Zobrazeno 1 - 10
of 31
pro vyhledávání: '"Fred E. Samson"'
Publikováno v:
Brain Research. 825:189-193
Kainic acid (KA)-induced seizures elicit edema associated with necrosis in susceptible brain regions (e.g., piriform cortex and hippocampal CA1 and CA3 regions). To test the hypothesis that hypoxia preconditioning protects against KA-induced edema fo
Publikováno v:
Neurochemical Research. 22:735-741
Microdialysis probe insertion into rat cerebral cortex significantly affects the levels of redox-active substances in brain extracellular fluid. Ascorbic acid levels are high immediately after probe insertion, decline rapidly, and then rise as the ra
Publikováno v:
Pharmacology & Toxicology. 68:346-354
Inositol-1-phosphate (Ins1P), an index of phosphoinositide (PI) turnover, was measured in frontal and piriform cortices, caudate, thalamus, hippocampus and cerebellum in saline or LiCl (5 m Eq./kg) pretreated rats 60 min. after graded doses of DFP, p
Publikováno v:
Neurotoxicity research. 5(3)
Public awareness of the dangers of chemical and biological warfare has been heightened in recent times. In particular, chemical nerve agents such as soman and its analogs have been developed and used in war as well as recent incidents, such as in Ira
Publikováno v:
Brain research. Brain research protocols. 4(3)
Hypoxia preconditioning states that a sublethal hypoxia episode will afford neuroprotection against a second challenge in the near future. We describe and discuss a procedure for the development of global hypoxia preconditioning in adult male Wistar
Publikováno v:
Metallothionein IV ISBN: 9783034897990
Chemicals that induce limbic hyperactivity which generalizes into a status epilepticus cause severe neurodegeneration in limbic-related structures (piriform cortex, amygdala, and CA1 and CA3 regions of the hippocampus) [1–5]. Two examples are soman
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_________::db752b1fe97112846432de12b1985ebb
https://doi.org/10.1007/978-3-0348-8847-9_50
https://doi.org/10.1007/978-3-0348-8847-9_50
Publikováno v:
Neuropharmacology. 37(2)
Kainic acid (KA) causes seizures and extensive brain damage in rats. To study the effects of KA on the redox state in cerebral cortex extracellular fluid (ECF), ascorbic and uric acid concentrations were measured in intracerebral microdialysis sample
Publikováno v:
Neuroscience letters. 236(2)
Delayed tissue damage is proposed to be caused by reactive oxygen species. We investigated the effects of microdialysis probe penetration into rat piriform cortex on hydrogen peroxide (H2O2) in brain extracellular fluid (ECF). H2O2 decreased immediat
Publikováno v:
Journal of neuroscience methods. 68(2)
Nitric oxide (NO') is involved in processes such as neurotransmission, memory, brain injury, vessel relaxation, etc. To study the functional and pathological roles of NO' in the brain, a reliable method to monitor NO' directly is needed. Since oxyhem
Publikováno v:
Neurochemistry international. 27(1)
Kainic acid-induced seizures in the rat brain cause severe brain damage that is thought to result, in part, from oxidative stress. In this study, we examine the consequences of systemic administration of kainic acid on expression of several genes tha