Zobrazeno 1 - 5
of 5
pro vyhledávání: '"Frederik Teicher Kirk"'
Autor:
Ditte Emilie Munk, Mikkel Holm Vendelbo, Frederik Teicher Kirk, Karina Stubkjær Rewitz, Dirk Andreas Bender, Karina Højrup Vase, Ole Lajord Munk, Hendrik Vilstrup, Peter Ott, Thomas Damgaard Sandahl
Publikováno v:
JHEP Reports, Vol 5, Iss 11, Pp 100916- (2023)
Background & Aims: In Wilson disease (WD), copper accumulation and increased non-ceruloplasmin-bound copper in plasma lead to liver and brain pathology. To better understand the fate of non-ceruloplasmin-bound copper, we used PET/CT to examine the wh
Externí odkaz:
https://doaj.org/article/f70cae192a9b45e88e0a564ea2ce2cbd
Autor:
Ditte Emilie Munk, Tea Lund Laursen, Frederik Teicher Kirk, Hendrik Vilstrup, Aftab Ala, Lars Christian Gormsen, Peter Ott, Thomas Damgaard Sandahl
Publikováno v:
Scientific Reports, Vol 12, Iss 1, Pp 1-10 (2022)
Abstract Zinc inhibits intestinal copper uptake, an effect utilized for treating Wilson’s disease (WD). We used copper-64 (64Cu) PET/CT to examine how much four weeks of treatment with different zinc regimens reduced the hepatic 64Cu content after
Externí odkaz:
https://doaj.org/article/2e7e1fd3793649e4ac2dd3acc0061b69
Autor:
Frederik Teicher Kirk, Ditte Emilie Munk, Jakob Ek, Lisbeth Birk Møller, Mette Bendixen Thorup, Erik Hvid Danielsen, Hendrik Vilstrup, Peter Ott, Thomas Damgaard Sandahl
Publikováno v:
Frontiers in Neurology, Vol 13 (2022)
BackgroundHuppke–Brendel (HB) syndrome is an autosomal recessive disease caused by variants in the SLC33A1 gene. Since 2012, less than ten patients have been reported, none survived year six. With neurologic involvement and ceruloplasmin deficiency
Externí odkaz:
https://doaj.org/article/8485e0fe81d84a37895977f761df3490
Autor:
Ditte Emilie Munk, Tea Lund Laursen, Frederik Teicher Kirk, Hendrik Vilstrup, Aftab Ala, Lars Christian Gormsen, Peter Ott, Thomas Damgaard Sandahl
Publikováno v:
Munk, D E, Lund Laursen, T, Teicher Kirk, F, Vilstrup, H, Ala, A, Gormsen, L C, Ott, P & Damgaard Sandahl, T 2022, ' Effect of oral zinc regimens on human hepatic copper content : a randomized intervention study ', Scientific Reports, vol. 12, no. 1, 14714 . https://doi.org/10.1038/s41598-022-18872-8
Zinc inhibits intestinal copper uptake, an effect utilized for treating Wilson’s disease (WD). We used copper-64 (64Cu) PET/CT to examine how much four weeks of treatment with different zinc regimens reduced the hepatic 64Cu content after oral 64Cu
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::a1a4f570f2adac8808e9c4bfe88c0ea8
https://pure.au.dk/portal/da/publications/effect-of-oral-zinc-regimens-on-human-hepatic-copper-content(5fe5f2e6-0e2a-485d-a644-b58d0d928e14).html
https://pure.au.dk/portal/da/publications/effect-of-oral-zinc-regimens-on-human-hepatic-copper-content(5fe5f2e6-0e2a-485d-a644-b58d0d928e14).html
Autor:
Frederik Teicher Kirk, Hendrik Vilstrup, Thomas Damgaard Sandahl, Ditte Emilie Munk, Mette Munk Lauridsen, Peter Ott, Tea Lund Laursen, Henning Grønbæk
Publikováno v:
Kirk, F T, Munk, D E, Laursen, T L, Vilstrup, H, Ott, P, Grønbæk, H, Lauridsen, M M & Sandahl, T D 2021, ' Cognitive impairment in stable Wilson disease across phenotype ', Metabolic Brain Disease, vol. 36, no. 7, pp. 2173-2177 . https://doi.org/10.1007/s11011-021-00804-6
Background: In Wilson disease (WD), mutations in the gene encoding the ATP7B copper transport protein causes accumulation of copper especially in liver and brain. WD typically presents with hepatic and/or neuropsychiatric symptoms. Impaired cognition