Zobrazeno 1 - 10
of 10
pro vyhledávání: '"Freidrich Anselmo"'
Autor:
Alexandru Tatomir, Freidrich Anselmo, Dallas Boodhoo, Hegang Chen, Armugam P. Mekala, Vinh Nguyen, Jacob Cuevas, Violeta Rus, Horea Rus
Publikováno v:
Frontiers in Immunology, Vol 15 (2024)
Regular assessment of disease activity in relapsing-remitting multiple sclerosis (RRMS) is required to optimize clinical outcomes. Biomarkers can be a valuable tool for measuring disease activity in multiple sclerosis (MS) if they reflect the patholo
Externí odkaz:
https://doaj.org/article/c1a6b37ddc154eb2b4edd7d48cc60a85
Autor:
Alexandru Tatomir, Gautam Rao, Dallas Boodhoo, Sonia I. Vlaicu, Austin Beltrand, Freidrich Anselmo, Violeta Rus, Horea Rus
Publikováno v:
Frontiers in Immunology, Vol 11 (2020)
Sublytic levels of C5b-9 increase the survival of oligodendrocytes (OLGs) and induce the cell cycle. We have previously observed that SIRT1 co-localizes with surviving OLGs in multiple sclerosis (MS) plaques, but it is not yet known whether SIRT1 is
Externí odkaz:
https://doaj.org/article/782962ac7e694e6f9da9a252777e91d6
Autor:
Freidrich Anselmo, Marcia S. Driscoll
Publikováno v:
Clinics in Dermatology. 39:745-756
Side effects of nutritional supplements, Edited by F. Anselmo, M.S. Driscoll Where on earth did the copy editor decide to do this?Abstract Vitamin and mineral supplement consumption is widespread. They are taken for a variety of conditions, including
Autor:
Horea Rus, Freidrich Anselmo, Dallas Boodhoo, Austin Beltrand, Violeta Rus, Gautam G. Rao, Alexandru Tatomir, Sonia I. Vlaicu
Publikováno v:
Frontiers in Immunology
Frontiers in Immunology, Vol 11 (2020)
Frontiers in Immunology, Vol 11 (2020)
Sublytic levels of C5b-9 increase the survival of oligodendrocytes (OLGs) and induce the cell cycle. We have previously observed that SIRT1 co-localizes with surviving OLGs in multiple sclerosis (MS) plaques, but it is not yet known whether SIRT1 is
Autor:
Freidrich Anselmo, Daniel Hewes, Horea Rus, Alexandru Tatomir, Violeta Rus, Dallas Boodhoo, Jonathan Ciriello
Publikováno v:
Experimental and Molecular Pathology. 105:175-180
We have previously shown that SIRT1 mRNA expression was significantly lower in relapsing MS patients compared to those in remission. Our goal was to longitudinally investigate the role of active, phosphorylated SIRT1 (p-SIRT1) as a potential biomarke
Autor:
Vinh Nguyen, Dallas Boodhoo, Horea Rus, Cornelia Cudrici, Anamaria Talpos-Caia, Armugam P. Mekala, Alexandru Tatomir, Cosmin Tegla, Tudor C. Badea, Freidrich Anselmo, Adam Sugarman, Violeta Rus, Alvaro Martin
Publikováno v:
Immunologic Research. 66:445-461
Extracellular matrix (ECM) deposition in active demyelinating multiple sclerosis (MS) lesions may impede axonal regeneration and can modify immune reactions. Response gene to complement (RGC)-32 plays an important role in the mediation of TGF-β down
Autor:
J. Michael Gagnier, Marcia S. Driscoll, Peter Rady, Freidrich Anselmo, Stephen K. Tyring, Umer Ansari
Publikováno v:
JAAD Case Reports
Autor:
Horea Rus, Freidrich Anselmo, Romeo Ioan Chira, Violeta Rus, Alexandru Tatomir, Sonia I. Vlaicu, Dallas Boodhoo
Publikováno v:
Immunologic research. 67(2-3)
The response gene to complement (RGC)-32 acts as a cell cycle regulator and mediator of TGF-β effects. However, recent studies have revealed other functions for RGC-32 in diverse processes such as cellular migration, differentiation, and fibrosis. I
Autor:
Vinh Nguyen, Alexandru Tatomir, Horea Rus, Freidrich Anselmo, Armugam P. Mekala, Dallas Boodhoo, Violeta Rus
Publikováno v:
Clinical Immunology. 210:108297
In this study, we investigated the role of JNK and phospho-Bcl-2 as possible biomarkers of multiple sclerosis (MS) relapse and of glatiramer acetate (GA) therapeutic response in relapsing-remitting MS patients. We enrolled a cohort of 15 GA-treated p
Autor:
Anamaria Talpos-Caia, Freidrich Anselmo, Dallas Boodhoo, Alexandru Tatomir, Horea Rus, Adam M. Kruszewski, Violeta Rus
Publikováno v:
Immunologic research. 65(6)
Multiple sclerosis (MS) is an inflammatory, demyelinating, and neurodegenerative disease of the central nervous system. The complement system has an established role in the pathogenesis of MS, and evidence suggests that its components can be used as