Zobrazeno 1 - 4
of 4
pro vyhledávání: '"Fortunay H. Diatta"'
Autor:
Bishoy Kamel, Andriy Anishkin, Mark Q. Martindale, Damian B. van Rossum, Mónica Medina, Sarah A. Rhodes, Xiaofan Li, Jose Chu Luo, Timothy Jegla, Fortunay H. Diatta, David Simmons, Hansi Liu, Jessica K. Sassic, Liana Trigg
Publikováno v:
Proceedings of the National Academy of Sciences. 112
We examined the origins and functional evolution of the Shaker and KCNQ families of voltage-gated K(+) channels to better understand how neuronal excitability evolved. In bilaterians, the Shaker family consists of four functionally distinct gene fami
Autor:
Michael J. Layden, Anna P. Sberna, Xiaofan Li, Alexandra S. Martinson, Timothy Jegla, Fortunay H. Diatta, Mark Q. Martindale, David Simmons
We examined the evolutionary origins of the ether-à-go-go (EAG) family of voltage-gated K+ channels, which have a strong influence on the excitability of neurons. The bilaterian EAG family comprises three gene subfamilies (Eag, Erg and Elk) distingu
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::be8f5c1fc691b7ad9f98f367c6794537
https://europepmc.org/articles/PMC4334144/
https://europepmc.org/articles/PMC4334144/
Autor:
Fortunay H. Diatta, Xiaofan Li, Sarah A. Rhodes, Timothy Jegla, Jessica K. Sassic, Mark Q. Martindale, Liana Trigg, Hansi Liu, David Simmons
Publikováno v:
Biophysical Journal. 108(2)
The Shaker family of voltage-gated K+ channels in bilaterians consists of four closely related gene subfamilies (Shaker, Shab, Shaw and Shal) that regulate neuronal excitability in diverse ways. We examined the evolutionary origins of the gene family
Autor:
Timothy Jegla, Michael J. Layden, Damian B. van Rossum, Fortunay H. Diatta, Mark Q. Martindale, Alexandra S. Martinson, Sarah A. Rhodes
Publikováno v:
Proceedings of the National Academy of Sciences of the United States of America. 111(15)
Mammalian Ether-a-go-go related gene (Erg) family voltage-gated K(+) channels possess an unusual gating phenotype that specializes them for a role in delayed repolarization. Mammalian Erg currents rectify during depolarization due to rapid, voltage-d