, Hideko Ohama, Shinya Fukunishi, Yasuhiro Tsuda, Makiko Kobayashi, Kazuhide Higuchi, Fujio Suzuki
Publikováno v:
OncoImmunology, Vol 6, Iss 4 (2017)
Despite major advances in curative and palliative approaches, hepatocellular carcinoma (HCC) is still the third leading cause of cancer-related death worldwide. M1 macrophages (Mφ) play a key role in host antitumor defenses in HCC. In our study, CD1
Externí odkaz:
https://doaj.org/article/2c57ed95d2a84400b951644e56c4115e
Autor:
Shohei Yoshida, Jong O Lee, Kiwamu Nakamura, Sumihiro Suzuki, David N Hendon, Makiko Kobayashi, Fujio Suzuki
Publikováno v:
PLoS ONE, Vol 9, Iss 2, p e82926 (2014)
Antimicrobial peptides are major host defense effectors against Pseudomonas aeruginosa skin infections. Due to the lack of such peptide production, severely burned hosts are greatly susceptible to P. aeruginosa burn wound infection. β-Defensin (HBD)
Externí odkaz:
https://doaj.org/article/9c7beeddbdca4b8e9797d7d5ea4c1519
Autor:
Shohei Yoshida, Jong O Lee, Kiwamu Nakamura, Sumihiro Suzuki, David N Hendon, Makiko Kobayashi, Fujio Suzuki
Publikováno v:
PLoS ONE, Vol 9, Iss 1, p e83747 (2014)
In our previous studies, peripheral blood lineage(-)CD34(+)CD31(+) cells (CD31(+) IMC) appearing in severely burned patients have been characterized as inhibitor cells for the production of β-defensins (HBDs) by human epidermal keratinocytes (NHEK).
Externí odkaz:
https://doaj.org/article/f54115a3d4fa452d9c59ad89a5796680
Publikováno v:
Gene Therapy. 30:172-179
Gut bacteria-associated sepsis is a serious concern in patients with gastrointestinal acute radiation syndrome (GIARS). In our previous studies, all mice exposed to 8 Gy of whole body γ-irradiation (8 Gy GIARS-mice) died by sepsis stemming from bact
Publikováno v:
The Journal of Immunology. 204:1255-1262
Gut bacteria–associated sepsis is a serious concern in patients with gastrointestinal acute radiation syndrome (GIARS). In our previous studies, gut bacteria–associated sepsis caused high mortality rates in mice exposed to 6–9 Gy of γ-rays. IL
Publikováno v:
The Journal of Immunology. 201:77-86
Macrophages (Mϕ) with the M2b phenotype (Pheno2b-Mϕ) in bacterial translocation sites have been described as cells responsible for the increased susceptibility of mice with gastrointestinal acute radiation syndrome to sepsis caused by gut bacteria.
Publikováno v:
J Immunol
Gut bacteria-associated sepsis is a serious concern in patients with gastrointestinal acute radiation syndrome (GIARS). In our previous studies, gut bacteria-associated sepsis caused high mortality rates in mice exposed to 6–9 Gy of γ-rays (6–9
Publikováno v:
Trends in Research. 2
Publikováno v:
Immunology & Cell Biology. 95:198-206
Mortality associated with Staphylococcus aureus infection remains high during the sub-acute phase of burn injury. In this study, we aimed to improve antibacterial resistance of sub-acutely burned mice through macrophage polarization. Sepsis did not d
Autor:
Tomoki Nishiguchi, Makiko Kobayashi, Kamlesh K. Bhopale, Jong O. Lee, Lawrence C. Sowers, David N. Herndon, Sumihiro Suzuki, Fujio Suzuki, Ichiaki Ito
Publikováno v:
Nucleic Acid Therapeutics. 26:269-276
M2b macrophages (Mφ) play a major role in the increased susceptibility of subacutely burned patients, to sepsis stemming from enterococcal translocation. Certain opportunistic infections in severely burned mice have been controlled by murine CCL1 an