Zobrazeno 1 - 9
of 9
pro vyhledávání: '"Ferenc Boldizsar"'
Autor:
Balint Farkas, Ferenc Boldizsar, Noemi Bohonyi, Nelli Farkas, Saska Marczi, Gabor L. Kovacs, Jozsef Bodis, Miklos Koppan
Publikováno v:
Journal of Ovarian Research, Vol 13, Iss 1, Pp 1-8 (2020)
Abstract Background Ovarian hyperstimulation syndrome (OHSS) is a rare, yet severe, iatrogenic complication of ovulation induction therapy during assisted reproductive procedures. Our group previously detected atypical cells in the ascitic fluid of O
Externí odkaz:
https://doaj.org/article/75fe51b2d6dd444393787c40465872e7
Autor:
Reka Kugyelka, Zoltan Kohl, Katalin Olasz, Katalin Mikecz, Tibor A. Rauch, Tibor T. Glant, Ferenc Boldizsar
Publikováno v:
Mediators of Inflammation, Vol 2016 (2016)
Rheumatoid arthritis (RA) is one of the most common autoimmune disorders characterized by the chronic and progressive inflammation of various organs, most notably the synovia of joints leading to joint destruction, a shorter life expectancy, and redu
Externí odkaz:
https://doaj.org/article/c5c3e4fb53594ea5a262be342f8a257d
Autor:
Oktávia Tarjányi, Julian Haerer, Mónika Vecsernyés, Gergely Berta, Alexandra Stayer-Harci, Bálint Balogh, Kornélia Farkas, Ferenc Boldizsár, József Szeberényi, György Sétáló
Publikováno v:
Scientific Reports, Vol 12, Iss 1, Pp 1-14 (2022)
Abstract Rat pheochromocytoma (PC12) cells were treated with the proteasome inhibitor MG-132 and morphological changes were recorded. Initially, neuronal differentiation was induced but after 24 h signs of morphological deterioration became apparent.
Externí odkaz:
https://doaj.org/article/ebb0e18ea15f423fafa627ca78a178af
Autor:
Szonja Gál, Erzsébet Gajdócsi, Esam Khanfar, Katalin Olasz, Diána Simon, Péter Balogh, Tímea Berki, Péter Németh, Ferenc Boldizsár
Publikováno v:
International Journal of Molecular Sciences, Vol 24, Iss 12, p 9809 (2023)
The natural autoantibody (natAAb) network is thought to play a role in immune regulation. These IgM antibodies react with evolutionary conserved antigens; however, they do not lead to pathological tissue destruction as opposed to pathological autoant
Externí odkaz:
https://doaj.org/article/e51d070fe37545519ea379296754ffa2
Autor:
Ádám Horváth, Éva Borbély, Kata Bölcskei, Nikolett Szentes, Tamás Kiss, Mátyás Belák, Tibor Rauch, Tibor Glant, Róza Zákány, Tamás Juhász, Edina Karanyicz, Ferenc Boldizsár, Zsuzsanna Helyes, Bálint Botz
Publikováno v:
Journal of Neuroinflammation, Vol 15, Iss 1, Pp 1-12 (2018)
Abstract Objective The regulatory role of capsaicin-sensitive peptidergic sensory nerves has been shown in acute inflammation, but little is known about their involvement in T/B-cell driven autoimmune arthritis. This study integratively characterized
Externí odkaz:
https://doaj.org/article/caf56b2373f0485180b48577b7338bc7
Autor:
Katalin Szabó, Ágnes Kemény, Noémi Balázs, Esam Khanfar, Zoltán Sándor, Ferenc Boldizsár, Rolland Gyulai, József Najbauer, Erika Pintér, Tímea Berki
Publikováno v:
Pharmaceuticals, Vol 15, Iss 1, p 57 (2022)
Transient Receptor Potential Ankyrin 1 (TRPA1) has been reported to influence neuroinflammation and lymphocyte function. We analysed the immune phenotype and activation characteristics of TRPA1-deficient mice (knockout—KO) generated by targeted del
Externí odkaz:
https://doaj.org/article/dd1036832b214e75a9da445371a6e86b
Autor:
Esam Khanfar, Katalin Olasz, Fanni Gábris, Erzsébet Gajdócsi, Bálint Botz, Tamás Kiss, Réka Kugyelka, Tímea Berki, Péter Balogh, Ferenc Boldizsár
Publikováno v:
International Journal of Molecular Sciences, Vol 21, Iss 17, p 6162 (2020)
B cells play a crucial role in the pathogenesis of rheumatoid arthritis. In Nkx2-3-deficient mice (Nkx2-3−/−) the spleen’s histological structure is fundamentally changed; therefore, B cell homeostasis is seriously disturbed. Based on this, we
Externí odkaz:
https://doaj.org/article/22284002b4a9498285f810b91b82f525
Autor:
Réka Kugyelka, Lilla Prenek, Katalin Olasz, Zoltán Kohl, Bálint Botz, Tibor T. Glant, Timea Berki, Ferenc Boldizsár
Publikováno v:
Cells, Vol 8, Iss 5, p 504 (2019)
T cells play an essential role in the pathogenesis of both human rheumatoid arthritis (RA) and its murine models. A key molecule in T cell activation is ZAP-70, therefore we aimed to investigate the effects of partial ZAP-70 deficiency on the pathoge
Externí odkaz:
https://doaj.org/article/a74d5afd08354b84a3fbfba0bd44a795
Publikováno v:
International Immunology; May2009, Vol. 21 Issue 5, p511-511, 1p