Zobrazeno 1 - 6
of 6
pro vyhledávání: '"Fengqun, Tian"'
Publikováno v:
Artificial Cells, Nanomedicine, and Biotechnology, Vol 47, Iss 1, Pp 2205-2212 (2019)
We, the Publisher and Editor of Artificial Cells, Nanomedicine, and Biotechnology, have retracted the following article:Lina Lv, Dandan Li, Fengqun Tian, Xia Li, Jing Zhang & Xiulian Yu (2019) Silence of lncRNA GAS5 alleviates high glucose toxicity t
Externí odkaz:
https://doaj.org/article/8870b895acd741539acca46bc2f71a1d
Publikováno v:
Artificial Cells, Nanomedicine, and Biotechnology, Vol 47, Iss 1, Pp 2940-2947 (2019)
Arbutin (ARB) has been widely used in skin pigmentation disorders. Nevertheless, the involvements of ARB in diabetic nephropathy (DN) are still unknown. We investigated the functions of ARB in high glucose (HG)-induced cell apoptosis and autophagy in
Externí odkaz:
https://doaj.org/article/38c05db467da46119a9dc748da10d693
Publikováno v:
Artificial Cells, Nanomedicine, and Biotechnology. 47:2205-2212
Renal tubular damage caused by persistent high glucose environment has been found to contribute to diabetic nephropathy. This study explored the effects of lncRNA growth arrest-specific 5 (GAS5) on high glucose-stimulated human renal tubular epitheli
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_________::7b7e5a1bee5fe657acb4ae82cc2ff1ae
https://doi.org/10.1080/21691401.2019.1616552
https://doi.org/10.1080/21691401.2019.1616552
Publikováno v:
Artificial cells, nanomedicine, and biotechnology. 47(1)
Arbutin (ARB) has been widely used in skin pigmentation disorders. Nevertheless, the involvements of ARB in diabetic nephropathy (DN) are still unknown. We investigated the functions of ARB in high glucose (HG)-induced cell apoptosis and autophagy in
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::f9025565dfe050782293cfce3c2b1682
https://pubmed.ncbi.nlm.nih.gov/35112627
https://pubmed.ncbi.nlm.nih.gov/35112627
Publikováno v:
Artificial cells, nanomedicine, and biotechnology. 47(1)
Renal tubular damage caused by persistent high glucose environment has been found to contribute to diabetic nephropathy. This study explored the effects of lncRNA growth arrest-specific 5 (GAS5) on high glucose-stimulated human renal tubular epitheli
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=pmid________::61276d7082ea1e1c52061ebb3992d87b
https://pubmed.ncbi.nlm.nih.gov/35112603
https://pubmed.ncbi.nlm.nih.gov/35112603
Publikováno v:
Artificial Cells, Nanomedicine & Biotechnology; Dec2019, Vol. 47 Issue 1, p2940-2947, 8p, 1 Diagram, 5 Graphs