Zobrazeno 1 - 10
of 69
pro vyhledávání: '"Fouad T. Chebib"'
Autor:
Stefan Milutinovic, Abraham Bell, Predrag Jancic, Dragana Stanojevic, Abdul Hamid Borghol, Jonathan Mina, Fouad T. Chebib, Ibrahim Khambati, Ricardo O. Escarcega, Malissa J. Wood
Publikováno v:
Journal of Personalized Medicine, Vol 14, Iss 7, p 702 (2024)
Spontaneous coronary artery dissection (SCAD) is a spontaneous intimal tear of the coronary artery wall. A factor rarely associated with SCAD is autosomal dominant polycystic kidney disease (ADPKD). Using the PRISMA guidelines, we identified 10 uniqu
Externí odkaz:
https://doaj.org/article/3eece81c5b364c9082230805962e5a54
Autor:
Dalia Zubidat, Christian Hanna, Amarjyot K. Randhawa, Byron H. Smith, Maroun Chedid, Daniel-Hasan N. Kaidbay, Luca Nardelli, Yaman G. Mkhaimer, Reem M. Neal, Charles D. Madsen, Sarah R. Senum, Adriana V. Gregory, Timothy L. Kline, Ziad M. Zoghby, Stephen M. Broski, Naim S. Issa, Peter C. Harris, Vicente E. Torres, Jad G. Sfeir, Fouad T. Chebib
Publikováno v:
Bone Reports, Vol 18, Iss , Pp 101655- (2023)
ADPKD is caused by pathogenic variants in PKD1 or PKD2, encoding polycystin-1 and -2 proteins. Polycystins are expressed in osteoblasts and chondrocytes in animal models, and loss of function is associated with low bone mineral density (BMD) and volu
Externí odkaz:
https://doaj.org/article/f14b4e1338eb4ca6b2a406927c9c0dc0
Publikováno v:
Kidney Research and Clinical Practice, Vol 41, Iss 4, Pp 422-431 (2022)
Autosomal dominant polycystic kidney disease (ADPKD) is the reported etiology in 10% of end-stage kidney disease (ESKD) patients and has an estimated prevalence of 12.5 million cases worldwide across all ethnicities. There have been major advancement
Externí odkaz:
https://doaj.org/article/2c9b6bfe201949b699cacb4051ab2822
Autor:
Christian Hanna, Theodora A. Potretzke, Maroun Chedid, Laureano J. Rangel, Jennifer Arroyo, Dalia Zubidat, Peter J. Tebben, Andrea G. Cogal, Vicente E. Torres, Peter C. Harris, David J. Sas, John C. Lieske, Dawn S. Milliner, Fouad T. Chebib
Publikováno v:
Kidney Medicine, Vol 4, Iss 3, Pp 100419- (2022)
Rationale & Objective: Hereditary hypophosphatemic rickets with hypercalciuria (HHRH) is a rare monogenic disorder caused by SLC34A3 pathogenic variants. HHRH is characterized by kidney phosphate wasting, hypophosphatemia, hypercalciuria, an elevated
Externí odkaz:
https://doaj.org/article/fb2b003278e2450194649456f90f450b
Autor:
Sonja Suvakov, Hajrunisa Cubro, Wendy M. White, Yvonne S. Butler Tobah, Tracey L. Weissgerber, Kyra L. Jordan, Xiang Y. Zhu, John R. Woollard, Fouad T. Chebib, Natasa M. Milic, Joseph P. Grande, Ming Xu, Tamara Tchkonia, James L. Kirkland, Lilach O. Lerman, Vesna D. Garovic
Publikováno v:
Biology of Sex Differences, Vol 10, Iss 1, Pp 1-13 (2019)
Abstract Background Preeclampsia is a pregnancy-specific hypertensive disorder characterized by impaired angiogenesis. We postulate that senescence of mesenchymal stem cells (MSC), multipotent cells with pro-angiogenic activities, is one of the mecha
Externí odkaz:
https://doaj.org/article/130b90b7b9b840e48df08fc38360e418
Autor:
Sonja Suvakov, Ranine Ghamrawi, Hajrunisa Cubro, Haitao Tu, Wendy M. White, Yvonne S. Butler Tobah, Natasa M. Milic, Joseph P. Grande, Julie M. Cunningham, Fouad T. Chebib, Larissa G.P. Langhi Prata, Yi Zhu, Tamara Tchkonia, James L. Kirkland, Karl A. Nath, Aleksandar Milosavljevic, Vesna D. Garovic
Publikováno v:
EBioMedicine, Vol 70, Iss , Pp 103536- (2021)
Background: Preeclampsia is a pregnancy-specific hypertensive disorder characterized by proteinuria and/or multisystem involvement. Disease-specific therapy has yet to be developed due to the lack of understanding of underlying mechanism(s). We postu
Externí odkaz:
https://doaj.org/article/2d6a2d2b00854797803a0525e383c515
Autor:
Tatsuya Suwabe, Alanna M. Chamberlain, Jill M. Killian, Bernard F. King, Adriana V. Gregory, Charles D. Madsen, Xiaofang Wang, Timothy L. Kline, Fouad T. Chebib, Marie C. Hogan, Patrick S. Kamath, Peter C. Harris, Vicente E. Torres
Publikováno v:
JHEP Reports, Vol 2, Iss 6, Pp 100166- (2020)
Background & Aims: Isolated autosomal-dominant polycystic liver disease (ADPLD) is generally considered a rare disease. However, the frequency of truncating mutations to ADPLD genes in large, population sequencing databases is 1:496. With the increas
Externí odkaz:
https://doaj.org/article/b0a46e63d2e441f8913c41529029e50d
Autor:
Fouad T. Chebib, Marie C. Hogan, Ziad M. El-Zoghby, Maria V. Irazabal, Sarah R. Senum, Christina M. Heyer, Charles D. Madsen, Emilie Cornec-Le Gall, Atta Behfar, Peter C. Harris, Vicente E. Torres
Publikováno v:
Kidney International Reports, Vol 2, Iss 5, Pp 913-923 (2017)
Mutations in PKD1 and PKD2 cause autosomal dominant polycystic kidney disease (ADPKD). Experimental evidence suggests an important role of the polycystins in cardiac development and myocardial function. To determine whether ADPKD may predispose to th
Externí odkaz:
https://doaj.org/article/1e7d8d2cc6b547e5b5c706459663d3b3
Autor:
Wisit Cheungpasitporn, Charat Thongprayoon, Patompong Ungprasert, Karn Wijarnpreecha, Wisit Kaewput, Napat Leeaphorn, Tarun Bathini, Fouad T. Chebib, Paul T. Kröner
Publikováno v:
Journal of Clinical Medicine, Vol 8, Iss 4, p 524 (2019)
Background: This study aimed to evaluate the hospitalization rates for subarachnoid hemorrhage (SAH) among renal transplant patients with adult polycystic kidney disease (ADPKD) and its outcomes, when compared to non-ADPKD renal transplant patients.
Externí odkaz:
https://doaj.org/article/974700834c324e2da1a6e65e0e9b3887
Autor:
Fouad T. Chebib, Ronald D. Perrone
Publikováno v:
Advances in Kidney Disease and Health. 30:261-284