Zobrazeno 1 - 10
of 10
pro vyhledávání: '"Fengyuan Qian"'
Publikováno v:
Journal of Experimental & Clinical Cancer Research, Vol 43, Iss 1, Pp 1-16 (2024)
Abstract Background Paclitaxel (PTX) treatment resistance is an important factor leading to poor prognosis in triple-negative breast cancer (TNBC), therefore there is an urgent need to identify new target for combination therapy. Neddylation is a pos
Externí odkaz:
https://doaj.org/article/1a422ba9c77b4f8f88c70135cf74760a
Autor:
Xiqian Zhou, Junyong Zhao, Tao Yan, Danrong Ye, Yuying Wang, Bai’an Zhou, Diya Liu, Xuehui Wang, Wenfang Zheng, Bowen Zheng, Fengyuan Qian, Yating Li, Dengfeng Li, Lin Fang
Publikováno v:
Cell Death and Disease, Vol 15, Iss 4, Pp 1-12 (2024)
Abstract Breast cancer has the highest global incidence and mortality rates among all cancer types. Abnormal expression of the Annexin family has been observed in different malignant tumors, including upregulated ANXA9 in breast cancer. We found high
Externí odkaz:
https://doaj.org/article/55ca147f83b640859b03689fffb4e523
Publikováno v:
Cancer Biology & Therapy, Vol 25, Iss 1 (2024)
Background Breast cancer (BC) is the most prevalent malignant tumor in women globally. Triple-negative breast cancer (TNBC) represents the most malignant and invasive subtype of BC. New therapeutic targets are urgently needed for TNBC owing to its re
Externí odkaz:
https://doaj.org/article/6dc2da755d1f4c21b8ef43831cf82c90
Publikováno v:
Journal of Healthcare Engineering. 2022:1-9
A common and most basic brain tumor is glioma that is exceptionally dangerous to health of various patients. A glioma segmentation, which is primarily magnetic resonance imaging (MRI) oriented, is considered as one of common tools developed for docto
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::80b7fb88976dbafdc41ef2fd2d70e0ca
https://doi.org/10.1155/2022/8507773
https://doi.org/10.1155/2022/8507773
Publikováno v:
Neoplasma. 68(1)
Our previous studies have illustrated that CacyBP/SIP (Calcyclin-binding protein or Siah-1-interacting protein) promoted the proliferation of glioma cells. However, the possible mechanism still needs to be clarified. In the current study, we aimed to
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::63b77ba2d9c7b39457d2aad7a8627a74
https://pubmed.ncbi.nlm.nih.gov/32880469
https://pubmed.ncbi.nlm.nih.gov/32880469
Autor:
Rutong Yu, Xiang Zhang, Zhichao Qiu, Hengliang Shi, Fengyuan Qian, Bin Wang, Lei Wang, Yufu Zhu
Publikováno v:
Journal of Neuro-Oncology. 131:31-39
Jab1 (Jun activation domain-binding protein 1), also known as CSN5 (COP9 signalosome subunit 5), is frequently overexpressed in several cancer types. However, the biological functions and the molecular mechanisms of the Jab1 protein in human gliomas
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::367f809952b29363681aaf13ec975b95
https://doi.org/10.1007/s11060-016-2279-6
https://doi.org/10.1007/s11060-016-2279-6
Autor:
Fengyuan Qian, Bin Wang, Shenshan Zou, Manyi Xie, Chunling Fu, Lei Wang, Hanmo Bao, Shangfeng Gao, Yufu Zhu, Xiang Zhang, Rutong Yu, Hengliang Shi
Publikováno v:
Molecular Neurobiology. 54:5008-5016
Human glioma causes substantial morbidity and mortality worldwide. However, the molecular mechanisms underlying glioma progression are still largely unknown. COP1 (constitutively photomorphogenic 1), an E3 ubiquitin ligase, is important in cell survi
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::96d5ddeea505bbcb1aabce7b058396a0
https://doi.org/10.1007/s12035-016-0033-x
https://doi.org/10.1007/s12035-016-0033-x
Publikováno v:
Panminerva Medica; Sep2023, Vol. 65 Issue 3, p410-411, 2p
Publikováno v:
Neoplasma; 2021, Vol. 68 Issue 1, p119-125, 7p
Autor:
Yuan, Tang, Wenjian, Zhan, Tong, Cao, Tianjin, Tang, Yong, Gao, Zhichao, Qiu, Chunling, Fu, Fengyuan, Qian, Rutong, Yu, Hengliang, Shi
Publikováno v:
IUBMB life. 68(3)
Calcyclin-binding protein or Siah-1-interacting protein (CacyBP/SIP) was previously reported to promote the proliferation of glioma cells. However, the effect of CacyBP/SIP on apoptosis of glioma is poorly understood. Here, our study shows that CacyB
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=pmid________::4bbd95a98807fae2c2376e5d6eab53f2
https://pubmed.ncbi.nlm.nih.gov/26825673
https://pubmed.ncbi.nlm.nih.gov/26825673