Zobrazeno 1 - 4
of 4
pro vyhledávání: '"Fernanda Trejo-Millán"'
Autor:
Lucia A Méndez-García, Tania Alvarado-Monroy, Galileo Escobedo, Fernanda Trejo-Millán, José Manuel Fragoso, Halili Minor-Borrego, Ana Laura Sánchez-Del Real, Sergio Islas-Andrade, José A Aguayo-Guerrero, Jahaziel Rosas-Salinas, Juan Carlos Briones-Garduño, Salma A. Rizo-Téllez
Publikováno v:
European Journal of Pediatrics. 180:2529-2537
Interleukin-10 (IL-10) and interferon-gamma (IFN-gamma) are associated with body weight alterations in children, adolescents, and adults. However, little is known regarding the role of IL-10 and IFN-gamma in birth weight of neonates. One hundred eigh
Autor:
Sergio Islas-Andrade, Miguel Carrero-Aguirre, Lucia A Méndez-García, Alfonso Olivos-García, Galileo Escobedo, Miguel Ángel Cid-Soto, José Manuel Fragoso, Fernanda Trejo-Millán, José A Aguayo-Guerrero
Publikováno v:
Journal of Interferon & Cytokine Research. 40:131-138
There is scant information regarding the role of interleukin (IL)-6 in obesity-related metabolic dysfunction in humans. Thus, we studied the serum levels of IL-6 in normal weight, overweight, and obese subjects, and examined associations of IL-6 with
Autor:
Galileo Escobedo, Miguel Carrero-Aguirre, Héctor Ulises Navarrete-Zarco, Fernanda Trejo-Millán, Antonio González-Chávez, Alejandro Chávez, Guillermo Meléndez, Lucia A Méndez-García
Publikováno v:
Archives of medical research. 50(4)
Background Acute myocardial infarction (AMI) is a leading cause of death in Mexico. Atherogenic lipid profile is a key component in AMI. Thus, it is imperative to find drug therapies able to reduce atherogenic lipids in AMI patients and prevent subse
Autor:
Lucia A Méndez-García, Aarón N. Manjarrez-Reyna, Julia Kzhyshkowska, Sergio Islas-Andrade, Camilo P. Martínez-Reyes, Araceli Rojas-Bernabé, Galileo Escobedo, Guillermo Melendez-Mier, Marcela Esquivel-Velázquez, Fernanda Trejo-Millán
Publikováno v:
Scandinavian Journal of Immunology. 88:e12716
Insulin resistance is the inability to respond to insulin and is considered a key pathophysiological factor in the development of type 2 diabetes. Tumor necrosis factor-alpha (TNF-alpha) can directly contribute to insulin resistance by disrupting the