Zobrazeno 1 - 10
of 47
pro vyhledávání: '"Ferenc Boldizsár"'
Autor:
Oktávia Tarjányi, Julian Haerer, Mónika Vecsernyés, Gergely Berta, Alexandra Stayer-Harci, Bálint Balogh, Kornélia Farkas, Ferenc Boldizsár, József Szeberényi, György Sétáló
Publikováno v:
Scientific Reports, Vol 12, Iss 1, Pp 1-14 (2022)
Abstract Rat pheochromocytoma (PC12) cells were treated with the proteasome inhibitor MG-132 and morphological changes were recorded. Initially, neuronal differentiation was induced but after 24 h signs of morphological deterioration became apparent.
Externí odkaz:
https://doaj.org/article/ebb0e18ea15f423fafa627ca78a178af
Autor:
Szonja Gál, Erzsébet Gajdócsi, Esam Khanfar, Katalin Olasz, Diána Simon, Péter Balogh, Tímea Berki, Péter Németh, Ferenc Boldizsár
Publikováno v:
International Journal of Molecular Sciences, Vol 24, Iss 12, p 9809 (2023)
The natural autoantibody (natAAb) network is thought to play a role in immune regulation. These IgM antibodies react with evolutionary conserved antigens; however, they do not lead to pathological tissue destruction as opposed to pathological autoant
Externí odkaz:
https://doaj.org/article/e51d070fe37545519ea379296754ffa2
Autor:
Ádám Horváth, Éva Borbély, Kata Bölcskei, Nikolett Szentes, Tamás Kiss, Mátyás Belák, Tibor Rauch, Tibor Glant, Róza Zákány, Tamás Juhász, Edina Karanyicz, Ferenc Boldizsár, Zsuzsanna Helyes, Bálint Botz
Publikováno v:
Journal of Neuroinflammation, Vol 15, Iss 1, Pp 1-12 (2018)
Abstract Objective The regulatory role of capsaicin-sensitive peptidergic sensory nerves has been shown in acute inflammation, but little is known about their involvement in T/B-cell driven autoimmune arthritis. This study integratively characterized
Externí odkaz:
https://doaj.org/article/caf56b2373f0485180b48577b7338bc7
Autor:
Katalin Szabó, Ágnes Kemény, Noémi Balázs, Esam Khanfar, Zoltán Sándor, Ferenc Boldizsár, Rolland Gyulai, József Najbauer, Erika Pintér, Tímea Berki
Publikováno v:
Pharmaceuticals, Vol 15, Iss 1, p 57 (2022)
Transient Receptor Potential Ankyrin 1 (TRPA1) has been reported to influence neuroinflammation and lymphocyte function. We analysed the immune phenotype and activation characteristics of TRPA1-deficient mice (knockout—KO) generated by targeted del
Externí odkaz:
https://doaj.org/article/dd1036832b214e75a9da445371a6e86b
Autor:
Esam Khanfar, Katalin Olasz, Fanni Gábris, Erzsébet Gajdócsi, Bálint Botz, Tamás Kiss, Réka Kugyelka, Tímea Berki, Péter Balogh, Ferenc Boldizsár
Publikováno v:
International Journal of Molecular Sciences, Vol 21, Iss 17, p 6162 (2020)
B cells play a crucial role in the pathogenesis of rheumatoid arthritis. In Nkx2-3-deficient mice (Nkx2-3−/−) the spleen’s histological structure is fundamentally changed; therefore, B cell homeostasis is seriously disturbed. Based on this, we
Externí odkaz:
https://doaj.org/article/22284002b4a9498285f810b91b82f525
Autor:
Réka Kugyelka, Lilla Prenek, Katalin Olasz, Zoltán Kohl, Bálint Botz, Tibor T. Glant, Timea Berki, Ferenc Boldizsár
Publikováno v:
Cells, Vol 8, Iss 5, p 504 (2019)
T cells play an essential role in the pathogenesis of both human rheumatoid arthritis (RA) and its murine models. A key molecule in T cell activation is ZAP-70, therefore we aimed to investigate the effects of partial ZAP-70 deficiency on the pathoge
Externí odkaz:
https://doaj.org/article/a74d5afd08354b84a3fbfba0bd44a795
Autor:
Esam Khanfar, Katalin Olasz, Erzsébet Gajdócsi, Xinkai Jia, Timea Berki, Péter Balogh, Ferenc Boldizsár
Publikováno v:
Clinical and experimental immunology. 209(2)
The spleen is the largest secondary lymphoid organ which is involved in the development of B cells and also in systemic (auto)immune responses. Using the recombinant human G1 domain-induced arthritis (GIA) model in splenectomized and control BALB/c m
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::c05a3e0b994eab3303183c4bcbcf8386
https://pubmed.ncbi.nlm.nih.gov/35576510
https://pubmed.ncbi.nlm.nih.gov/35576510
Autor:
Timea Berki, Tímea Litvai, Péter Németh, Noémi Balázs, Réka Kugyelka, József Najbauer, Ferenc Boldizsár, Lilla Prenek
Publikováno v:
Apoptosis
Earlier we have reported that thymic regulatory T cells (Treg) are resistant to in vivo glucocorticoid hormone (GC)-induced apoptosis, while the most GC-sensitive DP thymocytes died through the activation of mitochondrial apoptotic pathway. Here we a
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::5a7517ff43f636d5ebf6cfc97e342dab
https://pubmed.ncbi.nlm.nih.gov/32737651
https://pubmed.ncbi.nlm.nih.gov/32737651
Autor:
Fanni Gábris, Péter Balogh, Bálint Botz, Tamás Kiss, Réka Kugyelka, Ferenc Boldizsár, Katalin Olasz, Timea Berki, Erzsébet Gajdócsi, Esam Khanfar
Publikováno v:
International Journal of Molecular Sciences, Vol 21, Iss 6162, p 6162 (2020)
International Journal of Molecular Sciences
Volume 21
Issue 17
International Journal of Molecular Sciences
Volume 21
Issue 17
B cells play a crucial role in the pathogenesis of rheumatoid arthritis. In Nkx2-3-deficient mice (Nkx2-3&minus
/&minus
) the spleen&rsquo
s histological structure is fundamentally changed
therefore, B cell homeostasis is seriousl
/&minus
) the spleen&rsquo
s histological structure is fundamentally changed
therefore, B cell homeostasis is seriousl
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::e3133d494fd4ad11a4016ab2ef9b5e40
https://www.mdpi.com/1422-0067/21/17/6162
https://www.mdpi.com/1422-0067/21/17/6162
Autor:
József Bódis, Miklós Koppán, Saška Marczi, Noémi Bohonyi, Ferenc Boldizsár, Nelli Farkas, Balint Farkas, Gábor L. Kovács
Publikováno v:
Journal of Ovarian Research
Journal of Ovarian Research, Vol 13, Iss 1, Pp 1-8 (2020)
Journal of Ovarian Research, Vol 13, Iss 1, Pp 1-8 (2020)
Background Ovarian hyperstimulation syndrome (OHSS) is a rare, yet severe, iatrogenic complication of ovulation induction therapy during assisted reproductive procedures. Our group previously detected atypical cells in the ascitic fluid of OHSS patie
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::ff276a318870ef6e73141615ce5e0834
https://doi.org/10.1186/s13048-020-00624-9
https://doi.org/10.1186/s13048-020-00624-9
