Zobrazeno 1 - 4
of 4
pro vyhledávání: '"Flávia C. A. Gomes"'
Autor:
Danilo Predes, Lorena A. Maia, Isadora Matias, Hannah Paola Mota Araujo, Carolina Soares, Fernanda G. Q. Barros-Aragão, Luiz F. S. Oliveira, Renata R. Reis, Nathalia G. Amado, Alessandro B. C. Simas, Fabio A. Mendes, Flávia C. A. Gomes, Claudia P. Figueiredo, Jose G. Abreu
Publikováno v:
International Journal of Molecular Sciences, Vol 23, Iss 20, p 12078 (2022)
The Wnt/β-catenin signaling pathway dictates cell proliferation and differentiation during embryonic development and tissue homeostasis. Its deregulation is associated with many pathological conditions, including neurodegenerative disease, frequentl
Externí odkaz:
https://doaj.org/article/b6e7926c111541fdb55d182315a0acfc
Autor:
Sheila Espírito Santo Araújo, Henrique Rocha Mendonça, Natalie A. Wheeler, Paula Campello-Costa, Kimberle M. Jacobs, Flávia C. A. Gomes, Michael A. Fox, Babette Fuss
Publikováno v:
Journal of Neuroinflammation, Vol 14, Iss 1, Pp 1-13 (2017)
Abstract Background Multiple sclerosis (MS) is an inflammatory demyelinating disease classically associated with axonal damage and loss; more recently, however, synaptic changes have been recognized as additional contributing factors. An anatomical a
Externí odkaz:
https://doaj.org/article/72843ce7e51446ef8a2a412584ebcd8d
Autor:
Gerardo Rosciszewski, Vanesa Cadena, Jerónimo Auzmendi, María Belén Cieri, Jerónimo Lukin, Alicia R. Rossi, Veronica Murta, Alejandro Villarreal, Analia Reinés, Flávia C. A. Gomes, Alberto Javier Ramos
Publikováno v:
Frontiers in Cellular Neuroscience, Vol 13 (2019)
Temporal Lobe Epilepsy (TLE) is the most common form of human epilepsy and available treatments with antiepileptic drugs are not disease-modifying therapies. The neuroinflammation, neuronal death and exacerbated plasticity that occur during the silen
Externí odkaz:
https://doaj.org/article/9e6be2fdd3534d5a9d057700549d1873
Publikováno v:
The European journal of neuroscience. 33(1)
Cerebellar development in the postnatal period is mainly characterized by an intense cellular proliferation in the external granular layer, followed by migration of granular cells in the molecular layer along the Bergmann glia (BG) fibers. Cerebellar