Zobrazeno 1 - 5
of 5
pro vyhledávání: '"Florencia Casanada"'
Publikováno v:
Arteriosclerosis, thrombosis, and vascular biology, vol 17, iss 10
Abstract Investigations into the mechanisms by which diabetes accelerates atherosclerosis have been hampered by the lack of suitable animal models. We hypothesized that streptozotocin-treated LDL receptor-deficient mice would be a good model of diabe
Autor:
Joseph L. Witztum, Claudio Napoli, Mercedes Silvestre, Mario Condorelli, Wulf Palinski, F P D'Armiento, Filomena de Nigris, Florencia Casanada
Publikováno v:
Scopus-Elsevier
Circulation research, vol 89, iss 11
Circulation research, vol 89, iss 11
Maternal hypercholesterolemia during pregnancy is associated with enhanced fatty streak formation in human fetuses and faster progression of atherosclerosis during childhood even under normocholesterolemic conditions. A causal role of maternal hyperc
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::4c4f693943ed3e7a133fc15a93055f16
http://hdl.handle.net/11591/183585
http://hdl.handle.net/11591/183585
Autor:
Mercedes Silvestre, Florencia Casanada, Federico Calara, Natalie Yuan, Wulf Palinski, Claudio Napoli
Apolipoprotein E-deficient (apoE(-/-)) and LDL receptor-deficient (LDLR(-/-)) mice develop extensive atherosclerosis, but the occurrence of spontaneous plaque rupture and secondary thrombosis in these models has not been established. The goal of this
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::ba79aebe2bb753555054559e05790283
http://hdl.handle.net/11591/230209
http://hdl.handle.net/11591/230209
Publikováno v:
Arteriosclerosis, thrombosis, and vascular biology, vol 19, iss 5
Abstract —The role of insulin resistance (IR) in atherogenesis is poorly understood, in part because of a lack of appropriate animal models. We assumed that fructose-fed LDL receptor-deficient (LDLR −/− ) mice might be a model of IR and atheros
Autor:
Rajendra K. Tangirala, Daniel Steinberg, Elizabeth C. Miller, Wulf Palinski, Florencia Casanada, Joseph L. Witztum
Publikováno v:
Arteriosclerosis, thrombosis, and vascular biology. 15(10)
Abstract Apolipoprotein (apo) E–deficient mice develop atherosclerotic lesions that contain epitopes formed during the oxidative modification of lipoproteins, and they demonstrate high titers of circulating autoantibodies against such epitopes, sug