Zobrazeno 1 - 8
of 8
pro vyhledávání: '"Frieda, Chen"'
Publikováno v:
Molecular Biology of the Cell
Up-regulation of BMP2/4 signaling in trabecular bone and/or stromal cells increases osteoblast-specific marker expression in hyperactive Gja1Jrt/+ osteoblasts and may also increase bone marrow adipogenesis by up-regulation of Pparg2 in the Cx43-defic
Autor:
Ralph A Zirngibl, Wolfgang Vogel, Luisa Moreno, Tanya Zappitelli, William G. Cole, Janet Rossant, Lucy R. Osborne, Frieda Chen, Jane E. Aubin, Lee Adamson, Esther Rosenthal, Ruolin Guo, Marc D. Grynpas, Ann M. Flenniken, Shousaku Itoh
Publikováno v:
Journal of Bone and Mineral Research. 29:1412-1423
By using a genome-wide N-ethyl-N-nitrosourea (ENU)-induced dominant mutagenesis screen in mice, a founder with low bone mineral density (BMD) was identified. Mapping and sequencing revealed a T to C transition in a splice donor of the collagen alpha1
Autor:
Janet E. Henderson, Frieda Chen, Jane E Aubin, Marc D. Grynpas, Tanya Zappitelli, Ralph A Zirngibl, Luisa Moreno
Publikováno v:
Journal of Bone and Mineral Research. 28:2400-2413
We previously isolated a low bone mass mouse, Gja1(Jrt) / + , with a mutation in the gap junction protein, alpha 1 gene (Gja1), encoding for a dominant negative G60S Connexin 43 (Cx43) mutant protein. Similar to other Cx43 mutant mouse models describ
Autor:
Tanya, Zappitelli, Frieda, Chen, Luisa, Moreno, Ralph A, Zirngibl, Marc, Grynpas, Janet E, Henderson, Jane E, Aubin
Publikováno v:
Journal of bone and mineral research : the official journal of the American Society for Bone and Mineral Research. 28(11)
We previously isolated a low bone mass mouse, Gja1(Jrt) / + , with a mutation in the gap junction protein, alpha 1 gene (Gja1), encoding for a dominant negative G60S Connexin 43 (Cx43) mutant protein. Similar to other Cx43 mutant mouse models describ
Autor:
Frieda, Chen, Ruolin, Guo, Shousaku, Itoh, Luisa, Moreno, Esther, Rosenthal, Tanya, Zappitelli, Ralph A, Zirngibl, Ann, Flenniken, William, Cole, Marc, Grynpas, Lucy R, Osborne, Wolfgang, Vogel, Lee, Adamson, Janet, Rossant, Jane E, Aubin
Publikováno v:
Journal of bone and mineral research : the official journal of the American Society for Bone and Mineral Research. 29(6)
By using a genome-wide N-ethyl-N-nitrosourea (ENU)-induced dominant mutagenesis screen in mice, a founder with low bone mineral density (BMD) was identified. Mapping and sequencing revealed a T to C transition in a splice donor of the collagen alpha1
Autor:
Ann M. Flenniken, Jane E. Aubin, Lucy R. Osborne, Frieda Chen, S. Lee Adamson, Janet Rossant, Shoji Ichikawa, Celeste Owen
Publikováno v:
Journal of cellular biochemistry. 113(7)
X-linked hypophosphatemic rickets (XLH) is a dominantly inherited disease characterized by renal phosphate wasting, aberrant vitamin D metabolism, and defective bone mineralization. It is known that XLH in humans and in certain mouse models is caused
Autor:
Ralph A Zirngibl, Maya Boudiffa, Brigitte Burt-Pichat, Jean-Paul Roux, Marie-Hélène Lafage-Proust, Georges Boivin, Norbert Laroche, Jia Fei, Ndéye Marième Wade-Gueye, Laurence Vico, Joëlle Amédée, Jane E. Aubin, Pierre Jurdic, Frieda Chen, Luc Malaval, François Duboeuf, Janet Rossant
Publikováno v:
The Journal of Experimental Medecine
The Journal of Experimental Medecine, The Rockefeller University Press, 2008, 205 (5), pp.1145-53. ⟨10.1084/jem.20071294⟩
Journal of Experimental Medicine
Journal of Experimental Medicine, Rockefeller University Press, 2008, 205 (5), pp.1145-53. ⟨10.1084/jem.20071294⟩
The Journal of Experimental Medicine
Journal of Experimental Medicine, 2008, 205 (5), pp.1145-53. ⟨10.1084/jem.20071294⟩
The Journal of Experimental Medecine, The Rockefeller University Press, 2008, 205 (5), pp.1145-53. ⟨10.1084/jem.20071294⟩
Journal of Experimental Medicine
Journal of Experimental Medicine, Rockefeller University Press, 2008, 205 (5), pp.1145-53. ⟨10.1084/jem.20071294⟩
The Journal of Experimental Medicine
Journal of Experimental Medicine, 2008, 205 (5), pp.1145-53. ⟨10.1084/jem.20071294⟩
International audience; Bone sialoprotein (BSP) and osteopontin (OPN) are both highly expressed in bone, but their functional specificities are unknown. OPN knockout ((-/-)) mice do not lose bone in a model of hindlimb disuse (tail suspension), showi
Autor:
Celeste Owen, William L. Stanford, Ralph Zirngibi, Esther Lau, Lia Zitano, Jane E. Aubin, Nicole M. Anderson, Frieda Chen
Publikováno v:
Blood. 108:1392-1392
The interplay between the stroma and hematopoietic progenitors within the bone marrow niche is critical for the homeostatic regulation of both mesenchymal and blood lineages. Gap junctions play an important role in the communication between hematopoi